Cooperation between dE2F1 and Yki/Sd defines a distinct transcriptional program necessary to bypass cell cycle exit
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Drosophila as a Potential Model for Ocular TumorsThe Hippo Pathway Targets Rae1 to Regulate Mitosis and Organ Size and to Feed Back to Regulate Upstream Components Merlin, Hippo, and WartsLoss of dE2F compromises mitochondrial functionCombinatorial control of temporal gene expression in the Drosophila wing by enhancers and core promoters.Yap1 activation enables bypass of oncogenic Kras addiction in pancreatic cancerA kinase shRNA screen links LATS2 and the pRB tumor suppressorRole of Drosophila retinoblastoma protein instability element in cell growth and proliferationScalloped and Yorkie are required for cell cycle re-entry of quiescent cells after tissue damage.Control of mitochondrial structure and function by the Yorkie/YAP oncogenic pathwayEvidence for autoregulation and cell signaling pathway regulation from genome-wide binding of the Drosophila retinoblastoma protein.Drosophila GAGA factor is required for full activation of the dE2f1-Yki/Sd transcriptional program.Organ size control by Hippo and TOR pathways.Causes and consequences of genetic background effects illuminated by integrative genomic analysis.Hippo signaling in Drosophila: recent advances and insightsConserved RB functions in development and tumor suppression.The Hippo pathway: key interaction and catalytic domains in organ growth control, stem cell self-renewal and tissue regeneration.The components of Drosophila histone chaperone dCAF-1 are required for the cell death phenotype associated with rbf1 mutation.Control of Proliferation and Cancer Growth by the Hippo Signaling Pathway.Regulation of Drosophila glial cell proliferation by Merlin-Hippo signaling.Genome-wide association of Yorkie with chromatin and chromatin-remodeling complexesSav1 Loss Induces Senescence and Stat3 Activation Coinciding with Tubulointerstitial Fibrosis.miR-285-Yki/Mask double-negative feedback loop mediates blood-brain barrier integrity in Drosophila.Organ size control is dominant over Rb family inactivation to restrict proliferation in vivo.The Hippo pathway kinase Lats2 prevents DNA damage-induced apoptosis through inhibition of the tyrosine kinase c-Abl.Cabut/dTIEG associates with the transcription factor Yorkie for growth control.GTSE1: a novel TEAD4-E2F1 target gene involved in cell protrusions formation in triple-negative breast cancer cell models.Sweet DREAMs for Hippo.A Balance of Yki/Sd Activator and E2F1/Sd Repressor Complexes Controls Cell Survival and Affects Organ Size.Mechanical cue-induced YAP instructs Skp2-dependent cell cycle exit and oncogenic signaling.
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P2860
Cooperation between dE2F1 and Yki/Sd defines a distinct transcriptional program necessary to bypass cell cycle exit
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2011 nî lūn-bûn
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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
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Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
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Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
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Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
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Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
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Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
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prefLabel
Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
@ast
Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
@en
Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
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P2093
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P3181
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Cooperation between dE2F1 and ...... sary to bypass cell cycle exit
@en
P2093
Abul B M M K Islam
Battuya Bayarmagnai
Brandon N Nicolay
Maxim V Frolov
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P304
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P356
10.1101/GAD.1999211
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P577
2011-02-15T00:00:00Z