Ischemic Postconditioning as a Novel Avenue to Protect against Brain Injury after Stroke - Wikidata - awgldk - TriplyDB

Ischemic Postconditioning as a Novel Avenue to Protect against Brain Injury after Stroke

Ischemic Postconditioning as a Novel Avenue to Protect against Brain Injury after Stroke

http://www.wikidata.org/entity/Q29394624

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Chemical Conditioning as an Approach to Ischemic Stroke Tolerance: Mitochondria as the Target Ionic homeostasis in brain conditioning The Role of the PI3K Pathway in the Regeneration of the Damaged Brain by Neural Stem Cells after Cerebral Infarction Histone Deacetylases Exert Class-Specific Roles in Conditioning the Brain and Heart Against Acute Ischemic Injury Non-pharmaceutical therapies for stroke: mechanisms and clinical implications.The Timing of Raf/ERK and AKT Activation in Protecting PC12 Cells against Oxidative Stress NO signaling and S-nitrosylation regulate PTEN inhibition in neurodegeneration Protein Oxidative Modifications: Beneficial Roles in Disease and Health Vascular protection by angiotensin receptor antagonism involves differential VEGF expression in both hemispheres after experimental stroke Volatile anesthetic post-treatment induces protection via inhibition of glycogen synthase kinase 3β in human neuron-like cells.Protein redox modification as a cellular defense mechanism against tissue ischemic injury.Is there a place for cerebral preconditioning in the clinic?Multiple means to the same end: the genetic basis of acquired stress resistance in yeast.Mitochondrial mechanisms in cerebral vascular control: shared signaling pathways with preconditioning.Potential therapeutic benefits of strategies directed to mitochondria.Overexpression of mitochondrial uncoupling protein 2 inhibits inflammatory cytokines and activates cell survival factors after cerebral ischemia.Remote ischemic limb preconditioning after subarachnoid hemorrhage: a phase Ib study of safety and feasibility.Recanalization and reperfusion in acute ischemic stroke.Perconditioning and postconditioning: current knowledge, knowledge gaps, barriers to adoption, and future directions.Protective effects of incensole acetate on cerebral ischemic injury.Remote limb ischemic conditioning enhances motor learning in healthy humans.Reduced Apoptosis by Ethanol and Its Association with PKC-δ and Akt Signaling in Ischemic Stroke Rapamycin treatment improves neuron viability in an in vitro model of stroke.The dual neuroprotective-neurotoxic profile of cannabinoid drugs.Bundled postconditioning therapies improve hemodynamics and neurologic recovery after 17 min of untreated cardiac arrest.Function and mechanism of toll-like receptors in cerebral ischemic tolerance: from preconditioning to treatment.Stenting in the treatment of acute ischemic stroke: literature review Controlled pauses at the initiation of sodium nitroprusside-enhanced cardiopulmonary resuscitation facilitate neurological and cardiac recovery after 15 mins of untreated ventricular fibrillation Effects of Postconditioning on Neurogenesis and Angiogenesis During the Recovery Phase After Focal Cerebral Ischemia Tissue-type plasminogen activator regulates the neuronal uptake of glucose in the ischemic brain.Combination of early and delayed ischemic postconditioning enhances brain-derived neurotrophic factor production by upregulating the ERK-CREB pathway in rats with focal ischemia.Hurdles to clear before clinical translation of ischemic postconditioning against stroke.The role of the cerebral capillaries in acute ischemic stroke: the extended penumbra model.Remote limb ischemic postconditioning protects against neonatal hypoxic-ischemic brain injury in rat pups by the opioid receptor/Akt pathway.Limb remote ischemic per-conditioning in combination with post-conditioning reduces brain damage and promotes neuroglobin expression in the rat brain after ischemic stroke.Neuroprotection induced by post-conditioning following ischemia/reperfusion in mice is associated with altered microRNA expression.Sphingosine protects aging hearts from ischemia/reperfusion injury: Superiority to sphingosine 1-phosphate and ischemic pre- and post-conditioning.Cerebral ischemic post‑conditioning induces autophagy inhibition and a HMGB1 secretion attenuation feedback loop to protect against ischemia reperfusion injury in an oxygen glucose deprivation cellular model Alternative Interventions to Prevent Oxidative Damage following Ischemia/Reperfusion Preconditioning provides neuroprotection in models of CNS disease: paradigms and clinical significance.

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Ischemic Postconditioning as a Novel Avenue to Protect against Brain Injury after Stroke

http://www.wikidata.org/entity/Q29394624

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2009 nî lūn-bûn

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2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2009 թվականի մայիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Ischemic Postconditioning as a Novel Avenue to Protect against Brain Injury after Stroke

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Journal of Cerebral Blood Flow & Metabolism

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Ischemic Postconditioning as a Novel Avenue to Protect against Brain Injury after Stroke

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Heng Zhao

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P2860

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

Safety and efficacy of mechanical embolectomy in acute ischemic stroke: results of the MERCI trial

Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

Copper/zinc superoxide dismutase attenuates neuronal cell death by preventing extracellular signal-regulated kinase activation after transient focal cerebral ischemia in mice

Postconditioning with isoflurane reduced ischemia-induced brain injury in rats.

The onset of postischemic hypoperfusion in rats is precipitous and may be controlled by local neurons.

Delayed postconditioning protects against focal ischemic brain injury in rats.

Implication of cysteine proteases calpain, cathepsin and caspase in ischemic neuronal death of primates.

Delayed effects of sublethal ischemia on the acquisition of tolerance to ischemia.

Gene therapy against neurological insults: sparing neurons versus sparing function.

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873-885

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10.1038/JCBFM.2009.13

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19240739

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