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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

http://www.wikidata.org/entity/Q29614203

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Oxidation in rheumatoid arthritis.Structural and mechanistic studies on the inhibition of the hypoxia-inducible transcription factor hydroxylases by tricarboxylic acid cycle intermediates SENP3 is responsible for HIF-1 transactivation under mild oxidative stress via p300 de-SUMOylation Acute hypoxia selectively inhibits KCNA5 channels in pulmonary artery smooth muscle cells.Regulation of late G1/S phase transition and APC Cdh1 by reactive oxygen species Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress Autophagy in cancer associated fibroblasts promotes tumor cell survival: Role of hypoxia, HIF1 induction and NFκB activation in the tumor stromal microenvironment Oxidative stress in cancer associated fibroblasts drives tumor-stroma co-evolution: A new paradigm for understanding tumor metabolism, the field effect and genomic instability in cancer cells How mitochondria produce reactive oxygen species Stanniocalcin: A molecular guard of neurons during cerebral ischemia Hypoxic pulmonary vasoconstriction: role of voltage-gated potassium channels Oxygen-sensing mechanisms and the regulation of redox-responsive transcription factors in development and pathophysiology Detection of reactive oxygen species in isolated, perfused lungs by electron spin resonance spectroscopy.Hypoxia and its implications in rheumatoid arthritis "Oxygen Sensing" by Na,K-ATPase: These Miraculous Thiols Fundamentals of cancer metabolism Mitochondrial metabolic remodeling in response to genetic and environmental perturbations Reactive Nitrogen Species in Mitochondria and Their Implications in Plant Energy Status and Hypoxic Stress Tolerance Longevity of animals under reactive oxygen species stress and disease susceptibility due to global warming Relationship between Oxidative Stress, Circadian Rhythms, and AMD Cross Talk of Proteostasis and Mitostasis in Cellular Homeodynamics, Ageing, and Disease Hypoxia Inducible Factor Pathway and Physiological Adaptation: A Cell Survival Pathway?Antioxidant responses and cellular adjustments to oxidative stress Lung cell hypoxia: role of mitochondrial reactive oxygen species signaling in triggering responses To breathe or not to breathe: the haematopoietic stem/progenitor cells dilemma Reactive oxygen species in cancer stem cells Mitochondrial ROS in cancer: initiators, amplifiers or an Achilles' heel?Mitohormesis Interplay between sirtuins, MYC and hypoxia-inducible factor in cancer-associated metabolic reprogramming Mitochondria in the regulation of innate and adaptive immunity Hypoxia-inducible factors in physiology and medicine Targeting the redox balance in inflammatory skin conditions Mfn2 is Required for Mitochondrial Development and Synapse Formation in Human Induced Pluripotent Stem Cells/hiPSC Derived Cortical Neurons Genetic ablation of Bcl-x attenuates invasiveness without affecting apoptosis or tumor growth in a mouse model of pancreatic neuroendocrine cancer Reactive oxygen species-mediated regulation of mitochondrial biogenesis in the yeast Saccharomyces cerevisiae.Oxygen-regulated isoforms of cytochrome c oxidase have differential effects on its nitric oxide production and on hypoxic signaling Diaphragm Muscle Adaptation to Sustained Hypoxia: Lessons from Animal Models with Relevance to High Altitude and Chronic Respiratory Diseases Exploring the developmental mechanisms underlying Wolf-Hirschhorn Syndrome: Evidence for defects in neural crest cell migration Discovery of cytoglobin and its roles in physiology and pathology of hepatic stellate cells The ever-expanding role of HIF in tumour and stromal biology

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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

http://www.wikidata.org/entity/Q29614203

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1998 nî lūn-bûn

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1998 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

1998 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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1998年の論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年論文

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1998年论文

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name

Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

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Proceedings of the National Academy of Sciences of the United States of America

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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

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Chandel NS

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Goldwasser E

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Maltepe E

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Mathieu CE

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Schumacker PT

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Simon MC

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A nuclear factor induced by hypoxia via de novo protein synthesis binds to the human erythropoietin gene enhancer at a site required for transcriptional activation

Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

Transcriptional regulation of genes encoding glycolytic enzymes by hypoxia-inducible factor 1

Reactive oxygen intermediates as apparently widely used messengers in the activation of the NF-kappa B transcription factor and HIV-1

Activation of hypoxia-inducible transcription factor depends primarily upon redox-sensitive stabilization of its alpha subunit

Role of hydrogen peroxide in hypoxia-induced erythropoietin production

Human cells lacking mtDNA: repopulation with exogenous mitochondria by complementation

Hypoxia-inducible factor 1alpha (HIF-1alpha) protein is rapidly degraded by the ubiquitin-proteasome system under normoxic conditions. Its stabilization by hypoxia depends on redox-induced changes

Erythropoietin: structure, control of production, and function.

Functional analysis of an oxygen-regulated transcriptional enhancer lying 3' to the mouse erythropoietin gene.

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