Deletion of the ABL SH3 domain reactivates de-oligomerized BCR-ABL for growth factor independence.
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The NH(2)-terminal coiled-coil domain and tyrosine 177 play important roles in induction of a myeloproliferative disease in mice by Bcr-AblRecent advances in the molecular and cellular biology of chronic myeloid leukaemia: lessons to be learned from the laboratory.Modeling Philadelphia chromosome positive leukemias.
P2860
Deletion of the ABL SH3 domain reactivates de-oligomerized BCR-ABL for growth factor independence.
description
1996 nî lūn-bûn
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1996 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1996年の論文
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1996年学术文章
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1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
name
Deletion of the ABL SH3 domain ...... or growth factor independence.
@ast
Deletion of the ABL SH3 domain ...... or growth factor independence.
@en
type
label
Deletion of the ABL SH3 domain ...... or growth factor independence.
@ast
Deletion of the ABL SH3 domain ...... or growth factor independence.
@en
prefLabel
Deletion of the ABL SH3 domain ...... or growth factor independence.
@ast
Deletion of the ABL SH3 domain ...... or growth factor independence.
@en
P2093
P2860
P1433
P1476
Deletion of the ABL SH3 domain ...... or growth factor independence.
@en
P2093
P2860
P304
P356
10.1016/0014-5793(95)01518-3
P407
P577
1996-02-01T00:00:00Z