Palatability Can Drive Feeding Independent of AgRP Neurons. - Wikidata - awgldk - TriplyDB

Palatability Can Drive Feeding Independent of AgRP Neurons.

Palatability Can Drive Feeding Independent of AgRP Neurons.

http://www.wikidata.org/entity/Q30278852

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The Use of DREADDs to Deconstruct Behavior Hunger and Satiety Gauge Reward Sensitivity VMAT2-Mediated Neurotransmission from Midbrain Leptin Receptor Neurons in Feeding Regulation Cholinergic and ghrelinergic receptors and KCNQ channels in the medial PFC regulate the expression of palatability.The agouti-related peptide binds heparan sulfate through segments critical for its orexigenic effects.DREADDs for Neuroscientists.New inducible genetic method reveals critical roles of GABA in the control of feeding and metabolism.Pituitary Adenylate-Cyclase Activating Polypeptide Regulates Hunger- and Palatability-Induced Binge Eating New Technologies for Elucidating Opioid Receptor Function.Role of the gut microbiota in host appetite control: bacterial growth to animal feeding behaviour.Interacting Neural Processes of Feeding, Hyperactivity, Stress, Reward, and the Utility of the Activity-Based Anorexia Model of Anorexia Nervosa.Involvement of Astrocytes in Mediating the Central Effects of Ghrelin.Peripheral cannabinoid-1 receptor blockade restores hypothalamic leptin signaling.Hunger: The carrot and the stick.Perinatal Western Diet Consumption Leads to Profound Plasticity and GABAergic Phenotype Changes within Hypothalamus and Reward Pathway from Birth to Sexual Maturity in Rat.Nutritive, Post-ingestive Signals Are the Primary Regulators of AgRP Neuron Activity.Asprosin is a centrally acting orexigenic hormone.Overlapping Brain Circuits for Homeostatic and Hedonic Feeding.Effects of meal palatability on postprandial sensations.Chemogenetic Tools for Causal Cellular and Neuronal Biology.[Hunger and satiety factors in the regulation of pleasure associated with feeding behavior].The Parabrachial Nucleus: CGRP Neurons Function as a General Alarm.Brain to bone: What is the contribution of the brain to skeletal homeostasis?Prebiotics Supplementation Impact on the Reinforcing and Motivational Aspect of Feeding.Is Smaller Better? A Proposal to Use Bacteria For Neuroscientific Modeling.Estrogenic-dependent glutamatergic neurotransmission from kisspeptin neurons governs feeding circuits in females p53 in AgRP neurons is required for protection against diet-induced obesity via JNK1 Sugar Promotes Feeding in Flies via the Serine Protease Homolog scarface

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P2860

Palatability Can Drive Feeding Independent of AgRP Neurons.

http://www.wikidata.org/entity/Q30278852

description

2015 nî lūn-bûn

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2015 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի օգոստոսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Palatability Can Drive Feeding Independent of AgRP Neurons.

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Palatability Can Drive Feeding Independent of AgRP Neurons.

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Palatability Can Drive Feeding Independent of AgRP Neurons.

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Palatability Can Drive Feeding Independent of AgRP Neurons.

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J.CMET.2015.07.011

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Cell Metabolism

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Palatability Can Drive Feeding Independent of AgRP Neurons.

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Amélie Lacombe

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Anne-Sophie Delbès

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Aurélie Joly-Amado

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Bénédicte Dehouck

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Claude Rouch

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Céline Cansell

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Fanny Langlet

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Jean-Alain Fehrentz

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Julien Castel

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Michael J Krashes

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P2860

Ghrelin is a growth-hormone-releasing acylated peptide from stomach

Ghrelin increases intake of rewarding food in rodents

Ghrelin modulates the activity and synaptic input organization of midbrain dopamine neurons while promoting appetite

Effects of hypothalamic neurodegeneration on energy balance.

Leptin receptor signaling in midbrain dopamine neurons regulates feeding

The metabolic syndrome

Central nervous system control of food intake and body weight

Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats

Rapid, reversible activation of AgRP neurons drives feeding behavior in mice

Transient activation of specific neurons in mice by selective expression of the capsaicin receptor.

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646-657

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scholarly article

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10.1016/J.CMET.2015.07.011

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4

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22

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Christophe Magnan

Marie Schaeffer

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2015-08-12T00:00:00Z

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281082174

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26278050

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5024566

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