Effects of hypothalamic neurodegeneration on energy balance. - Wikidata - awgldk - TriplyDB

Effects of hypothalamic neurodegeneration on energy balance.

Effects of hypothalamic neurodegeneration on energy balance.

http://www.wikidata.org/entity/Q24810998

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SnoRNA Snord116 (Pwcr1/MBII-85) deletion causes growth deficiency and hyperphagia in mice Hypothalamic miRNAs: emerging roles in energy balance control Central nervous control of energy and glucose balance: focus on the central melanocortin system Hypothalamic malonyl-CoA and the control of food intake Hypothalamic Agrp neurons drive stereotypic behaviors beyond feeding.Magel2 is required for leptin-mediated depolarization of POMC neurons in the hypothalamic arcuate nucleus in mice Hypothalamic neurodegeneration and adult-onset obesity in mice lacking the Ubb polyubiquitin gene Dmbx1 is essential in agouti-related protein action Abdominal fat distribution and its relationship to brain changes: the differential effects of age on cerebellar structure and function: a cross-sectional, exploratory study PDK1-Foxo1 in agouti-related peptide neurons regulates energy homeostasis by modulating food intake and energy expenditure Central nervous system control of food intake and body weight Palatability Can Drive Feeding Independent of AgRP Neurons.Postnatal ablation of POMC neurons induces an obese phenotype characterized by decreased food intake and enhanced anxiety-like behavior.AMPK is essential for energy homeostasis regulation and glucose sensing by POMC and AgRP neurons.Rapid, reversible activation of AgRP neurons drives feeding behavior in mice Whole-brain mapping of the direct inputs and axonal projections of POMC and AgRP neurons.Proliferative hypothalamic neurospheres express NPY, AGRP, POMC, CART and Orexin-A and differentiate to functional neurons.PDK1-FoxO1 pathway in AgRP neurons of arcuate nucleus promotes bone formation via GHRH-GH-IGF1 axis.Functional role of c-Jun-N-terminal kinase in feeding regulation.Gamma-protocadherins regulate the functional integrity of hypothalamic feeding circuitry in mice.Liver ERα regulates AgRP neuronal activity in the arcuate nucleus of female mice The tyrosine kinase receptor Tyro3 enhances lifespan and neuropeptide Y (Npy) neuron survival in the mouse anorexia (anx) mutation.Functional role of suppressor of cytokine signaling 3 upregulation in hypothalamic leptin resistance and long-term energy homeostasis.Agrp neurons mediate Sirt1's action on the melanocortin system and energy balance: roles for Sirt1 in neuronal firing and synaptic plasticity.20 years of leptin: leptin and reproduction: past milestones, present undertakings, and future endeavors.mTOR-mediated dedifferentiation of the retinal pigment epithelium initiates photoreceptor degeneration in mice.De novo neurogenesis in adult hypothalamus as a compensatory mechanism to regulate energy balance.Hypothalamic proopiomelanocortin processing and the regulation of energy balance Hypothalamic survival circuits: blueprints for purposive behaviors Evidence for NG2-glia derived, adult-born functional neurons in the hypothalamus.Leptin action on GABAergic neurons prevents obesity and reduces inhibitory tone to POMC neurons Loss of agouti-related peptide does not significantly impact the phenotype of murine POMC deficiency.Remodeling of the arcuate nucleus energy-balance circuit is inhibited in obese mice.Fasting activation of AgRP neurons requires NMDA receptors and involves spinogenesis and increased excitatory tone.Hypothalamic Non-AgRP, Non-POMC GABAergic Neurons Are Required for Postweaning Feeding and NPY Hyperphagia.Regulation of GABA and glutamate release from proopiomelanocortin neuron terminals in intact hypothalamic networks The melanocortin-4 receptor: physiology, pharmacology, and pathophysiology.The MC4 receptor and control of appetite.The role of GluN2A and GluN2B NMDA receptor subunits in AgRP and POMC neurons on body weight and glucose homeostasis.Hypothalamic AgRP-neurons control peripheral substrate utilization and nutrient partitioning

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P2860

Effects of hypothalamic neurodegeneration on energy balance.

http://www.wikidata.org/entity/Q24810998

description

2005 nî lūn-bûn

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2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Effects of hypothalamic neurodegeneration on energy balance

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Effects of hypothalamic neurodegeneration on energy balance.

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Effects of hypothalamic neurodegeneration on energy balance.

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type

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Effects of hypothalamic neurodegeneration on energy balance

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Effects of hypothalamic neurodegeneration on energy balance.

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Effects of hypothalamic neurodegeneration on energy balance.

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prefLabel

Effects of hypothalamic neurodegeneration on energy balance

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Effects of hypothalamic neurodegeneration on energy balance.

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Effects of hypothalamic neurodegeneration on energy balance.

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JOURNAL.PBIO.0030415

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Effects of hypothalamic neurodegeneration on energy balance.

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Allison Wanting Xu

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Christopher B Kaelin

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Denis G Baskin

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Gregory J Morton

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Gregory S Barsh

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James Graham

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Kayoko Ogimoto

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Kimber Stanhope

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Michael W Schwartz

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Peter Havel

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P2860

Premature ageing in mice expressing defective mitochondrial DNA polymerase

Antagonism of central melanocortin receptors in vitro and in vivo by agouti-related protein

Neither agouti-related protein nor neuropeptide Y is critically required for the regulation of energy homeostasis in mice

Reducing hypothalamic AGRP by RNA interference increases metabolic rate and decreases body weight without influencing food intake.

Generalized lacZ expression with the ROSA26 Cre reporter strain

Central nervous system control of food intake

Mitochondria, oxidants, and aging

Leptin activates anorexigenic POMC neurons through a neural network in the arcuate nucleus

Mitochondrial transcription factor A is necessary for mtDNA maintenance and embryogenesis in mice

PI3K integrates the action of insulin and leptin on hypothalamic neurons.

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e415

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scholarly article

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10.1371/JOURNAL.PBIO.0030415

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