Severe inflammatory defect and reduced viability in CD18 and E-selectin double-mutant mice.

Severe inflammatory defect and reduced viability in CD18 and E-selectin double-mutant mice.

Item
http://www.wikidata.org/entity/Q30306581
A novel method to analyze leukocyte rolling behavior in vivoCXCR2- and E-selectin-induced neutrophil arrest during inflammation in vivo.Targeted delivery of therapeutics to endothelium.Loss of estrogen receptor beta decreases mitochondrial energetic potential and increases thrombogenicity of platelets in aged female mice.Atorvastatin protects against cerebral ischemia/reperfusion injury through anti-inflammatory and antioxidant effectsThe state diagram for cell adhesion mediated by two receptors.Sphingosine-1-phosphate lyase deficiency produces a pro-inflammatory response while impairing neutrophil trafficking.Suppression of prostate cancer cell rolling and adhesion to endothelium by 1α,25-dihydroxyvitamin D3.Heterogeneity of endothelial cells from different organ sites in T-cell subset recruitment.NG2 proteoglycan-dependent recruitment of tumor macrophages promotes pericyte-endothelial cell interactions required for brain tumor vascularization.Ly6G ligation blocks recruitment of neutrophils via a β2-integrin-dependent mechanismNG2 Proteoglycan-Dependent Contributions of Pericytes and Macrophages to Brain Tumor Vascularization and Progression.E-selectin gene polymorphism (A561C) and essential hypertension. Meta-analysis in the Chinese population.NG2 Proteoglycan Enhances Brain Tumor Progression by Promoting Beta-1 Integrin Activation in both Cis and Trans Orientations.VEGF₁₆₄ differentially regulates neutrophil and T cell adhesion through ItgaL- and ItgaM-dependent mechanisms.More than the sum of the parts: cooperation between leukocyte adhesion receptors during extravasation.A common missense single nucleotide polymorphism in the E-selectin gene is significantly associated with essential hypertension in the Han population but only weakly associated in the Uygur population.
P2860

Severe inflammatory defect and reduced viability in CD18 and E-selectin double-mutant mice.

Item
http://www.wikidata.org/entity/Q30306581
description
2000 nî lūn-bûn
@nan
2000 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
name
Severe inflammatory defect and ...... E-selectin double-mutant mice.
@ast
Severe inflammatory defect and ...... E-selectin double-mutant mice.
@en
type
label
Severe inflammatory defect and ...... E-selectin double-mutant mice.
@ast
Severe inflammatory defect and ...... E-selectin double-mutant mice.
@en
prefLabel
Severe inflammatory defect and ...... E-selectin double-mutant mice.
@ast
Severe inflammatory defect and ...... E-selectin double-mutant mice.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P921
P932
P356
P1433
P1476
Severe inflammatory defect and ...... E-selectin double-mutant mice.
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P921
P932