Caspase-3-mediated cleavage of amyloid precursor protein and formation of amyloid Beta peptide in traumatic axonal injury.
about
Epidemiology of Alzheimer diseaseMolecular mechanisms of cognitive dysfunction following traumatic brain injuryEffects of low-level blast exposure on the nervous system: is there really a controversy?BRAIN MYELINATION IN PREVALENT NEUROPSYCHIATRIC DEVELOPMENTAL DISORDERS: PRIMARY AND COMORBID ADDICTION.Novel model of frontal impact closed head injury in the rat.Axonal pathology in traumatic brain injury.Inflammation and white matter degeneration persist for years after a single traumatic brain injury.A lack of amyloid beta plaques despite persistent accumulation of amyloid beta in axons of long-term survivors of traumatic brain injuryModulation of ABCA1 by an LXR agonist reduces β-amyloid levels and improves outcome after traumatic brain injuryLong-term accumulation of amyloid-beta, beta-secretase, presenilin-1, and caspase-3 in damaged axons following brain traumaEnhanced β-secretase processing alters APP axonal transport and leads to axonal defects.Amyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies.Brain injury, neuroinflammation and Alzheimer's disease.Glial reactions and degeneration of myelinated processes in spinal cord gray matter in chronic experimental autoimmune encephalomyelitis.Role of calpains in the injury-induced dysfunction and degeneration of the mammalian axon.Axon degeneration is key component of neuronal death in amyloid-β toxicity.Experimental traumatic brain injury induces rapid aggregation and oligomerization of amyloid-beta in an Alzheimer's disease mouse model.Traumatic brain injury and amyloid-β pathology: a link to Alzheimer's disease?Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humansStrong Correlation of Genome-Wide Expression after Traumatic Brain Injury In Vitro and In Vivo Implicates a Role for SORLA.Disordered APP metabolism and neurovasculature in trauma and aging: Combined risks for chronic neurodegenerative disorders.The Role of Microglia in the Etiology and Evolution of Chronic Traumatic Encephalopathy.Inhibition of Endocannabinoid Degradation Improves Outcomes from Mild Traumatic Brain Injury: A Mechanistic Role for Synaptic Hyperexcitability.Traumatic Brain Injury as a Trigger of Neurodegeneration.Extensive degradation of myelin basic protein isoforms by calpain following traumatic brain injury.Neuromodulatory Effect of Thymoquinone in Attenuating Glutamate-Mediated Neurotoxicity Targeting the Amyloidogenic and Apoptotic Pathways.
P2860
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P2860
Caspase-3-mediated cleavage of amyloid precursor protein and formation of amyloid Beta peptide in traumatic axonal injury.
description
2002 nî lūn-bûn
@nan
2002 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Caspase-3-mediated cleavage of ...... de in traumatic axonal injury.
@ast
Caspase-3-mediated cleavage of ...... de in traumatic axonal injury.
@en
type
label
Caspase-3-mediated cleavage of ...... de in traumatic axonal injury.
@ast
Caspase-3-mediated cleavage of ...... de in traumatic axonal injury.
@en
prefLabel
Caspase-3-mediated cleavage of ...... de in traumatic axonal injury.
@ast
Caspase-3-mediated cleavage of ...... de in traumatic axonal injury.
@en
P2093
P1476
Caspase-3-mediated cleavage of ...... de in traumatic axonal injury.
@en
P2093
David O Okonkwo
Gregory A Helm
James R Stone
John T Povlishock
Leman K Mutlu
Richard H Singleton
P304
P356
10.1089/089771502753754073
P577
2002-05-01T00:00:00Z