Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humans - Wikidata - awgldk - TriplyDB

Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humans

Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humans

http://www.wikidata.org/entity/Q37691584

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Chronic Traumatic Encephalopathy in Athletes: Progressive Tauopathy After Repetitive Head Injury Alzheimer's disease as homeostatic responses to age-related myelin breakdown Chronic traumatic encephalopathy: a potential late effect of sport-related concussive and subconcussive head trauma Tau elevations in the brain extracellular space correlate with reduced amyloid-β levels and predict adverse clinical outcomes after severe traumatic brain injury Therapeutic potential of mood stabilizers lithium and valproic acid: beyond bipolar disorder A new look at an old drug: neuroprotective effects and therapeutic potentials of lithium salts Neurobiological consequences of traumatic brain injury The airbag problem-a potential culprit for bench-to-bedside translational efforts: relevance for Alzheimer's disease Chronic traumatic encephalopathy: a spectrum of neuropathological changes following repetitive brain trauma in athletes and military personnel Axonal transport of APP and the spatial regulation of APP cleavage and function in neuronal cells Evaluating and treating neurobehavioral symptoms in professional American football players: Lessons from a case series.Reducing synuclein accumulation improves neuronal survival after spinal cord injury Involvement of aberrant cyclin-dependent kinase 5/p25 activity in experimental traumatic brain injury Neurochemical profile of dementia pugilistica Molecular mechanisms of cognitive dysfunction following traumatic brain injury Self-reported head injury and risk of late-life impairment and AD pathology in an AD center cohort Examining the relationship between head trauma and neurodegenerative disease: A review of epidemiology, pathology and neuroimaging techniques The neuropathology of traumatic brain injury.Biochemical assessment of precuneus and posterior cingulate gyrus in the context of brain aging and Alzheimer's disease Effects of low-level blast exposure on the nervous system: is there really a controversy?Chronic traumatic encephalopathy: clinical-biomarker correlations and current concepts in pathogenesis.Acute blast injury reduces brain abeta in two rodent species.What's New in Traumatic Brain Injury: Update on Tracking, Monitoring and Treatment The evolution of cost-efficiency in neural networks during recovery from traumatic brain injury Rationale for therapeutic silencing of alpha-synuclein in Parkinson's disease Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury.A neprilysin polymorphism and amyloid-beta plaques after traumatic brain injury.A new avenue for lithium: intervention in traumatic brain injury.Increased Amyloid Precursor Protein and Tau Expression Manifests as Key Secondary Cell Death in Chronic Traumatic Brain Injury.Genetic inhibition of phosphorylation of the translation initiation factor eIF2α does not block Aβ-dependent elevation of BACE1 and APP levels or reduce amyloid pathology in a mouse model of Alzheimer's disease.Pathological correlations between traumatic brain injury and chronic neurodegenerative diseases.Multiple oxygen tension environments reveal diverse patterns of transcriptional regulation in primary astrocytes.Distinct temporal and anatomical distributions of amyloid-β and tau abnormalities following controlled cortical impact in transgenic mice Mild traumatic brain injury: a risk factor for neurodegeneration.TDP-43 proteinopathy and motor neuron disease in chronic traumatic encephalopathy.Immunoexcitotoxicity as a central mechanism in chronic traumatic encephalopathy-A unifying hypothesis.Axonal pathology in traumatic brain injury.Dual vulnerability of tau to calpains and caspase-3 proteolysis under neurotoxic and neurodegenerative conditions.Alpha-synuclein aggregation induced by brief ischemia negatively impacts neuronal survival in vivo: a study in [A30P]alpha-synuclein transgenic mouse α-Synuclein levels are elevated in cerebrospinal fluid following traumatic brain injury in infants and children: the effect of therapeutic hypothermia.

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Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humans

http://www.wikidata.org/entity/Q37691584

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 July 2007

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Multiple proteins implicated i ...... s after brain trauma in humans

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Multiple proteins implicated i ...... after brain trauma in humans.

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Multiple proteins implicated i ...... s after brain trauma in humans

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Multiple proteins implicated i ...... after brain trauma in humans.

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Multiple proteins implicated i ...... s after brain trauma in humans

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Multiple proteins implicated i ...... after brain trauma in humans.

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J.EXPNEUROL.2007.06.018

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Experimental Neurology

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Multiple proteins implicated i ...... s after brain trauma in humans

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Dan Martinez

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David I Graham

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Douglas H Smith

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Kevin D Browne

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Kunihiro Uryu

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Victoria E Johnson

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Virginia M-Y Lee

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Xiao-Han Chen

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P2860

Beta-secretase cleavage of Alzheimer's amyloid precursor protein by the transmembrane aspartic protease BACE

Neuropathological stageing of Alzheimer-related changes

Alzheimer's disease: genes, proteins, and therapy

Axonal transport of amyloid precursor protein is mediated by direct binding to the kinesin light chain subunit of kinesin-I

Deficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor protein

Axonal transport, amyloid precursor protein, kinesin-1, and the processing apparatus: revisited

Kinesin-mediated axonal transport of a membrane compartment containing beta-secretase and presenilin-1 requires APP

Caspase-3-mediated cleavage of amyloid precursor protein and formation of amyloid Beta peptide in traumatic axonal injury.

Ischaemic brain damage is still common in fatal non-missile head injury.

Beta amyloid protein deposition in the brain after severe head injury: implications for the pathogenesis of Alzheimer's disease.

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185-192

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scholarly article

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10.1016/J.EXPNEUROL.2007.06.018

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2

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208

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John Q. Trojanowski

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2007-07-10T00:00:00Z

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6013289

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17826768

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neurodegeneration

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3979356

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