Breaking tolerance to double stranded DNA, nucleosome, and other nuclear antigens is not required for the pathogenesis of lupus glomerulonephritis. - Wikidata - awgldk - TriplyDB

Breaking tolerance to double stranded DNA, nucleosome, and other nuclear antigens is not required for the pathogenesis of lupus glomerulonephritis.

Breaking tolerance to double stranded DNA, nucleosome, and other nuclear antigens is not required for the pathogenesis of lupus glomerulonephritis.

http://www.wikidata.org/entity/Q30312212

about

Role of Natural Autoantibodies and Natural IgM Anti-Leucocyte Autoantibodies in Health and Disease Anti-dsDNA Antibodies are one of the many autoantibodies in systemic lupus erythematosus Modelling clinical systemic lupus erythematosus: similarities, differences and success stories The Role of Anti-DNA Antibodies in the Development of Lupus Nephritis: A Complementary, or Alternative, Viewpoint?Podocyte Activation of NLRP3 Inflammasomes Contributes to the Development of Proteinuria in Lupus Nephritis.Pathogenesis of proliferative lupus nephritis from a historical and personal perspective.A Central Role for HLA-DR3 in Anti-Smith Antibody Responses and Glomerulonephritis in a Transgenic Mouse Model of Spontaneous Lupus Genetics of systemic lupus erythematosus: immune responses and end organ resistance to damage.Interferon alpha on NZM2328.Lc1R27: enhancing autoimmunity and immune complex-mediated glomerulonephritis without end stage renal failure Cgnz1 allele confers kidney resistance to damage preventing progression of immune complex-mediated acute lupus glomerulonephritis.Autoimmunity, end organ damage, and the origin of autoantibodies and autoreactive T cells in systemic lupus erythematosus.HLA-DR3 restricted T cell epitope mimicry in induction of autoimmune response to lupus-associated antigen SmD Pathogenesis of systemic lupus erythematosus revisited 2011: end organ resistance to damage, autoantibody initiation and diversification, and HLA-DR.Deficiency of a transcriptional regulator, inhibitor of differentiation 3, induces glomerulonephritis in apolipoprotein E-deficient mice: a model linking hyperlipidemia and renal disease.Novel therapeutic approaches to lupus glomerulonephritis: translating animal models to clinical practice.Lupus glomerulonephritis revisited 2004: autoimmunity and end-organ damage.Pathogenesis of kidney disease in systemic lupus erythematosus.Genetic complementation results in augmented autoantibody responses to lupus-associated antigens.Anti-alpha8 integrin immunoliposomes in glomeruli of lupus-susceptible mice: a novel system for delivery of therapeutic agents to the renal glomerulus in systemic lupus erythematosus.Inflammatory stimuli accelerate Sjögren's syndrome-like disease in (NZB x NZW)F1 mice.Genetic approach to study lupus glomerulonephritis.Glomerular autoimmune multicomponents of human lupus nephritis in vivo: α-enolase and annexin AI.Recent advances in the understanding of renal inflammation and fibrosis in lupus nephritis.Identification of autoantibody clusters that best predict lupus disease activity using glomerular proteome arrays.Peptides and peptidomimetics as immunomodulators.Murine models of systemic lupus erythematosus Therapeutic impact of the ethyl acetate extract of Tripterygium wilfordii Hook F on nephritis in NZB/W F1 mice Generation of a unique small molecule peptidomimetic that neutralizes lupus autoantibody activity.Cutting edge: Calcium/Calmodulin-dependent protein kinase type IV is essential for mesangial cell proliferation and lupus nephritis.Podocyte-associated mRNA profiles in kidney tissue and in urine of patients with active lupus nephritis.Lupus nephritis: an overview of recent findings.Genetics of lupus nephritis.Changing the concepts of immune-mediated glomerular diseases through proteomics.SLE: translating lessons from model systems to human disease Epidermal injury promotes nephritis flare in lupus-prone mice.Toll-like receptor 9 controls anti-DNA autoantibody production in murine lupus The natural killer T cell ligand alpha-galactosylceramide prevents or promotes pristane-induced lupus in mice Blockade of CD354 (TREM-1) Ameliorates Anti-GBM-Induced Nephritis Genetics of clinical expression in SLE.Regulatory B cells (B10 cells) have a suppressive role in murine lupus: CD19 and B10 cell deficiency exacerbates systemic autoimmunity.

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P2860

Breaking tolerance to double stranded DNA, nucleosome, and other nuclear antigens is not required for the pathogenesis of lupus glomerulonephritis.

http://www.wikidata.org/entity/Q30312212

description

2004 nî lūn-bûn

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2004 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2004 թվականի հունվարին հրատարակված գիտական հոդված

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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name

Breaking tolerance to double s ...... s of lupus glomerulonephritis.

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Breaking tolerance to double s ...... s of lupus glomerulonephritis.

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type

label

Breaking tolerance to double s ...... s of lupus glomerulonephritis.

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Breaking tolerance to double s ...... s of lupus glomerulonephritis.

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prefLabel

Breaking tolerance to double s ...... s of lupus glomerulonephritis.

@ast

Breaking tolerance to double s ...... s of lupus glomerulonephritis.

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Journal of Experimental Medicine

P1476

Breaking tolerance to double s ...... s of lupus glomerulonephritis.

@en

P2093

Chao Jiang

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Felicia Gaskin

http://www.w3.org/2001/XMLSchema#string

Harini Bagavant

http://www.w3.org/2001/XMLSchema#string

Kenneth S K Tung

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Marcia McDuffie

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Samuel T Waters

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Umesh S Deshmukh

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P2860

Theoretical and empirical issues for marker-assisted breeding of congenic mouse strains

The 1982 revised criteria for the classification of systemic lupus erythematosus

Updating the American College of Rheumatology revised criteria for the classification of systemic lupus erythematosus

NZM2328: a new mouse model of systemic lupus erythematosus with unique genetic susceptibility loci.

Immune responses to Ro60 and its peptides in mice. I. The nature of the immunogen and endogenous autoantigen determine the specificities of the induced autoantibodies

Human rheumatoid factors with restrictive specificity for rabbit immunoglobulin G: auto- and multi-reactivity, diverse VH gene segment usage and preferential usage of V lambda IIIb.

Lupus anti-DNA autoantibodies cross-react with a glomerular structural protein: a case for tissue injury by molecular mimicry.

Genesis and evolution of antichromatin autoantibodies in murine lupus implicates T-dependent immunization with self antigen.

The HIV-1 regulatory proteins Tat and Rev are frequently targeted by cytotoxic T lymphocytes derived from HIV-1-infected individuals

The major murine systemic lupus erythematosus susceptibility locus, Sle1, is a cluster of functionally related genes.

P304

255-264

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10.1084/JEM.20031519

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2

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199

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2004-01-12T00:00:00Z

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8922072

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14718514

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glomerulonephritis

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2211766

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