Viral replication rate regulates clinical outcome and CD8 T cell responses during highly pathogenic H5N1 influenza virus infection in mice
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An Ultrasensitive Mechanism Regulates Influenza Virus-Induced InflammationTreating the host response to emerging virus diseases: lessons learned from sepsis, pneumonia, influenza and Ebola.Virulence attenuation during an influenza A/H5N1 pandemicA novel genetic locus linked to pro-inflammatory cytokines after virulent H5N1 virus infection in mice.Residues in the PB2 and PA genes contribute to the pathogenicity of avian H7N3 influenza A virus in DBA/2 mice.Sensitization with vaccinia virus encoding H5N1 hemagglutinin restores immune potential against H5N1 influenza virus.The potential for respiratory droplet-transmissible A/H5N1 influenza virus to evolve in a mammalian host.Oligomeric recombinant H5 HA1 vaccine produced in bacteria protects ferrets from homologous and heterologous wild-type H5N1 influenza challenge and controls viral loads better than subunit H5N1 vaccine by eliciting high-affinity antibodies.The controversy over H5N1 transmissibility research: an opportunity to define a practical response to a global threatH5N1 pathogenesis studies in mammalian models.Postinfection A77-1726 treatment improves cardiopulmonary function in H1N1 influenza-infected miceLack of PD-L1 expression by iNKT cells improves the course of influenza A infection.Specific mutations in H5N1 mainly impact the magnitude and velocity of the host response in mice.The virulence of 1997 H5N1 influenza viruses in the mouse model is increased by correcting a defect in their NS1 proteins.H5N1 influenza virus pathogenesis in genetically diverse mice is mediated at the level of viral load.Depressed Hypoxic and Hypercapnic Ventilatory Responses at Early Stage of Lethal Avian Influenza A Virus Infection in Mice.Infection of the upper respiratory tract with seasonal influenza A(H3N2) virus induces protective immunity in ferrets against infection with A(H1N1)pdm09 virus after intranasal, but not intratracheal, inoculation.The universal epitope of influenza A viral neuraminidase fundamentally contributes to enzyme activity and viral replicationPost-exposure treatment with whole inactivated H5N1 avian influenza virus protects against lethal homologous virus infection in mice.Moving H5N1 studies into the era of systems biology.A novel 2006 Indian outbreak strain of Chikungunya virus exhibits different pattern of infection as compared to prototype strain.Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.Uncompromised NK cell activation is essential for virus-specific CTL activity during acute influenza virus infection.MERS-CoV and H5N1 influenza virus antagonize antigen presentation by altering the epigenetic landscape.
P2860
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P2860
Viral replication rate regulates clinical outcome and CD8 T cell responses during highly pathogenic H5N1 influenza virus infection in mice
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Viral replication rate regulat ...... luenza virus infection in mice
@ast
Viral replication rate regulat ...... luenza virus infection in mice
@en
type
label
Viral replication rate regulat ...... luenza virus infection in mice
@ast
Viral replication rate regulat ...... luenza virus infection in mice
@en
prefLabel
Viral replication rate regulat ...... luenza virus infection in mice
@ast
Viral replication rate regulat ...... luenza virus infection in mice
@en
P2093
P2860
P50
P1433
P1476
Viral replication rate regulat ...... luenza virus infection in mice
@en
P2093
Kyoko Shinya
Masato Hatta
Richard R Dubielzig
Sean C Proll
Yasuko Hatta
P2860
P304
P356
10.1371/JOURNAL.PPAT.1001139
P577
2010-10-07T00:00:00Z