Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange. - Wikidata - awgldk - TriplyDB

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

http://www.wikidata.org/entity/Q30427998

about

Regulation of L-type calcium channel by phospholemman in cardiac myocytes.Phospholemman deficiency in postinfarct hearts: enhanced contractility but increased mortality.Methylene blue counteracts H2S toxicity-induced cardiac depression by restoring L-type Ca channel activity A splice variant of the human ion channel TRPM2 modulates neuroblastoma tumor growth through hypoxia-inducible factor (HIF)-1/2α.Ca²⁺ entry via Trpm2 is essential for cardiac myocyte bioenergetics maintenance.Targeting cardiomyocyte Ca2+ homeostasis in heart failure.Constitutive overexpression of phosphomimetic phospholemman S68E mutant results in arrhythmias, early mortality, and heart failure: potential involvement of Na+/Ca2+ exchanger.Intracellular trafficking of FXYD1 (phospholemman) and FXYD7 proteins in Xenopus oocytes and mammalian cells.Induced overexpression of Na(+)/Ca(2+) exchanger does not aggravate myocardial dysfunction induced by transverse aortic constriction.BAG3 regulates contractility and Ca(2+) homeostasis in adult mouse ventricular myocytes The second member of transient receptor potential-melastatin channel family protects hearts from ischemia-reperfusion injury.Cardiac Dysfunction in HIV-1 Transgenic Mouse: Role of Stress and BAG3 β-arrestin-biased signaling through the β2-adrenergic receptor promotes cardiomyocyte contraction.Adeno-associated Virus Serotype 9 - Driven Expression of BAG3 Improves Left Ventricular Function in Murine Hearts with Left Ventricular Dysfunction Secondary to a Myocardial Infarction.TRPM2 channels protect against cardiac ischemia-reperfusion injury: role of mitochondria.Induced overexpression of phospholemman S68E mutant improves cardiac contractility and mortality after ischemia-reperfusion.Depletion of the Human Ion Channel TRPM2 in Neuroblastoma Demonstrates Its Key Role in Cell Survival through Modulation of Mitochondrial Reactive Oxygen Species and Bioenergetics.β-adrenergic receptor-mediated cardiac contractility is inhibited via vasopressin type 1A-receptor-dependent signaling.Update on innovative initiatives for the American Journal of Physiology-Heart and Circulatory Physiology.How does pressure overload cause cardiac hypertrophy and dysfunction? High-ouabain affinity cardiac Na+ pumps are crucial.Pivotal role of α2 Na+ pumps and their high affinity ouabain binding site in cardiovascular health and disease.Haplo-insufficiency of Bcl2-associated Athanogene 3 in Mice Results in Progressive Left Ventricular Dysfunction, β-Adrenergic Insensitivity and Increased Apoptosis.Methylene blue counteracts cyanide cardiotoxicity: cellular mechanisms.Methylene Blue Counteracts H2S-Induced Cardiac Ion Channel Dysfunction and ATP Reduction.

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Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

http://www.wikidata.org/entity/Q30427998

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

@ast

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

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type

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Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

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Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

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prefLabel

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

@ast

Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

@en

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AJPHEART.00894.2010

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American Journal of Physiology - Heart and Circulatory Physiology

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Regulation of in vivo cardiac contractility by phospholemman: role of Na+/Ca2+ exchange.

@en

P2093

Amy L Tucker

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Arthur M Feldman

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Jianliang Song

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Joseph Rabinowitz

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Joseph Y Cheung

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Jufang Wang

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Walter J Koch

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Xue-Qian Zhang

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P2860

The FXYD gene family of small ion transport regulators or channels: cDNA sequence, protein signature sequence, and expression

Cardiac excitation-contraction coupling

Stable myocardial-specific AAV6-S100A1 gene therapy results in chronic functional heart failure rescue

Hypertrophy, increased ejection fraction, and reduced Na-K-ATPase activity in phospholemman-deficient mice

Effects of phospholemman downregulation on contractility and [Ca(2+)]i transients in adult rat cardiac myocytes

Identification of an endogenous inhibitor of the cardiac Na+/Ca2+ exchanger, phospholemman

Phospholemman and beta-adrenergic stimulation in the heart.

Phospholemman-mediated activation of Na/K-ATPase limits [Na]i and inotropic state during beta-adrenergic stimulation in mouse ventricular myocytes.

Regulation of cardiac myocyte contractility by phospholemman: Na+/Ca2+ exchange versus Na+ -K+ -ATPase

Altered contractility and [Ca2+]i homeostasis in phospholemman-deficient murine myocytes: role of Na+/Ca2+ exchange

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