Ionizing radiation induces prostate cancer neuroendocrine differentiation through interplay of CREB and ATF2: implications for disease progression. - Wikidata - awgldk - TriplyDB

Ionizing radiation induces prostate cancer neuroendocrine differentiation through interplay of CREB and ATF2: implications for disease progression.

Ionizing radiation induces prostate cancer neuroendocrine differentiation through interplay of CREB and ATF2: implications for disease progression.

http://www.wikidata.org/entity/Q30432236

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Wnt-11 promotes neuroendocrine-like differentiation, survival and migration of prostate cancer cells PKCε promotes oncogenic functions of ATF2 in the nucleus while blocking its apoptotic function at mitochondria Siah2-dependent concerted activity of HIF and FoxA2 regulates formation of neuroendocrine phenotype and neuroendocrine prostate tumors Neuroendocrine differentiation in prostate cancer Mechanistic Insights into Molecular Targeting and Combined Modality Therapy for Aggressive, Localized Prostate Cancer Neuroendocrine differentiation in prostate cancer: a mechanism of radioresistance and treatment failure The many faces of neuroendocrine differentiation in prostate cancer progression Control of CREB expression in tumors: from molecular mechanisms and signal transduction pathways to therapeutic target β-Adrenergic Receptor Signaling in Prostate Cancer Prognostic role of neuroendocrine differentiation in prostate cancer, putting together the pieces of the puzzle.Regulation of Cancer Cell Responsiveness to Ionizing Radiation Treatment by Cyclic AMP Response Element Binding Nuclear Transcription Factor.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Activating transcription factor 2 (ATF2) controls tolfenamic acid-induced ATF3 expression via MAP kinase pathways.Targeting CREB for cancer therapy: friend or foe A Role for OCT4 in Tumor Initiation of Drug-Resistant Prostate Cancer Cells Functional cyclic AMP response element in the breast cancer resistance protein (BCRP/ABCG2) promoter modulates epidermal growth factor receptor pathway- or androgen withdrawal-mediated BCRP/ABCG2 transcription in human cancer cells.Prostate cancer and neuroendocrine differentiation: more neuronal, less endocrine?Bim and VDAC1 are hierarchically essential for mitochondrial ATF2 mediated cell death Ionizing radiation induces neuroendocrine differentiation of prostate cancer cells in vitro, in vivo and in prostate cancer patients Critical role of N-terminal end-localized nuclear export signal in regulation of activating transcription factor 2 (ATF2) subcellular localization and transcriptional activity.GSK-3α Is a Novel Target of CREB and CREB-GSK-3α Signaling Participates in Cell Viability in Lung Cancer.ATF2 - at the crossroad of nuclear and cytosolic functions.Co-clustering phenome-genome for phenotype classification and disease gene discovery.Mitochondrial ATF2 translocation contributes to apoptosis induction and BRAF inhibitor resistance in melanoma through the interaction of Bim with VDAC1.Fractionated radiation exposure amplifies the radioresistant nature of prostate cancer cells.Celastrol potentiates radiotherapy by impairment of DNA damage processing in human prostate cancer.GRK3 is a direct target of CREB activation and regulates neuroendocrine differentiation of prostate cancer cells.Hypoxia-mediated alterations and their role in the HER-2/neuregulated CREB status and localization.Redox-mediated and ionizing-radiation-induced inflammatory mediators in prostate cancer development and treatment.Exploring the FGFR3-related oncogenic mechanism in bladder cancer using bioinformatics strategy.The Siah2-HIF-FoxA2 axis in prostate cancer – new markers and therapeutic opportunities.Small cell carcinoma of the prostate after high-dose-rate brachytherapy for low-risk prostatic adenocarcinoma.Leading causes of castration-resistant prostate cancer.Neuroendocrine tumours in rare sites: differences in nomenclature and diagnostics-a rare and ubiquitous histotype.The E3 ubiquitin ligase CHIP mediates ubiquitination and proteasomal degradation of PRMT5.Emerging trends in the evaluation and management of small cell prostate cancer: a clinical and molecular perspective.Transcriptional activation of PRMT5 by NF-Y is required for cell growth and negatively regulated by the PKC/c-Fos signaling in prostate cancer cells.The role of high cell density in the promotion of neuroendocrine transdifferentiation of prostate cancer cells.Cross modulation between the androgen receptor axis and protocadherin-PC in mediating neuroendocrine transdifferentiation and therapeutic resistance of prostate cancer.ATF2, a paradigm of the multifaceted regulation of transcription factors in biology and disease.

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P2860

Ionizing radiation induces prostate cancer neuroendocrine differentiation through interplay of CREB and ATF2: implications for disease progression.

http://www.wikidata.org/entity/Q30432236

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2008 nî lūn-bûn

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2008 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2008 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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0008-5472.CAN-08-2229

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Cancer Research

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Ionizing radiation induces pro ...... tions for disease progression.

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Chang-Deng Hu

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Evan T Keller

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Han Liu

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Jiaoti Huang

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Liang Cheng

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Sarah J Parsons

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Xuehong Deng

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P2860

Transcription factor ATF cDNA clones: an extensive family of leucine zipper proteins able to selectively form DNA-binding heterodimers

Leucine zipper structure of the protein CRE-BP1 binding to the cyclic AMP response element in brain

Mutual regulation of c-Jun and ATF2 by transcriptional activation and subcellular localization

Akt-and CREB-mediated prostate cancer cell proliferation inhibition by Nexrutine, a Phellodendron amurense extract

AP-1--Introductory remarks

Transcriptional regulation by the phosphorylation-dependent factor CREB

Leptomycin B inhibition of signal-mediated nuclear export by direct binding to CRM1

Changes in androgen receptor nongenotropic signaling correlate with transition of LNCaP cells to androgen independence

The role of protein kinase A pathway and cAMP responsive element-binding protein in androgen receptor-mediated transcription at the prostate-specific antigen locus

CREB: a stimulus-induced transcription factor activated by a diverse array of extracellular signals

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10.1158/0008-5472.CAN-08-2229

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23

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68

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23560076

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19047143

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3100895

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