Neonatal exposure to bisphenol a alters reproductive parameters and gonadotropin releasing hormone signaling in female rats
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Neonatal bisphenol-a exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neuronsBisphenol A Interaction With Brain Development and FunctionsPolycystic ovary syndrome: do endocrine-disrupting chemicals play a role?Bisphenol A and the female reproductive tract: an overview of recent laboratory evidence and epidemiological studiesEDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting ChemicalsEpigenetic effects of endocrine-disrupting chemicals on female reproduction: an ovarian perspectiveEvidence for bisphenol A-induced female infertility: a review (2007-2016)Bisphenol a and reproductive health: update of experimental and human evidence, 2007-2013Chronic estradiol-17β exposure suppresses hypothalamic norepinephrine release and the steroid-induced luteinizing hormone surge: role of nitration of tyrosine hydroxylaseGonadotropin-releasing hormone pulse sensitivity of follicle-stimulating hormone-beta gene is mediated by differential expression of positive regulatory activator protein 1 factors and corepressors SKIL and TGIF1.Neonatal exposure to bisphenol a and reproductive and endocrine alterations resembling the polycystic ovarian syndrome in adult rats.Developmental programming: impact of fetal exposure to endocrine-disrupting chemicals on gonadotropin-releasing hormone and estrogen receptor mRNA in sheep hypothalamus.Placental transfer of conjugated bisphenol A and subsequent reactivation in the rat fetus.Industrial endocrine disruptors and polycystic ovary syndrome.Potential protective effect of Tualang honey on BPA-induced ovarian toxicity in prepubertal rat.Bisphenol A, benzophenone-type ultraviolet filters, and phthalates in relation to uterine leiomyoma.The effects of in utero bisphenol A exposure on reproductive capacity in several generations of miceEndocrine disrupters: a review of some sources, effects, and mechanisms of actions on behaviour and neuroendocrine systems.Endocrine disrupting chemicals: Multiple effects on testicular signaling and spermatogenesis.Using bisphenol-a to study the onset of polycystic ovarian syndrome.Early programing of uterine tissue by bisphenol A: Critical evaluation of evidence from animal exposure studies.Disrupted organization of RFamide pathways in the hypothalamus is associated with advanced puberty in female rats neonatally exposed to bisphenol A.Prenatal exposure to low doses of bisphenol A increases pituitary proliferation and gonadotroph number in female mice offspring at birth.Icam5 Expression Exhibits Sex Differences in the Neonatal Pituitary and Is Regulated by Estradiol and Bisphenol ADevelopmental programming: impact of prenatal exposure to bisphenol-A and methoxychlor on steroid feedbacks in sheep.Mixtures of xenoestrogens disrupt estradiol-induced non-genomic signaling and downstream functions in pituitary cells.Bisphenol A exposure during adulthood causes augmentation of follicular atresia and luteal regression by decreasing 17β-estradiol synthesis via downregulation of aromatase in rat ovary.BPA Directly Decreases GnRH Neuronal Activity via Noncanonical PathwayRegulation of arcuate genes by developmental exposures to endocrine-disrupting compounds in female rats.Effects of maternal dietary exposure to cadmium during pregnancy on mammary cancer risk among female offspring.Stereological study on the effect of vitamin C in preventing the adverse effects of bisphenol A on rat ovary.Human infertility: are endocrine disruptors to blame?Environmental estrogens differentially engage the histone methyltransferase EZH2 to increase risk of uterine tumorigenesis.Relationships between urinary biomarkers of phytoestrogens, phthalates, phenols, and pubertal stages in girls.Urinary bisphenol a levels in girls with idiopathic central precocious pubertyBisphenol A and children's health.Early neuroendocrine disruption in hypothalamus and hippocampus: developmental effects including female sexual maturation and implications for endocrine disrupting chemical screening.Lack of functional GABA(B) receptors alters GnRH physiology and sexual dimorphic expression of GnRH and GAD-67 in the brain.The effect of maternal exposure to endocrine disrupting chemicals on fetal and neonatal development: A review on the major concerns.The Effect of Bisphenol A on Puberty: A Critical Review of the Medical Literature.
P2860
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P2860
Neonatal exposure to bisphenol a alters reproductive parameters and gonadotropin releasing hormone signaling in female rats
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Neonatal exposure to bisphenol ...... rmone signaling in female rats
@ast
Neonatal exposure to bisphenol ...... rmone signaling in female rats
@en
type
label
Neonatal exposure to bisphenol ...... rmone signaling in female rats
@ast
Neonatal exposure to bisphenol ...... rmone signaling in female rats
@en
prefLabel
Neonatal exposure to bisphenol ...... rmone signaling in female rats
@ast
Neonatal exposure to bisphenol ...... rmone signaling in female rats
@en
P2093
P2860
P356
P1476
Neonatal exposure to bisphenol ...... rmone signaling in female rats
@en
P2093
Carlos Libertun
Maria Bianchi
Marina Fernández
Victoria Lux-Lantos
P2860
P304
P356
10.1289/EHP.0800267
P577
2009-01-07T00:00:00Z