Upregulation of the T-type calcium current in small rat sensory neurons after chronic constrictive injury of the sciatic nerve - Wikidata - awgldk - TriplyDB

Upregulation of the T-type calcium current in small rat sensory neurons after chronic constrictive injury of the sciatic nerve

Upregulation of the T-type calcium current in small rat sensory neurons after chronic constrictive injury of the sciatic nerve

http://www.wikidata.org/entity/Q30439844

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The Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic Potential Food-Derived Natural Compounds for Pain Relief in Neuropathic Pain T-type calcium channels contribute to colonic hypersensitivity in a rat model of irritable bowel syndrome Silencing the α2 subunit of γ-aminobutyric acid type A receptors in rat dorsal root ganglia reveals its major role in antinociception posttraumatic nerve injury.Immunohistological demonstration of CaV3.2 T-type voltage-gated calcium channel expression in soma of dorsal root ganglion neurons and peripheral axons of rat and mouse.T-type calcium channel blockers as neuroprotective agents.T-type voltage-gated calcium channels as targets for the development of novel pain therapies.Molecular mechanisms of lipoic acid modulation of T-type calcium channels in pain pathway.A modeling study of T-type Ca2+ channel gating and modulation by L-cysteine in rat nociceptors.In vivo silencing of the Ca(V)3.2 T-type calcium channels in sensory neurons alleviates hyperalgesia in rats with streptozocin-induced diabetic neuropathy.Role of voltage-gated calcium channels in ascending pain pathways Neuropathic pain: role for presynaptic T-type channels in nociceptive signaling.CRMP-2 peptide mediated decrease of high and low voltage-activated calcium channels, attenuation of nociceptor excitability, and anti-nociception in a model of AIDS therapy-induced painful peripheral neuropathy Characterization of novel cannabinoid based T-type calcium channel blockers with analgesic effects.1,4-Dihydropyridine derivatives with T-type calcium channel blocking activity attenuate inflammatory and neuropathic pain.Identification of CaV channel types expressed in muscle afferent neurons.Regulation of voltage-gated Ca(2+) currents by Ca(2+)/calmodulin-dependent protein kinase II in resting sensory neurons.Neurotoxicity induced by bupivacaine via T-type calcium channels in SH-SY5Y cells NMP-7 inhibits chronic inflammatory and neuropathic pain via block of Cav3.2 T-type calcium channels and activation of CB2 receptors.Alleviation of neuropathic pain by regulating T-type calcium channels in rat anterior cingulate cortex Small organic molecule disruptors of Cav3.2 - USP5 interactions reverse inflammatory and neuropathic pain.Roscovitine inhibits CaV3.1 (T-type) channels by preferentially affecting closed-state inactivation.Up-regulation of platelet-activating factor synthases and its receptor in spinal cord contribute to development of neuropathic pain following peripheral nerve injury.Effects of familial hemiplegic migraine type 1 mutation T666M on voltage-gated calcium channel activities in trigeminal ganglion neurons Topical application of disodium isostearyl 2-O-L-ascorbyl phosphate, an amphiphilic ascorbic acid derivative, reduces neuropathic hyperalgesia in rats.Inflammatory mediator bradykinin increases population of sensory neurons expressing functional T-type Ca(2+) channels.Antinociceptive effects of amiloride and benzamil in neuropathic pain model rats Pharmacological Inhibition of Voltage-gated Ca(2+) Channels for Chronic Pain Relief.T-type calcium channels are regulated by hypoxia/reoxygenation in ventricular myocytes.Sustained relief of neuropathic pain by AAV-targeted expression of CBD3 peptide in rat dorsal root ganglion.Targeting voltage-gated calcium channels for neuropathic pain management.Nociceptive neurons differentially express fast and slow T-type Ca²⁺ currents in different types of diabetic neuropathy.Calcium channel functions in pain processing Ion channel blockers for the treatment of neuropathic pain.Pharmacological treatment of diabetic neuropathic pain.Targeting voltage-gated calcium channels for the treatment of neuropathic pain: a review of drug development.The design and discovery of T-type calcium channel inhibitors for the treatment of central nervous system disorders.Calcium-permeable ion channels in pain signaling.T-type calcium channels in chronic pain: mouse models and specific blockers.A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain.

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P2860

Upregulation of the T-type calcium current in small rat sensory neurons after chronic constrictive injury of the sciatic nerve

http://www.wikidata.org/entity/Q30439844

description

2008 nî lūn-bûn

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2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2008 թվականի ապրիլին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Upregulation of the T-type cal ...... ve injury of the sciatic nerve

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Upregulation of the T-type cal ...... ve injury of the sciatic nerve

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type

label

Upregulation of the T-type cal ...... ve injury of the sciatic nerve

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Upregulation of the T-type cal ...... ve injury of the sciatic nerve

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Upregulation of the T-type cal ...... ve injury of the sciatic nerve

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Upregulation of the T-type cal ...... ve injury of the sciatic nerve

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Journal of Neurophysiology

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Upregulation of the T-type cal ...... ve injury of the sciatic nerve

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Ajit K Naik

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Michael T Nelson

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Miljen M Jagodic

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Pavle M Joksovic

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Peihan Su

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Slobodan M Todorovic

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Sriyani Pathirathna

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WooYong Lee

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P2860

Molecular physiology of low-voltage-activated t-type calcium channels

Attenuated pain responses in mice lacking Ca(V)3.2 T-type channels

Differential distribution of three members of a gene family encoding low voltage-activated (T-type) calcium channels.

Redox modulation of peripheral T-type Ca2+ channels in vivo: alteration of nerve injury-induced thermal hyperalgesia.

5alpha-reduced neuroactive steroids alleviate thermal and mechanical hyperalgesia in rats with neuropathic pain.

The endogenous redox agent L-cysteine induces T-type Ca2+ channel-dependent sensitization of a novel subpopulation of rat peripheral nociceptors.

The role of peripheral T-type calcium channels in pain transmission.

Cell-specific alterations of T-type calcium current in painful diabetic neuropathy enhance excitability of sensory neurons.

Reducing agents sensitize C-type nociceptors by relieving high-affinity zinc inhibition of T-type calcium channels.

Nickel block of three cloned T-type calcium channels: low concentrations selectively block alpha1H.

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3151-3156

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scholarly article

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10.1152/JN.01031.2007

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P433

6

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99

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Vesna Jevtovic-Todorovic

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2008-04-16T00:00:00Z

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5434301

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18417624

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2667888

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