Androgen-independent growth and tumorigenesis of prostate cancer cells are enhanced by the presence of PKA-differentiated neuroendocrine cells. - Wikidata - awgldk - TriplyDB

Androgen-independent growth and tumorigenesis of prostate cancer cells are enhanced by the presence of PKA-differentiated neuroendocrine cells.

Androgen-independent growth and tumorigenesis of prostate cancer cells are enhanced by the presence of PKA-differentiated neuroendocrine cells.

http://www.wikidata.org/entity/Q30443433

about

Wnt-11 promotes neuroendocrine-like differentiation, survival and migration of prostate cancer cells Neuroendocrine differentiation in prostate cancer Neuroendocrine differentiation in prostate cancer: a mechanism of radioresistance and treatment failure Autophagy pathway is required for IL-6 induced neuroendocrine differentiation and chemoresistance of prostate cancer LNCaP cells Neuroendocrine-derived peptides promote prostate cancer cell survival through activation of IGF-1R signaling.Ionizing radiation induces prostate cancer neuroendocrine differentiation through interplay of CREB and ATF2: implications for disease progression.The neuroendocrine-derived peptide parathyroid hormone-related protein promotes prostate cancer cell growth by stabilizing the androgen receptor.Neuroendocrine Cells of the Prostate Derive from the Neural Crest.Prognostic role of neuroendocrine differentiation in prostate cancer, putting together the pieces of the puzzle.The nuclear factor-kappaB pathway controls the progression of prostate cancer to androgen-independent growth.Targeting CREB for cancer therapy: friend or foe Genomic analysis of fibrolamellar hepatocellular carcinoma.Prostate cancer and neuroendocrine differentiation: more neuronal, less endocrine?Ionizing radiation induces neuroendocrine differentiation of prostate cancer cells in vitro, in vivo and in prostate cancer patients Oncogenic activation of androgen receptor.GRK3 is a direct target of CREB activation and regulates neuroendocrine differentiation of prostate cancer cells.Anti-androgen enzalutamide enhances prostate cancer neuroendocrine (NE) differentiation via altering the infiltrated mast cells → androgen receptor (AR) → miRNA32 signals.Neuroendocrine differentiation of prostate cancer.Overexpressing PKIB in prostate cancer promotes its aggressiveness by linking between PKA and Akt pathways.Down-regulation of RE-1 silencing transcription factor (REST) in advanced prostate cancer by hypoxia-induced miR-106b~25.Protein kinase A-mediated phosphorylation of RhoA on serine 188 triggers the rapid induction of a neuroendocrine-like phenotype in prostate cancer epithelial cells.Amorphous silica nanoparticles induce malignant transformation and tumorigenesis of human lung epithelial cells via P53 signaling.Hyperactive mTOR induces neuroendocrine differentiation in prostate cancer cell with concurrent up-regulation of IRF1.Blockade of p53 by HIF-2α, but not HIF-1α, is involved in arsenite-induced malignant transformation of human bronchial epithelial cells.

P2860

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P2860

Androgen-independent growth and tumorigenesis of prostate cancer cells are enhanced by the presence of PKA-differentiated neuroendocrine cells.

http://www.wikidata.org/entity/Q30443433

description

2007 nî lūn-bûn

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2007 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2007 թվականի ապրիլին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Androgen-independent growth an ...... entiated neuroendocrine cells.

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Androgen-independent growth an ...... entiated neuroendocrine cells.

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Androgen-independent growth an ...... entiated neuroendocrine cells.

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Androgen-independent growth an ...... entiated neuroendocrine cells.

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Androgen-independent growth an ...... entiated neuroendocrine cells.

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Androgen-independent growth an ...... entiated neuroendocrine cells.

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0008-5472.CAN-06-2616

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Cancer Research

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Androgen-independent growth an ...... entiated neuroendocrine cells.

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P2093

Eli V Casarez

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George P Amorino

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Henry F Frierson

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Jodie B Palmer

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Michael E Cox

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Paul D Deeble

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Robert A Sikes

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Robert J Davidson

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Sarah J Parsons

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3663-3672

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scholarly article

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10.1158/0008-5472.CAN-06-2616

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8

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67

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2007-04-01T00:00:00Z

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17440078

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