Velocity differences between L- and P-selectin-dependent neutrophil rolling in venules of mouse cremaster muscle in vivo.
about
On, around, and through: neutrophil-endothelial interactions in innate immunityRAGE controls activation and anti-inflammatory signalling of protein CGlycosyltransferase-programmed stereosubstitution (GPS) to create HCELL: engineering a roadmap for cell migrationEndothelial CD47 promotes vascular endothelial-cadherin tyrosine phosphorylation and participates in T cell recruitment at sites of inflammation in vivoSevere reduction in leukocyte adhesion and monocyte extravasation in mice deficient in CC chemokine receptor 2.P-selectin glycoprotein ligand-1 mediates L-selectin-dependent leukocyte rolling in venules.PSGL-1 engagement by E-selectin signals through Src kinase Fgr and ITAM adapters DAP12 and FcR gamma to induce slow leukocyte rolling.Threshold levels of fluid shear promote leukocyte adhesion through selectins (CD62L,P,E)Transit time of leukocytes rolling through venules controls cytokine-induced inflammatory cell recruitment in vivoOptimal selectin-mediated rolling of leukocytes during inflammation in vivo requires intercellular adhesion molecule-1 expression.IL-17 and TNF-α sustain neutrophil recruitment during inflammation through synergistic effects on endothelial activation.Neutrophil adhesion and activation under flow.Biomechanics of leukocyte rollingCD44 and HCELL: preventing hematogenous metastasis at step 1.Multiple, targeted deficiencies in selectins reveal a predominant role for P-selectin in leukocyte recruitment.Engineering cellular trafficking via glycosyltransferase-programmed stereosubstitutionInactivation of heparan sulfate 2-O-sulfotransferase accentuates neutrophil infiltration during acute inflammation in mice.The cutaneous lymphocyte antigen is an essential component of the L-selectin ligand induced on human vascular endothelial cellsTargeted gene disruption demonstrates that P-selectin glycoprotein ligand 1 (PSGL-1) is required for P-selectin-mediated but not E-selectin-mediated neutrophil rolling and migrationEmerging understanding of roles for arterioles in inflammation.TNF-alpha activation of arterioles and venules alters distribution and levels of ICAM-1 and affects leukocyte-endothelial cell interactions.Fetal wound healing using a genetically modified murine model: the contribution of P-selectin.Leukocyte recruitment in the airways: an intravital microscopic study of rat tracheal microcirculation.Mechanisms of decreased leukocyte localization in the developing host.
P2860
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P2860
Velocity differences between L- and P-selectin-dependent neutrophil rolling in venules of mouse cremaster muscle in vivo.
description
1996 nî lūn-bûn
@nan
1996 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Velocity differences between L ...... ouse cremaster muscle in vivo.
@ast
Velocity differences between L ...... ouse cremaster muscle in vivo.
@en
type
label
Velocity differences between L ...... ouse cremaster muscle in vivo.
@ast
Velocity differences between L ...... ouse cremaster muscle in vivo.
@en
prefLabel
Velocity differences between L ...... ouse cremaster muscle in vivo.
@ast
Velocity differences between L ...... ouse cremaster muscle in vivo.
@en
P2093
P1476
Velocity differences between L ...... ouse cremaster muscle in vivo.
@en
P2093
P304
P356
10.1152/AJPHEART.1996.271.6.H2740
P407
P433
P577
1996-12-01T00:00:00Z