Hydrogen sulfide attenuates myocardial ischemia-reperfusion injury by preservation of mitochondrial function - Wikidata - awgldk - TriplyDB

Hydrogen sulfide attenuates myocardial ischemia-reperfusion injury by preservation of mitochondrial function

Hydrogen sulfide attenuates myocardial ischemia-reperfusion injury by preservation of mitochondrial function

http://www.wikidata.org/entity/Q30480176

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The mechanism of Intralipid®-mediated cardioprotection complex IV inhibition by the active metabolite, palmitoylcarnitine, generates reactive oxygen species and activates reperfusion injury salvage kinases Induction of VMAT-1 and TPH-1 expression induces vesicular accumulation of serotonin and protects cells and tissue from cooling/rewarming injury A new gaseous signaling molecule emerges: cardioprotective role of hydrogen sulfide "Oxygen Sensing" by Na,K-ATPase: These Miraculous Thiols Cysteine and hydrogen sulphide in the regulation of metabolism: insights from genetics and pharmacology Emergence of hydrogen sulfide as an endogenous gaseous signaling molecule in cardiovascular disease Hydrogen sulfide chemical biology: pathophysiological roles and detection The role of gasotransmitters NO, H2S and CO in myocardial ischaemia/reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioning Role of cystathionine γ-lyase/hydrogen sulfide pathway in cardiovascular disease: a novel therapeutic strategy?Hydrogen sulfide prodrugs-a review H2S during circulatory shock: some unresolved questions Renal adaptation during hibernation Signaling molecules: hydrogen sulfide and polysulfide Mitochondrial biogenesis: regulation by endogenous gases during inflammation and organ stress Hydrogen sulfide donor NaHS reduces organ injury in a rat model of pneumococcal pneumosepsis, associated with improved bio-energetic status Structure and Kinetic Analysis of H2S Production by Human Mercaptopyruvate Sulfurtransferase Relative contributions of cystathionine beta-synthase and gamma-cystathionase to H2S biogenesis via alternative trans-sulfuration reactions.Essential Roles of Natural Products and Gaseous Mediators on Neuronal Cell Death or Survival The Cardioprotective Effects of Hydrogen Sulfide in Heart Diseases: From Molecular Mechanisms to Therapeutic Potential The effect of certain conditions in the regulation of cystathionine γ-lyase by exogenous hydrogen sulfide in mammalian cells Cytoprotection by the modulation of mitochondrial electron transport chain: the emerging role of mitochondrial STAT3 Mitochondrial function in hypoxic ischemic injury and influence of aging H2S signals through protein S-sulfhydration.Cardioprotective effects of a novel hydrogen sulfide agent-controlled release formulation of S-propargyl-cysteine on heart failure rats and molecular mechanisms A H2S-Nampt dependent energetic circuit is critical to survival and cytoprotection from damage in cancer cells S-Propargyl-Cysteine, a Novel Hydrogen Sulfide Donor, Inhibits Inflammatory Hepcidin and Relieves Anemia of Inflammation by Inhibiting IL-6/STAT3 Pathway Comparative exploration of hydrogen sulfide and water transmembrane free energy surfaces via orthogonal space tempering free energy sampling Subcellular Energetics and Metabolism: Potential Therapeutic Applications.miR-761 regulates the mitochondrial network by targeting mitochondrial fission factor.Delivery of Hydrogen Sulfide by Ultrasound Targeted Microbubble Destruction Attenuates Myocardial Ischemia-reperfusion Injury H2S biogenesis by human cystathionine gamma-lyase leads to the novel sulfur metabolites lanthionine and homolanthionine and is responsive to the grade of hyperhomocysteinemia.Hydrogen sulfide-induced hypometabolism prevents renal ischemia/reperfusion injury Hydrogen sulfide improves survival after cardiac arrest and cardiopulmonary resuscitation via a nitric oxide synthase 3-dependent mechanism in mice.Hydrogen sulfide is an endogenous stimulator of angiogenesis.Hydrogen sulfide increases hypoxia-inducible factor-1 activity independently of von Hippel-Lindau tumor suppressor-1 in C. elegans.Redox biochemistry of hydrogen sulfide.Suspended animation inducer hydrogen sulfide is protective in an in vivo model of ventilator-induced lung injury Genetic and pharmacologic hydrogen sulfide therapy attenuates ischemia-induced heart failure in mice.Hydrogen sulfide (H2S) metabolism in mitochondria and its regulatory role in energy production.Assessment of H2S in vivo using the newly developed mitochondria-targeted mass spectrometry probe MitoA

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P2860

Hydrogen sulfide attenuates myocardial ischemia-reperfusion injury by preservation of mitochondrial function

http://www.wikidata.org/entity/Q30480176

description

2007 nî lūn-bûn

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2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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Chi-Wing Chow

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Csaba Szabo

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David J Lefer

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David W Kraus

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Jeannette E Doeller

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Levente Kiss

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Ling Tao

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Xiangying Jiao

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P2860

Hydrogen sulfide is an endogenous modulator of leukocyte-mediated inflammation

H2S induces a suspended animation-like state in mice

Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

Murine cystathionine gamma-lyase: complete cDNA and genomic sequences, promoter activity, tissue distribution and developmental expression

Mitochondrial membrane permeabilization in cell death

Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide

Pro-apoptotic effect of endogenous H2S on human aorta smooth muscle cells.

Mitochondrial dysfunction in cardiac disease: ischemia--reperfusion, aging, and heart failure.

Inhaled nitric oxide therapy in adults.

Inhibition of caspase 1 reduces human myocardial ischemic dysfunction via inhibition of IL-18 and IL-1beta

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15560-15565

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17878306

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reperfusion injury

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2000503

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