Arrhythmogenic consequences of intracellular calcium waves. - Wikidata - awgldk - TriplyDB

Arrhythmogenic consequences of intracellular calcium waves.

Arrhythmogenic consequences of intracellular calcium waves.

http://www.wikidata.org/entity/Q30490734

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Towards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium Channels Inherited dysfunction of sarcoplasmic reticulum Ca2+ handling and arrhythmogenesis Cell model of catecholaminergic polymorphic ventricular tachycardia reveals early and delayed afterdepolarizations MicroRNA-1 and -133 increase arrhythmogenesis in heart failure by dissociating phosphatase activity from RyR2 complex.Nonlinear and Stochastic Dynamics in the Heart The role of mitochondria for the regulation of cardiac alternans.The relationship between arrhythmogenesis and impaired contractility in heart failure: role of altered ryanodine receptor function.Carvedilol and its new analogs suppress arrhythmogenic store overload-induced Ca2+ release Novel Analysis Software for Detecting and Classifying Ca2+ Transient Abnormalities in Stem Cell-Derived Cardiomyocytes.Role of substrate and triggers in the genesis of cardiac alternans, from the myocyte to the whole heart: implications for therapy.Revisiting the ionic mechanisms of early afterdepolarizations in cardiomyocytes: predominant by Ca waves or Ca currents?Role of the transient outward potassium current in the genesis of early afterdepolarizations in cardiac cells.Enhanced sarcoplasmic reticulum Ca2+ leak and increased Na+-Ca2+ exchanger function underlie delayed afterdepolarizations in patients with chronic atrial fibrillation.Slow [Na]i Changes and Positive Feedback Between Membrane Potential and [Ca]i Underlie Intermittent Early Afterdepolarizations and Arrhythmias.Calcium alternans in cardiac myocytes: order from disorder.Non-β-blocking R-carvedilol enantiomer suppresses Ca2+ waves and stress-induced ventricular tachyarrhythmia without lowering heart rate or blood pressure.Nonlinear dynamics in cardiology.Aldehyde stress and up-regulation of Nrf2-mediated antioxidant systems accompany functional adaptations in cardiac mitochondria from mice fed n-3 polyunsaturated fatty acids.Novel carvedilol analogues that suppress store-overload-induced Ca2+ release A translational approach to probe the proarrhythmic potential of cardiac alternans: a reversible overture to arrhythmogenesis?Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytes.Alternans and arrhythmias: from cell to heart.Modeling calcium regulation of contraction, energetics, signaling, and transcription in the cardiac myocyte.Ca²⁺-induced delayed afterdepolarizations are triggered by dyadic subspace Ca2²⁺ affirming that increasing SERCA reduces aftercontractions.Functional Impact of Ryanodine Receptor Oxidation on Intracellular Calcium Regulation in the Heart.Regulation of sarcoplasmic reticulum Ca2+ release by serine-threonine phosphatases in the heart.Early afterdepolarizations and cardiac arrhythmias.Dynamics of spatiotemporal line defects and chaos control in complex excitable systems.Non-linear dynamics of cardiac alternans: subcellular to tissue-level mechanisms of arrhythmia.Feedback-control induced pattern formation in cardiac myocytes: a mathematical modeling study.Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca(2+) currents and promote proarrhythmic behavior in ventricular myocytes.Dual Vm/Ca imaging of premature ventricular contractions: bridging the gap of anatomical scales.Extinguishing intracellular calcium leak: a promising antiarrhythmic approach.Ranolazine is an Effective and Safe Treatment of Adults with Symptomatic Premature Ventricular Contractions due to Triggered Ectopy.Transverse tubular network structures in the genesis of intracellular calcium alternans and triggered activity in cardiac cells.A Precision Medicine Approach to the Rescue of Function on Malignant Calmodulinopathic Long-QT Syndrome.Age- and strain-related aberrant Ca2+ release is associated with sudden cardiac death in the ACTC E99K mouse model of hypertrophic cardiomyopathy.Involvement of mitochondrial permeability transition pore (mPTP) in cardiac arrhythmias: Evidence from cyclophilin D knockout mice.Influence of Amiodarone and Dronedarone on the Force-Interval Dependence of Rat Myocardium

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P2860

Arrhythmogenic consequences of intracellular calcium waves.

http://www.wikidata.org/entity/Q30490734

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Arrhythmogenic consequences of intracellular calcium waves.

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Arrhythmogenic consequences of intracellular calcium waves.

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type

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Arrhythmogenic consequences of intracellular calcium waves.

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Arrhythmogenic consequences of intracellular calcium waves.

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Arrhythmogenic consequences of intracellular calcium waves.

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Arrhythmogenic consequences of intracellular calcium waves.

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AJPHEART.00390.2009

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American Journal of Physiology - Heart and Circulatory Physiology

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Arrhythmogenic consequences of intracellular calcium waves.

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James N Weiss

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Lai-Hua Xie

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P2860

Efficacy assessment of meloxicam, a preferential cyclooxygenase-2 inhibitor, in acute coronary syndromes without ST-segment elevation: the Nonsteroidal Anti-Inflammatory Drugs in Unstable Angina Treatment-2 (NUT-2) pilot study

Pacing-induced heterogeneities in intracellular Ca2+ signaling, cardiac alternans, and ventricular arrhythmias in intact rat heart.

Intracellular Ca(2+) dynamics and the stability of ventricular tachycardia.

Turing instability mediated by voltage and calcium diffusion in paced cardiac cells

Local calcium gradients during excitation-contraction coupling and alternans in atrial myocytes.

Dynamic origin of spatially discordant alternans in cardiac tissue.

Role of calcium cycling versus restitution in the mechanism of repolarization alternans.

Arrhythmogenesis and contractile dysfunction in heart failure: Roles of sodium-calcium exchange, inward rectifier potassium current, and residual beta-adrenergic responsiveness.

Depressed ryanodine receptor activity increases variability and duration of the systolic Ca2+ transient in rat ventricular myocytes.

Subcellular Ca2+ alternans represents a novel mechanism for the generation of arrhythmogenic Ca2+ waves in cat atrial myocytes.

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10.1152/AJPHEART.00390.2009

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