The acute nociceptive signals induced by bradykinin in rat sensory neurons are mediated by inhibition of M-type K+ channels and activation of Ca2+-activated Cl- channels.
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Characterization of the oligomeric structure of the Ca(2+)-activated Cl- channel Ano1/TMEM16AVoltage- and calcium-dependent gating of TMEM16A/Ano1 chloride channels are physically coupled by the first intracellular loopPlasma kallikrein-kinin system and diabetic retinopathyProlactin receptor in regulation of neuronal excitability and channelsRedox-Dependent Modulation of T-Type Ca(2+) Channels in Sensory Neurons Contributes to Acute Anti-Nociceptive Effect of Substance PCellular functions of TMEM16/anoctaminCalmodulin regulation of TMEM16A and 16B Ca(2+)-activated chloride channelsFractionation of a herbal antidiarrheal medicine reveals eugenol as an inhibitor of Ca2+-Activated Cl- channel TMEM16AGPR39 is coupled to TMEM16A in intestinal fibroblast-like cellsInhibition of ANO1/TMEM16A Chloride Channel by Idebenone and Its Cytotoxicity to Cancer Cell LinesRNA-Seq Analysis of Human Trigeminal and Dorsal Root Ganglia with a Focus on ChemoreceptorsK(V)7/KCNQ channels are functionally expressed in oligodendrocyte progenitor cellsCalmodulin-dependent activation and inactivation of anoctamin calcium-gated chloride channelsEtiology of distinct membrane excitability in pre- and posthearing auditory neurons relies on activity of Cl- channel TMEM16AActivation of ATP-sensitive potassium channels antagonize nociceptive behavior and hyperexcitability of DRG neurons from ratsReactive oxygen species are second messengers of neurokinin signaling in peripheral sensory neuronsAnoctamin-1 Cl(-) channels in nociception: activation by an N-aroylaminothiazole and capsaicin and inhibition by T16A[inh]-A01Local GABAergic signaling within sensory ganglia controls peripheral nociceptive transmission.Acidic amino acids in the first intracellular loop contribute to voltage- and calcium- dependent gating of anoctamin1/TMEM16A.Peripheral KV7 channels regulate visceral sensory function in mouse and human colon.Activation of the Cl- channel ANO1 by localized calcium signals in nociceptive sensory neurons requires coupling with the IP3 receptorRole of the Ca2+ -activated Cl- channels bestrophin and anoctamin in epithelial cells.Anoctamins.International Union of Basic and Clinical Pharmacology. LXXXV: calcium-activated chloride channels.Transient receptor potential V1 regulates activation and modulation of transient receptor potential A1 by Ca2+Combined phosphoinositide and Ca2+ signals mediating receptor specificity toward neuronal Ca2+ channels.The role of potassium channel activation in celecoxib-induced analgesic action.Control of somatic membrane potential in nociceptive neurons and its implications for peripheral nociceptive transmission.Preassociated apocalmodulin mediates Ca2+-dependent sensitization of activation and inactivation of TMEM16A/16B Ca2+-gated Cl- channelsRedox and nitric oxide-mediated regulation of sensory neuron ion channel functionATP-sensitive potassium channels alleviate postoperative pain through JNK-dependent MCP-1 expression in spinal cordSensitization of neonatal rat lumbar motoneuron by the inflammatory pain mediator bradykininCharacteristics and molecular basis of celecoxib modulation on K(v)7 potassium channels.Kv7.5 is the primary Kv7 subunit expressed in C-fibers.Development of the Olfactory Epithelium and Nasal Glands in TMEM16A-/- and TMEM16A+/+ Mice.Activation of peripheral KCNQ channels relieves gout pain.An antinociceptive role for substance P in acid-induced chronic muscle pain.Role of anoctamin-1 and bestrophin-1 in spinal nerve ligation-induced neuropathic pain in rats.Anoctamin Calcium-Activated Chloride Channels May Modulate Inhibitory Transmission in the Cerebellar Cortex.17(R)-resolvin D1 specifically inhibits transient receptor potential ion channel vanilloid 3 leading to peripheral antinociception.
P2860
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P2860
The acute nociceptive signals induced by bradykinin in rat sensory neurons are mediated by inhibition of M-type K+ channels and activation of Ca2+-activated Cl- channels.
description
2010 nî lūn-bûn
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2010 թուականի Մարտին հրատարակուած գիտական յօդուած
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2010 թվականի մարտին հրատարակված գիտական հոդված
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2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@ast
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@en
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@en-gb
type
label
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@ast
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@en
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@en-gb
prefLabel
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@ast
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@en
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@en-gb
P2093
P2860
P50
P356
P1476
The acute nociceptive signals ...... f Ca2+-activated Cl- channels.
@en
P2093
Hailin Zhang
Xuan Zhang
P2860
P304
P356
10.1172/JCI41084
P407
P577
2010-03-24T00:00:00Z