Local GABAergic signaling within sensory ganglia controls peripheral nociceptive transmission. - Wikidata - awgldk - TriplyDB

Local GABAergic signaling within sensory ganglia controls peripheral nociceptive transmission.

Local GABAergic signaling within sensory ganglia controls peripheral nociceptive transmission.

http://www.wikidata.org/entity/Q30847299

about

Targeting dorsal root ganglia and primary sensory neurons for the treatment of chronic pain.Itchy channels and where to find them.Adult Mouse DRG Explant and Dissociated Cell Models to Investigate Neuroplasticity and Responses to Environmental Insults Including Viral Infection.

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Local GABAergic signaling within sensory ganglia controls peripheral nociceptive transmission.

http://www.wikidata.org/entity/Q30847299

description

2017 nî lūn-bûn

@nan

2017 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի ապրիլին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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name

Local GABAergic signaling with ...... eral nociceptive transmission.

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Journal of Clinical Investigation

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Bingcai Guan

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Caixue Wang

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David B Jaffe

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Ewa Jaworska

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Hailin Zhang

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Han Hao

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Ian Edwards

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Jinlong Qi

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Sha Huang

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Xiaona Du

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P2860

Evolving the lock to fit the key to create a family of G protein-coupled receptors potently activated by an inert ligand

The synaptic vesicle protein SV2 is complexed with an alpha5-containing laminin on the nerve terminal surface

Pain mechanisms: a new theory

Satellite glial cells in sensory ganglia: from form to function

Cellular and Molecular Mechanisms of Pain

Silencing the α2 subunit of γ-aminobutyric acid type A receptors in rat dorsal root ganglia reveals its major role in antinociception posttraumatic nerve injury.

GABAA receptor modulation in dorsal root ganglia in vivo affects chronic pain after nerve injury.

The acute nociceptive signals induced by bradykinin in rat sensory neurons are mediated by inhibition of M-type K+ channels and activation of Ca2+-activated Cl- channels.

Electrical excitability of the soma of sensory neurons is required for spike invasion of the soma, but not for through-conduction.

GABA blocks pathological but not acute TRPV1 pain signals.

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1741-1756

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scholarly article

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10.1172/JCI86812

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5

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127

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Rosmaliza Ramli

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18499508

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2017-04-04T00:00:00Z

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1084511724

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2605065784

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28375159

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