Hippocampal FGF-2 and BDNF overexpression attenuates epileptogenesis-associated neuroinflammation and reduces spontaneous recurrent seizures. - Wikidata - awgldk - TriplyDB

Hippocampal FGF-2 and BDNF overexpression attenuates epileptogenesis-associated neuroinflammation and reduces spontaneous recurrent seizures.

Hippocampal FGF-2 and BDNF overexpression attenuates epileptogenesis-associated neuroinflammation and reduces spontaneous recurrent seizures.

http://www.wikidata.org/entity/Q30497503

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Current prospects and challenges for epilepsy gene therapy Silencing Status Epilepticus-Induced BDNF Expression with Herpes Simplex Virus Type-1 Based Amplicon Vectors The potential of antiseizure drugs and agents that act on novel molecular targets as antiepileptogenic treatments.FGF-2 released from degenerating neurons exerts microglial-induced neuroprotection via FGFR3-ERK signaling pathway.Administration of simvastatin after kainic acid-induced status epilepticus restrains chronic temporal lobe epilepsy.Optogenetic and potassium channel gene therapy in a rodent model of focal neocortical epilepsy.Excitotoxic brain injury in adult zebrafish stimulates neurogenesis and long-distance neuronal integration.Gene therapy in epilepsy-is it time for clinical trials?Plasma BDNF and PDGF-AA levels are associated with high TCD velocity and stroke in children with sickle cell anemia.Environmental enrichment attenuates hippocampal neuroinflammation and improves cognitive function during influenza infection.IL-1RAcPb signaling regulates adaptive mechanisms in neurons that promote their long-term survival following excitotoxic insults.Neuroprotective and antiepileptogenic effects of combination of anti-inflammatory drugs in the immature brain.Gene Transfer of Brain-derived Neurotrophic Factor (BDNF) Prevents Neurodegeneration Triggered by FXN Deficiency Implication of fibroblast growth factors in epileptogenesis-associated circuit rearrangements.Finding a better drug for epilepsy: antiepileptogenesis targets.Status epilepticus triggers early and late alterations in brain-derived neurotrophic factor and NMDA glutamate receptor Grin2b DNA methylation levels in the hippocampus.Novel approaches to epilepsy treatment.Progress in gene therapy for neurological disorders.Design of innovative therapeutics for pharmacoresistant epilepsy: challenges and needs.Causes of CNS inflammation and potential targets for anticonvulsants.Regenerative medicine for epilepsy: from basic research to clinical application.A Molecular Approach to Epilepsy Management: from Current Therapeutic Methods to Preconditioning Efforts.May BDNF Be Implicated in the Exercise-Mediated Regulation of Inflammation? Critical Review and Synthesis of Evidence.Inflammatory aspects of epileptogenesis: contribution of molecular inflammatory mechanisms.Neuropeptide Y mitigates neuropathology and motor deficits in mouse models of Machado-Joseph disease.Transplantation of bone marrow mononuclear cells modulates hippocampal expression of growth factors in chronically epileptic animals.Peroxisome proliferator-activated receptor-γ agonist inhibits the mammalian target of rapamycin signaling pathway and has a protective effect in a rat model of status epilepticus.Multiple Autologous Bone Marrow-Derived CD271+ Mesenchymal Stem Cell Transplantation Overcomes Drug-Resistant Epilepsy in Children.Physical exercise alters the activation of downstream proteins related to BDNF-TrkB signaling in male Wistar rats with epilepsy.Alpha-Linolenic Acid-Induced Increase in Neurogenesis is a Key Factor in the Improvement in the Passive Avoidance Task After Soman Exposure.Altered cytokine and BDNF levels in autism spectrum disorder.Dietary eicosapentaenoic acid normalizes hippocampal omega-3 and 6 polyunsaturated fatty acid profile, attenuates glial activation and regulates BDNF function in a rodent model of neuroinflammation induced by central interleukin-1β administration.Metallic gold beads in hyaluronic acid: a novel form of gold-based immunosuppression? Investigations of the immunosuppressive effects of metallic gold on cultured J774 macrophages and on neuronal gene expression in experimental autoimmune encephalom Translating regenerative medicine techniques for the treatment of epilepsy Regenerative Strategies for the Central Nervous System Mechanism of BDNF Modulation in GABAergic Synaptic Transmission in Healthy and Disease Brains

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Hippocampal FGF-2 and BDNF overexpression attenuates epileptogenesis-associated neuroinflammation and reduces spontaneous recurrent seizures.

http://www.wikidata.org/entity/Q30497503

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Hippocampal FGF-2 and BDNF ove ...... pontaneous recurrent seizures.

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Hippocampal FGF-2 and BDNF ove ...... pontaneous recurrent seizures.

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Hippocampal FGF-2 and BDNF ove ...... pontaneous recurrent seizures.

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Hippocampal FGF-2 and BDNF ove ...... pontaneous recurrent seizures.

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Hippocampal FGF-2 and BDNF ove ...... pontaneous recurrent seizures.

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Hippocampal FGF-2 and BDNF ove ...... pontaneous recurrent seizures.

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Journal of Neuroinflammation

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Hippocampal FGF-2 and BDNF ove ...... spontaneous recurrent seizures

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Andrea Marzola

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Beatrice Paradiso

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Donata Rodi

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Elena Berto

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Flavia Merigo

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Francesco Osculati

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Graciela Navarro Mora

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Paolo F Fabene

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Roberta Bovolenta

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Silvia Zucchini

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P2860

The emerging role for chemokines in epilepsy

Functions of fibroblast growth factor (FGF)-2 and FGF-5 in astroglial differentiation and blood-brain barrier permeability: evidence from mouse mutants

Tooth pulp inflammation increases brain-derived neurotrophic factor expression in rodent trigeminal ganglion neurons

Localized delivery of fibroblast growth factor-2 and brain-derived neurotrophic factor reduces spontaneous seizures in an epilepsy model.

A role for leukocyte-endothelial adhesion mechanisms in epilepsy

Enhancement of GABA(A)-current run-down in the hippocampus occurs at the first spontaneous seizure in a model of temporal lobe epilepsy.

Antagonism of peripheral inflammation reduces the severity of status epilepticus.

Enhanced astrocytic Ca2+ signals contribute to neuronal excitotoxicity after status epilepticus

Seizure-promoting effect of blood-brain barrier disruption.

Antiepileptogenesis and seizure prevention trials with antiepileptic drugs: meta-analysis of controlled trials.

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10.1186/1742-2094-7-81

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7

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Michele Simonato

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2010-11-18T00:00:00Z

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47814651

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1033625896

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21087489

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2993685

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