Caspase-independent pathways of hair cell death induced by kanamycin in vivo. - Wikidata - awgldk - TriplyDB

Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

http://www.wikidata.org/entity/Q30499344

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Hepatocyte growth factor mimetic protects lateral line hair cells from aminoglycoside exposure New developments in aminoglycoside therapy and ototoxicity Age-related hearing impairment and the triad of acquired hearing loss.XBP1 mitigates aminoglycoside-induced endoplasmic reticulum stress and neuronal cell death.Functional hair cell mechanotransducer channels are required for aminoglycoside ototoxicity An otoprotective role for the apoptosis inhibitor protein survivin.Extracellular divalent cations modulate aminoglycoside-induced hair cell death in the zebrafish lateral line Metformin protects against gentamicin-induced hair cell death in vitro but not ototoxicity in vivo.Assessment of nutrient supplement to reduce gentamicin-induced ototoxicity.Tumor Necrosis Factor-α-Induced Ototoxicity in Mouse Cochlear Organotypic Culture.Sensory hair cell death and regeneration in fishes Molecular regulation of auditory hair cell death and approaches to protect sensory receptor cells and/or stimulate repair following acoustic trauma.Mechanism of alpha-lipoic acid in attenuating kanamycin-induced ototoxicity.Receptor-interacting protein kinases modulate noise-induced sensory hair cell death Cisplatin and aminoglycoside antibiotics: hearing loss and its prevention.Screening for chemicals that affect hair cell death and survival in the zebrafish lateral line Intracellular mechanisms of aminoglycoside-induced cytotoxicity.Apoptosis in acquired and genetic hearing impairment: the programmed death of the hair cell Gentamicin rapidly inhibits mitochondrial metabolism in high-frequency cochlear outer hair cells.Aminoglycoside-induced histone deacetylation and hair cell death in the mouse cochlea.Corticotropin-releasing factor-2 activation prevents gentamicin-induced oxidative stress in cells derived from the inner ear Expression of fractalkine receptor CX3CR1 on cochlear macrophages influences survival of hair cells following ototoxic injury.Drug screening for hearing loss: using the zebrafish lateral line to screen for drugs that prevent and cause hearing loss.A protective mechanism against antibiotic-induced ototoxicity: role of prestin Mechanisms of rapid sensory hair-cell death following co-administration of gentamicin and ethacrynic acid.Activation of cell death pathways in the inner ear of the aging CBA/J mouse.Identification of FDA-approved drugs and bioactives that protect hair cells in the zebrafish (Danio rerio) lateral line and mouse (Mus musculus) utricle.Apoptosis-related genes change their expression with age and hearing loss in the mouse cochlea.The adult mouse utricle as an in vitro preparation for studies of ototoxic-drug-induced sensory hair cell death.Synergistic ototoxicity due to noise exposure and aminoglycoside antibiotics Comparison of activated caspase detection methods in the gentamicin-treated chick cochlea JNK signaling in neomycin-induced vestibular hair cell death.Closure of supporting cell scar formations requires dynamic actin mechanisms.Sox2 and JAGGED1 expression in normal and drug-damaged adult mouse inner ear.Rapid hair cell loss: a mouse model for cochlear lesions.Uptake of fluorescent gentamicin by vertebrate sensory cells in vivo.Mechanisms of noise-induced hearing loss indicate multiple methods of prevention.In vivo overexpression of X-linked inhibitor of apoptosis protein protects against neomycin-induced hair cell loss in the apical turn of the cochlea during the ototoxic-sensitive period.Apoptosis in skeletal muscle and its relevance to atrophy In vitro and in vivo models of drug ototoxicity: studying the mechanisms of a clinical problem.

P2860

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P2860

Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

http://www.wikidata.org/entity/Q30499344

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2006年の論文

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Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

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Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

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Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

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Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

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Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

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Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

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Cell Death & Differentiation

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Caspase-independent pathways of hair cell death induced by kanamycin in vivo.

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A Forge

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H Jiang

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J Schacht

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

The calpain system

Endonuclease G is an apoptotic DNase when released from mitochondria

Implications of CAD and DNase II in ischemic neuronal necrosis specific for the primate hippocampus

Rescue of hearing, auditory hair cells, and neurons by CEP-1347/KT7515, an inhibitor of c-Jun N-terminal kinase activation

Involvement of the mitochondrial permeability transition in gentamicin ototoxicity.

Aminoglycosides: nephrotoxicity

Aminoglycoside antibiotics.

Specific aspartyl and calpain proteases are required for neurodegeneration in C. elegans.

Four deaths and a funeral: from caspases to alternative mechanisms.

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10.1038/SJ.CDD.4401706

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