Mesenchymal gene program-expressing ovarian cancer spheroids exhibit enhanced mesothelial clearance - Wikidata - awgldk - TriplyDB

Mesenchymal gene program-expressing ovarian cancer spheroids exhibit enhanced mesothelial clearance

Mesenchymal gene program-expressing ovarian cancer spheroids exhibit enhanced mesothelial clearance

http://www.wikidata.org/entity/Q30579225

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Rethinking ovarian cancer II: reducing mortality from high-grade serous ovarian cancer EMT-Inducing Molecular Factors in Gynecological Cancers The Mesothelial Origin of Carcinoma Associated-Fibroblasts in Peritoneal Metastasis Mutant p53 regulates ovarian cancer transformed phenotypes through autocrine matrix deposition.Cisplatin Resistant Spheroids Model Clinically Relevant Survival Mechanisms in Ovarian Tumors Dynamic Change in p63 Protein Expression during Implantation of Urothelial Cancer Clusters.Novel Intraperitoneal Treatment With Non-Thermal Plasma-Activated Medium Inhibits Metastatic Potential of Ovarian Cancer Cells Mesothelial cells promote early ovarian cancer metastasis through fibronectin secretion.Ovarian cancer stem cells express ROR1, which can be targeted for anti-cancer-stem-cell therapy.Three-dimensional modeling of ovarian cancer.Characterization of twenty-five ovarian tumour cell lines that phenocopy primary tumours.Establishment of a New Ovarian Cancer Cell Line CA5171 Lysophosphatidic Acid Initiates Epithelial to Mesenchymal Transition and Induces β-Catenin-mediated Transcription in Epithelial Ovarian Carcinoma.Novel Axl-driven signaling pathway and molecular signature characterize high-grade ovarian cancer patients with poor clinical outcome.Cyclin G2 inhibits epithelial-to-mesenchymal transition by disrupting Wnt/β-catenin signaling Functional characterization of a panel of high-grade serous ovarian cancer cell lines as representative experimental models of the disease SUSD2 expression in high-grade serous ovarian cancer correlates with increased patient survival and defective mesothelial clearance.Recent Advances in Understanding, Diagnosing, and Treating Ovarian Cancer.The expression of Nectin-4 on the surface of ovarian cancer cells alters their ability to adhere, migrate, aggregate, and proliferate.Cadherin composition and multicellular aggregate invasion in organotypic models of epithelial ovarian cancer intraperitoneal metastasis.Post-translational modification of the membrane type 1 matrix metalloproteinase (MT1-MMP) cytoplasmic tail impacts ovarian cancer multicellular aggregate dynamics.The Unique Molecular and Cellular Microenvironment of Ovarian Cancer Genes Linked to Endometriosis by GWAS Are Integral to Cytoskeleton Regulation and Suggests That Mesothelial Barrier Homeostasis Is a Factor in the Pathogenesis of Endometriosis.Inhibition of the integrin/FAK signaling axis and c-Myc synergistically disrupts ovarian cancer malignancy.Ginsenoside-Rb1 targets chemotherapy-resistant ovarian cancer stem cells via simultaneous inhibition of Wnt/β-catenin signaling and epithelial-to-mesenchymal transition.E-cadherin: A determinant molecule associated with ovarian cancer progression, dissemination and aggressiveness.An activity-dependent proximity ligation platform for spatially resolved quantification of active enzymes in single cells.Ovarian Cancers: Genetic Abnormalities, Tumor Heterogeneity and Progression, Clonal Evolution and Cancer Stem Cells.TWIST1 induces expression of discoidin domain receptor 2 to promote ovarian cancer metastasis.Consensus reference gene(s) for gene expression studies in human cancers: end of the tunnel visible?Role of tumor microenvironment in ovarian cancer pathobiology.ADH1B promotes mesothelial clearance and ovarian cancer infiltration.Mucins and Truncated -Glycans Unveil Phenotypic Discrepancies between Serous Ovarian Cancer Cell Lines and Primary Tumours Role of tumor microenvironment in the pathobiology of ovarian cancer: Insights and therapeutic opportunities Regulation of Ovarian Cancer Prognosis by Immune Cells in the Tumor Microenvironment Targeting the Microenvironment in High Grade Serous Ovarian Cancer

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Mesenchymal gene program-expressing ovarian cancer spheroids exhibit enhanced mesothelial clearance

http://www.wikidata.org/entity/Q30579225

description

2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Mesenchymal gene program-expre ...... enhanced mesothelial clearance

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Mesenchymal gene program-expre ...... enhanced mesothelial clearance

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Mesenchymal gene program-expre ...... enhanced mesothelial clearance

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Mesenchymal gene program-expre ...... enhanced mesothelial clearance

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Mesenchymal gene program-expre ...... enhanced mesothelial clearance

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Mesenchymal gene program-expre ...... enhanced mesothelial clearance

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Mesenchymal gene program-expre ...... enhanced mesothelial clearance

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Alison Karst

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Dennis J Slamon

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Gordon B Mills

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Gottfried E Konecny

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Huiying Piao

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Joan S Brugge

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Laura M Selfors

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Marcin P Iwanicki

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Michael G Drage

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Rachel A Davidowitz

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P2860

The basics of epithelial-mesenchymal transition

Transforming properties of YAP, a candidate oncogene on the chromosome 11q22 amplicon

The epithelial-mesenchymal transition: new insights in signaling, development, and disease

Disaggregation and invasion of ovarian carcinoma ascites spheroids

Cancer statistics, 2012

A comprehensive analysis of PAX8 expression in human epithelial tumors

Possible involvement of TWIST in enhanced peritoneal metastasis of epithelial ovarian carcinoma

Snail blocks the cell cycle and confers resistance to cell death

The transcription factor snail controls epithelial-mesenchymal transitions by repressing E-cadherin expression

Vascular cell adhesion molecule-1 is a regulator of ovarian cancer peritoneal metastasis.

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2611-2625

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10.1172/JCI69815

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6

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124

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4038562

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