Robust expression of TNF-alpha, IL-1beta, RANTES, and IP-10 by human microglial cells during nonproductive infection with herpes simplex virus.
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Role of microglia in central nervous system infectionsHerpes Simplex Virus Type 1 and Other Pathogens are Key Causative Factors in Sporadic Alzheimer's DiseaseDeciphering Human Cell-Autonomous Anti-HSV-1 Immunity in the Central Nervous SystemNeuronal CXCL10 directs CD8+ T-cell recruitment and control of West Nile virus encephalitis.Interferon lambda inhibits herpes simplex virus type I infection of human astrocytes and neuronsRIG-I mediates the co-induction of tumor necrosis factor and type I interferon elicited by myxoma virus in primary human macrophagesPassively administered pooled human immunoglobulins exert IL-10 dependent anti-inflammatory effects that protect against fatal HSV encephalitis.Microglia-induced IL-6 protects against neuronal loss following HSV-1 infection of neural progenitor cells.Rabies virus-induced activation of mitogen-activated protein kinase and NF-kappaB signaling pathways regulates expression of CXC and CC chemokine ligands in microgliaCould lysine supplementation prevent Alzheimer's dementia? A novel hypothesis.Overexpression of tumor necrosis factor alpha by a recombinant rabies virus attenuates replication in neurons and prevents lethal infection in miceSeizures and epilepsy in herpes simplex virus encephalitis: current concepts and future directions of pathogenesis and management.Mouse hepatitis virus infection upregulates genes involved in innate immune responses.A role for DNA-dependent activator of interferon regulatory factor in the recognition of herpes simplex virus type 1 by glial cellsReactive oxygen species drive herpes simplex virus (HSV)-1-induced proinflammatory cytokine production by murine microglia.Novel antiviral activity of mung bean sprouts against respiratory syncytial virus and herpes simplex virus -1: an in vitro study on virally infected Vero and MRC-5 cell lines.Latent herpesvirus infection in human trigeminal ganglia causes chronic immune response.Infiltration Pattern of Blood Monocytes into the Central Nervous System during Experimental Herpes Simplex Virus Encephalitis.RIG-I is required for VSV-induced cytokine production by murine glia and acts in combination with DAI to initiate responses to HSV-1.RNA Sequencing of Tumor-Associated Microglia Reveals Ccl5 as a Stromal Chemokine Critical for Neurofibromatosis-1 Glioma Growth.Cerebrospinal fluid cytokine levels and cognitive impairment in cerebral malaria.Impaired intrinsic immunity to HSV-1 in human iPSC-derived TLR3-deficient CNS cells.Uniocular anterior chamber inoculation of a tumor necrosis factor alpha-expressing recombinant of herpes simplex virus type 1 results in more rapid destruction and increased viral replication in the retina of the uninoculated eye.The Impact of Macrophage- and Microglia-Secreted TNFα on Oncolytic HSV-1 Therapy in the Glioblastoma Tumor MicroenvironmentThe immune response to herpes simplex virus type 1 infection in susceptible mice is a major cause of central nervous system pathology resulting in fatal encephalitis.The Interleukin-1 Balance During Encephalitis Is Associated With Clinical Severity, Blood-Brain Barrier Permeability, Neuroimaging Changes, and Disease Outcome.Microglia are the major cellular source of inducible nitric oxide synthase during experimental herpes encephalitisToll-like receptor 7 suppresses virus replication in neurons but does not affect viral pathogenesis in a mouse model of Langat virus infection.Absence of CXCL10 aggravates herpes stromal keratitis with reduced primary neutrophil influx in mice.Prolonged microglial cell activation and lymphocyte infiltration following experimental herpes encephalitisThe growth compromised HSV-2 mutant DeltaRR prevents kainic acid-induced apoptosis and loss of function in organotypic hippocampal culturesCytokines and the neurodevelopmental basis of mental illnessTumor necrosis factor-alpha mediated signaling in neuronal homeostasis and dysfunction.Microglial self-defence mediated through GLT-1 and glutathione.Viral CNS infections: role of glial pattern recognition receptors in neuroinflammationAnimal models of herpes simplex virus immunity and pathogenesis.Signal transducer and activator of transcription 3 (Stat3) regulates host defense and protects mice against herpes simplex virus-1 (HSV-1) infection.The ribonucleotide reductase R1 subunits of herpes simplex virus types 1 and 2 protect cells against TNFα- and FasL-induced apoptosis by interacting with caspase-8.Cigarette smoke decreases innate responses of epithelial cells to rhinovirus infection.CXCL10 production from cytomegalovirus-stimulated microglia is regulated by both human and viral interleukin-10.
P2860
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P2860
Robust expression of TNF-alpha, IL-1beta, RANTES, and IP-10 by human microglial cells during nonproductive infection with herpes simplex virus.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Robust expression of TNF-alpha ...... ion with herpes simplex virus.
@ast
Robust expression of TNF-alpha ...... ion with herpes simplex virus.
@en
type
label
Robust expression of TNF-alpha ...... ion with herpes simplex virus.
@ast
Robust expression of TNF-alpha ...... ion with herpes simplex virus.
@en
prefLabel
Robust expression of TNF-alpha ...... ion with herpes simplex virus.
@ast
Robust expression of TNF-alpha ...... ion with herpes simplex virus.
@en
P2093
P1476
Robust expression of TNF-alpha ...... tion with herpes simplex virus
@en
P2093
P2888
P304
P356
10.1080/13550280152403254
P577
2001-06-01T00:00:00Z
P5875
P6179
1030999112