Prolonged microglial cell activation and lymphocyte infiltration following experimental herpes encephalitis - Wikidata - awgldk - TriplyDB

Prolonged microglial cell activation and lymphocyte infiltration following experimental herpes encephalitis

Prolonged microglial cell activation and lymphocyte infiltration following experimental herpes encephalitis

http://www.wikidata.org/entity/Q37037960

about

Inflammatory monocytes and the pathogenesis of viral encephalitis Nitric Oxide Protects against Infection-Induced Neuroinflammation by Preserving the Stability of the Blood-Brain Barrier Angiotensin-converting enzyme overexpression in myelomonocytes prevents Alzheimer's-like cognitive decline Persistent humoral immune responses in the CNS limit recovery of reactivated murine cytomegalovirus Herpes simplex type I (HSV-1) infection of the nervous system: is an immune response a good thing?Astragalus Polysaccharide Protects Astrocytes from Being Infected by HSV-1 through TLR3/NF-κB Signaling Pathway.Passively administered pooled human immunoglobulins exert IL-10 dependent anti-inflammatory effects that protect against fatal HSV encephalitis.Microglia-induced IL-6 protects against neuronal loss following HSV-1 infection of neural progenitor cells.Chronic cortical and subcortical pathology with associated neurological deficits ensuing experimental herpes encephalitis.Herpes simplex virus induces neural oxidative damage via microglial cell Toll-like receptor-2.Glial cells suppress postencephalitic CD8+ T lymphocytes through PD-L1.Interferon regulatory factor 3-dependent pathways are critical for control of herpes simplex virus type 1 central nervous system infection.Excess neutrophil infiltration during cytomegalovirus brain infection of interleukin-10-deficient mice Overview of cellular immunotherapy for patients with glioblastoma.Immunoexcitotoxicity as a central mechanism in chronic traumatic encephalopathy-A unifying hypothesis.Modulation of experimental herpes encephalitis-associated neurotoxicity through sulforaphane treatment.Seizures and epilepsy in herpes simplex virus encephalitis: current concepts and future directions of pathogenesis and management.Mouse hepatitis virus infection upregulates genes involved in innate immune responses.Infiltrating regulatory B cells control neuroinflammation following viral brain infection SOCS1/3 expression levels in HSV-1-infected, cytokine-polarized and -unpolarized macrophages A role for DNA-dependent activator of interferon regulatory factor in the recognition of herpes simplex virus type 1 by glial cells Activated CD8+ T lymphocytes inhibit neural stem/progenitor cell proliferation: role of interferon-gamma.Reactive oxygen species drive herpes simplex virus (HSV)-1-induced proinflammatory cytokine production by murine microglia.Memory T cells persisting in the brain following MCMV infection induce long-term microglial activation via interferon-γ.Granulocytic immune infiltrates are essential for the efficient formation of breast cancer liver metastases Chronic morphine and HIV-1 Tat promote differential central nervous system trafficking of CD3+ and Ly6C+ immune cells in a murine Streptococcus pneumoniae infection model.Infiltration Pattern of Blood Monocytes into the Central Nervous System during Experimental Herpes Simplex Virus Encephalitis.Inflammatory monocytes damage the hippocampus during acute picornavirus infection of the brain.Induction of Multiple miR-200/182 Members in the Brains of Mice Are Associated with Acute Herpes Simplex Virus 1 Encephalitis.Chronic reactive gliosis following regulatory T cell depletion during acute MCMV encephalitis.TGFβ Treatment Enhances Glioblastoma Virotherapy by Inhibiting the Innate Immune Response The Impact of Macrophage- and Microglia-Secreted TNFα on Oncolytic HSV-1 Therapy in the Glioblastoma Tumor Microenvironment Microglia and a functional type I IFN pathway are required to counter HSV-1-driven brain lateral ventricle enlargement and encephalitis TLR7-mediated skin inflammation remotely triggers chemokine expression and leukocyte accumulation in the brain NK cells impede glioblastoma virotherapy through NKp30 and NKp46 natural cytotoxicity receptors.T-cell reconstitution during murine acquired immunodeficiency syndrome (MAIDS) produces neuroinflammation and mortality in animals harboring opportunistic viral brain infection The case for immunomodulatory approaches in treating HSV encephalitis.p53 Is a Host Cell Regulator during Herpes Simplex Encephalitis.Modulation of neural stem/progenitor cell proliferation during experimental Herpes Simplex encephalitis is mediated by differential FGF-2 expression in the adult brain Phenotypic changes in immune cell subsets reflect increased infarct volume in male vs. female mice.

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Prolonged microglial cell activation and lymphocyte infiltration following experimental herpes encephalitis

http://www.wikidata.org/entity/Q37037960

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

@wuu

2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

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2008年學術文章

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name

Prolonged microglial cell acti ...... perimental herpes encephalitis

@en

Prolonged microglial cell acti ...... erimental herpes encephalitis.

@nl

type

label

Prolonged microglial cell acti ...... perimental herpes encephalitis

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Prolonged microglial cell acti ...... erimental herpes encephalitis.

@nl

prefLabel

Prolonged microglial cell acti ...... perimental herpes encephalitis

@en

Prolonged microglial cell acti ...... erimental herpes encephalitis.

@nl

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Journal of Immunology

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Prolonged microglial cell acti ...... perimental herpes encephalitis

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Cristina P Marques

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James R Lokensgard

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Joseph M Palmquist

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Shuxian Hu

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Stina L Urban

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Herpes simplex encephalitis treated with acyclovir: diagnosis and long term outcome

Robust expression of TNF-alpha, IL-1beta, RANTES, and IP-10 by human microglial cells during nonproductive infection with herpes simplex virus.

Role of viral persistence in retaining CD8(+) T cells within the central nervous system

Herpes simplex virus-specific memory CD8+ T cells are selectively activated and retained in latently infected sensory ganglia

Kinetics of virus-specific CD8+ -T-cell expansion and trafficking following central nervous system infection.

Gamma interferon expression during acute and latent nervous system infection by herpes simplex virus type 1.

Neutrophil-mediated suppression of virus replication after herpes simplex virus type 1 infection of the murine cornea.

Relapse of herpes encephalitis after acyclovir therapy: report of two new cases and review of the literature.

Dysregulated interferon-gamma responses during lethal cytomegalovirus brain infection of IL-10-deficient mice.

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6417-6426

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Maxim C-J Cheeran

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23403216

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18941232

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