Chromosomal imbalances in Barrett's adenocarcinoma and the metaplasia-dysplasia-carcinoma sequence.
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Early events during neoplastic progression in Barrett's esophagusThe aurora kinase A inhibitor MLN8237 enhances cisplatin-induced cell death in esophageal adenocarcinoma cellsThe molecular biology of esophageal adenocarcinoma.Persistent p53 mutations in single cells from normal human skinQuantitative gene expression analysis in microdissected archival formalin-fixed and paraffin-embedded tumor tissue.Her-2/neu gene amplification, elevated mRNA expression, and protein overexpression in the metaplasia-dysplasia-adenocarcinoma sequence of Barrett's esophagus.Evaluation of p53 mutations in premalignant esophageal lesions and esophageal adenocarcinoma using laser capture microdissection.Molecular alterations of Barrett's esophagus on microdissected endoscopic biopsies.Comparison of DNA histograms by standard flow cytometry and image cytometry on sections in Barrett's adenocarcinomaAdenocarcinoma of the stomach: a review.Epigenetic and genetic silencing of CHFR in esophageal adenocarcinomas.Origin of metastases: subspecies of cancers generated by intrinsic karyotypic variations.Genetic pathways involved in the progression of Barrett's metaplasia to adenocarcinoma.c-myc amplification is frequent in esophageal adenocarcinoma and correlated with the upregulation of VEGF-A expressionAntireflux surgery for Barrett's oesophagus.Chromosome 4 hyperploidy represents an early genetic aberration in premalignant Barrett's oesophagus.Telomeres, stem cells, senescence, and cancer.Barrett's esophagus: histopathologic definitions and diagnostic criteria.Association of HER2/ErbB2 expression and gene amplification with pathologic features and prognosis in esophageal adenocarcinomas.The development of a fluorescence in situ hybridization assay for the detection of dysplasia and adenocarcinoma in Barrett's esophagus.Molecular cytogenetic evaluation of gastric cardia adenocarcinoma and precursor lesions.Integrative genomics identified RFC3 as an amplified candidate oncogene in esophageal adenocarcinoma.Molecular basis of Barrett's oesophagus and oesophageal adenocarcinomaSimilarity of aberrant DNA methylation in Barrett's esophagus and esophageal adenocarcinoma.Correlation of histology with biomarker status after photodynamic therapy in Barrett esophagus.Changes in screening, prognosis and therapy for esophageal adenocarcinoma in Barrett's esophagusChromosomal instability and copy number alterations in Barrett's esophagus and esophageal adenocarcinoma.Comparative genomic hybridization analysis of genetic aberrations associated with development of esophageal squamous cell carcinoma in Henan, China.Microsatellite alterations in phenotypically normal esophageal squamous epithelium and metaplasia-dysplasia-adenocarcinoma sequence.Chromosomal abnormalities and novel disease-related regions in progression from Barrett's esophagus to esophageal adenocarcinoma.Utility of biomarkers in prediction of response to ablative therapy in Barrett's esophagus.Genome-wide catalogue of chromosomal aberrations in barrett's esophagus and esophageal adenocarcinoma: a high-density single nucleotide polymorphism array analysis.Genetic disparity between primary tumours, disseminated tumour cells, and manifest metastasis.The '-omics' revolution and oesophageal adenocarcinoma.Chromosomal gains and genomic loss of p53 and p16 genes in Barrett's esophagus detected by fluorescence in situ hybridization of cytology specimens.Prolonged exposure to acid and bile induces chromosome abnormalities that precede malignant transformation of benign Barrett's epithelium.Oesophageal adenocarcinoma is associated with a deregulation in the MYC/MAX/MAD network.A comparison of conventional cytology, DNA ploidy analysis, and fluorescence in situ hybridization for the detection of dysplasia and adenocarcinoma in patients with Barrett's esophagus.Metaplastic esophageal columnar epithelium without goblet cells shows DNA content abnormalities similar to goblet cell-containing epithelium.Chromosomal changes during development and progression of prostate adenocarcinomas.
P2860
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P2860
Chromosomal imbalances in Barrett's adenocarcinoma and the metaplasia-dysplasia-carcinoma sequence.
description
2000 nî lūn-bûn
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2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Chromosomal imbalances in Barr ...... -dysplasia-carcinoma sequence.
@ast
Chromosomal imbalances in Barr ...... -dysplasia-carcinoma sequence.
@en
type
label
Chromosomal imbalances in Barr ...... -dysplasia-carcinoma sequence.
@ast
Chromosomal imbalances in Barr ...... -dysplasia-carcinoma sequence.
@en
prefLabel
Chromosomal imbalances in Barr ...... -dysplasia-carcinoma sequence.
@ast
Chromosomal imbalances in Barr ...... -dysplasia-carcinoma sequence.
@en
P2093
P2860
P1476
Chromosomal imbalances in Barr ...... -dysplasia-carcinoma sequence.
@en
P2093
D Angermeier
H Braselmann
H F Zitzelsberger
J R Siewert
P2860
P304
P356
10.1016/S0002-9440(10)64760-8
P407
P577
2000-02-01T00:00:00Z