Neurogenic inflammation is associated with development of edema and functional deficits following traumatic brain injury in rats.
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New Prognostic Biomarkers in Patients With Traumatic Brain InjuryNeuroprotective strategies for traumatic brain injury: improving clinical translationNeurogenic inflammation after traumatic brain injury and its potentiation of classical inflammationTRPV1 activation results in disruption of the blood-brain barrier in the ratMultifunctional drugs for head injuryNK1 receptor blockade is ineffective in improving outcome following a balloon compression model of spinal cord injuryThe Role of Substance P in Secondary Pathophysiology after Traumatic Brain Injury.Insulin alleviates the inflammatory response and oxidative stress injury in cerebral tissues in septic ratsAging and animal models of systemic insult: trauma, burn, and sepsis.Sequential expression of cyclooxygenase-2, glutamate receptor-2, and platelet activating factor receptor in rat hippocampal neurons after fluid percussion injury.Neuroprotection for traumatic brain injury: translational challenges and emerging therapeutic strategiesTreatment with a substance P receptor antagonist is neuroprotective in the intrastriatal 6-hydroxydopamine model of early Parkinson's diseaseThe mammalian tachykinin ligand-receptor system: an emerging target for central neurological disordersStress and traumatic brain injury: a behavioral, proteomics, and histological study.Serum substance P levels are associated with severity and mortality in patients with severe traumatic brain injury.Blast-Associated Shock Waves Result in Increased Brain Vascular Leakage and Elevated ROS Levels in a Rat Model of Traumatic Brain Injury.Recent advances in the development of multifactorial therapies for the treatment of traumatic brain injury.Inflammation in acute CNS injury: a focus on the role of substance PIs the ischemic blood-brain barrier insufficiency responsible for full-blown Alzheimer's disease?Recent advances in neuroprotection for treating traumatic brain injury.Blocking neurogenic inflammation for the treatment of acute disorders of the central nervous system.Brain ischemia and ischemic blood-brain barrier as etiological factors in sporadic Alzheimer's diseaseThe effect of venovenous extra-corporeal membrane oxygenation (ECMO) therapy on immune inflammatory response of cerebral tissues in porcine modelEarly Inflammatory Response following Traumatic Brain Injury in Rabbits Using USPIO- and Gd-Enhanced MRI.The multiple faces of quercetin in neuroprotection.Mechanisms underlying the inability to induce area CA1 LTP in the mouse after traumatic brain injury.Substance P antagonists as a therapeutic approach to improving outcome following traumatic brain injuryPhysical exercise ameliorates deficits induced by traumatic brain injury.The pathophysiology of brain swelling associated with subdural hemorrhage: the role of the trigeminovascular system.The role of substance p in ischaemic brain injury.Changes in substance P and NK1 receptor immunohistochemistry following human spinal cord injury.Impact Acceleration Model of Diffuse Traumatic Brain Injury.Serum Levels of Substance P and Mortality in Patients with a Severe Acute Ischemic Stroke.The Role of Neurogenic Inflammation in Blood-Brain Barrier Disruption and Development of Cerebral Oedema Following Acute Central Nervous System (CNS) Injury.Capsaicin exhibits neuroprotective effects in a model of transient global cerebral ischemia in Mongolian gerbils.Substance P is associated with the development of brain edema and functional deficits after traumatic brain injury.Walker 256 tumour cells increase substance P immunoreactivity locally and modify the properties of the blood-brain barrier during extravasation and brain invasion.A substance P antagonist increases brain intracellular free magnesium concentration after diffuse traumatic brain injury in rats.Neurogenic mediators contribute to local edema induced by Micrurus lemniscatus venom.Preclinical modelling of militarily relevant traumatic brain injuries: Challenges and recommendations for future directions.
P2860
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P2860
Neurogenic inflammation is associated with development of edema and functional deficits following traumatic brain injury in rats.
description
2004 nî lūn-bûn
@nan
2004 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Neurogenic inflammation is ass ...... raumatic brain injury in rats.
@ast
Neurogenic inflammation is ass ...... raumatic brain injury in rats.
@en
type
label
Neurogenic inflammation is ass ...... raumatic brain injury in rats.
@ast
Neurogenic inflammation is ass ...... raumatic brain injury in rats.
@en
prefLabel
Neurogenic inflammation is ass ...... raumatic brain injury in rats.
@ast
Neurogenic inflammation is ass ...... raumatic brain injury in rats.
@en
P2093
P1433
P1476
Neurogenic inflammation is ass ...... raumatic brain injury in rats.
@en
P2093
P356
10.1016/J.NPEP.2003.12.003
P577
2004-02-01T00:00:00Z