Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
about
Motor protein Myo1c is a podocyte protein that facilitates the transport of slit diaphragm protein Neph1 to the podocyte membraneStructural Analysis of the Myo1c and Neph1 Complex Provides Insight into the Intracellular Movement of Neph1Comparison of methylprednisolone plus prednisolone with prednisolone alone as initial treatment in adult-onset minimal change disease: a retrospective cohort studyExpression patterns of podocyte-associated mRNAs in patients with proliferative or non-proliferative glomerulopathiesTGF-β-activated kinase 1 is crucial in podocyte differentiation and glomerular capillary formation.Induction of podocyte VEGF164 overexpression at different stages of development causes congenital nephrosis or steroid-resistant nephrotic syndromeVascular endothelial growth factor receptor 2 direct interaction with nephrin links VEGF-A signals to actin in kidney podocytesNephrin Preserves Podocyte Viability and Glomerular Structure and Function in Adult Kidneys.Role of podocyte slit diaphragm as a filtration barrier.Myo1c is an unconventional myosin required for zebrafish glomerular development.Functions of the podocyte proteins nephrin and Neph3 and the transcriptional regulation of their genes.Therapeutic target for nephrotic syndrome: Identification of novel slit diaphragm associated molecules.Pathogenesis of childhood idiopathic nephrotic syndrome: a paradigm shift from T-cells to podocytes.Pathogenesis of proteinuria in idiopathic minimal change disease: molecular mechanisms.CRIM1 is localized to the podocyte filtration slit diaphragm of the adult human kidney.aPKCλ maintains the integrity of the glomerular slit diaphragm through trafficking of nephrin to the cell surface.Neph1 is reduced in primary focal segmental glomerulosclerosis, minimal change nephrotic syndrome, and corresponding experimental animal models of adriamycin-induced nephropathy and puromycin aminonucleoside nephrosis.Ultrastructural study on nephrin expression in experimental puromycin aminonucleoside nephrosis.Dissociation of NEPH1 from nephrin is involved in development of a rat model of focal segmental glomerulosclerosis.Proximal Tubular Expression Patterns of Megalin and Cubilin in Proteinuric Nephropathies.Nephrin Loss Can Be Used to Predict Remission and Long-term Renal Outcome in Patients With Minimal Change Disease.Reduced INF2 expression in nephrotic syndrome is possibly related to clinical severity of steroid resistance in children.Nephrin Signaling in the Podocyte: An Updated View of Signal Regulation at the Slit Diaphragm and Beyond.Characterization of a novel disease-associated mutation within NPHS1 and its effects on nephrin phosphorylation and signaling
P2860
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P2860
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
@ast
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
@en
type
label
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
@ast
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
@en
prefLabel
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
@ast
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
@en
P2093
P356
P1476
Altered ultrastructural distribution of nephrin in minimal change nephrotic syndrome.
@en
P2093
Erna Pettersson
Fredrik Dunér
Karl Tryggvason
Magnus Söderberg
Silwa Mengarelli Widholm
Vesa Ruotsalainen
P356
10.1093/NDT/18.1.70
P407
P577
2003-01-01T00:00:00Z