p53-independent upregulation of KILLER/DR5 TRAIL receptor expression by glucocorticoids and interferon-gamma.
about
In chronic pancreatitis, widespread emergence of TRAIL receptors in epithelia coincides with neoexpression of TRAIL by pancreatic stellate cells of early fibrotic areasMechanism of action for N-substituted benzamide-induced apoptosisInhibition of ataxia telangiectasia mutated kinase activity enhances TRAIL-mediated apoptosis in human melanoma cellsBetulinic acid suppresses constitutive and TNFalpha-induced NF-kappaB activation and induces apoptosis in human prostate carcinoma PC-3 cells.DR5 knockout mice are compromised in radiation-induced apoptosisTissue specific expression of p53 target genes suggests a key role for KILLER/DR5 in p53-dependent apoptosis in vivo.Insights into cancer therapeutic design based on p53 and TRAIL receptor signaling.Chemotherapeutic approaches for targeting cell death pathways.Cleavage of p53-vimentin complex enhances tumor necrosis factor-related apoptosis-inducing ligand-mediated apoptosis of rheumatoid arthritis synovial fibroblastsDeath receptors in chemotherapy and cancer.Triptolide sensitizes AML cells to TRAIL-induced apoptosis via decrease of XIAP and p53-mediated increase of DR5.ERK mediated upregulation of death receptor 5 overcomes the lack of p53 functionality in the diaminothiazole DAT1 induced apoptosis in colon cancer models: efficiency of DAT1 in Ras-Raf mutated cells.Expanding roles for CD4 T cells and their subpopulations in tumor immunity and therapy.The role of TRAIL death receptors in the treatment of hematological malignancies.The herbal compound cryptotanshinone restores sensitivity in cancer cells that are resistant to the tumor necrosis factor-related apoptosis-inducing ligandNotch1 signaling sensitizes tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis in human hepatocellular carcinoma cells by inhibiting Akt/Hdm2-mediated p53 degradation and up-regulating p53-dependent DR5 expressionBetulinic Acid-Mediated Apoptosis in Human Prostate Cancer Cells Involves p53 and Nuclear Factor-Kappa B (NF-κB) Pathways.Inhibition of Notch1 signaling overcomes resistance to the death ligand Trail by specificity protein 1-dependent upregulation of death receptor 5.Triptolide sensitizes TRAIL-induced apoptosis in prostate cancer cells via p53-mediated DR5 up-regulation.Inhibition of Aurora B kinase sensitizes a subset of human glioma cells to TRAIL concomitant with induction of TRAIL-R2.Cytotoxic drug-induced, p53-mediated upregulation of caspase-8 in tumor cells.The farnesyltransferase inhibitor lonafarnib induces CCAAT/enhancer-binding protein homologous protein-dependent expression of death receptor 5, leading to induction of apoptosis in human cancer cells.TRAIL is a key target in S-phase slowing-dependent apoptosis induced by interferon-beta in cervical carcinoma cells.Cooperation of betulinic acid and TRAIL to induce apoptosis in tumor cells.Histone deacetylase inhibitors upregulate death receptor 5/TRAIL-R2 and sensitize apoptosis induced by TRAIL/APO2-L in human malignant tumor cells.Translational control of tumor necrosis factor-related apoptosis-inducing ligand death receptor expression in melanoma cells.Coupling of endoplasmic reticulum stress to CDDO-Me-induced up-regulation of death receptor 5 via a CHOP-dependent mechanism involving JNK activation.Overaccumulation of p53-mediated autophagy protects against betulinic acid-induced apoptotic cell death in colorectal cancer cells.Alveolar epithelial cells in idiopathic pulmonary fibrosis display upregulation of TRAIL, DR4 and DR5 expression with simultaneous preferential over-expression of pro-apoptotic marker p53.Modulation of inflammasome-mediated pulmonary immune activation by type I IFNs protects bone marrow homeostasis during systemic responses to Pneumocystis lung infection.Targeting apoptosis signaling pathways for anticancer therapyPromoter structure and transcription initiation sites of the human death receptor 5/TRAIL-R2 gene.Betulinic Acid for cancer treatment and prevention.The bile acid glycochenodeoxycholate induces trail-receptor 2/DR5 expression and apoptosis.Bile acids stimulate cFLIP phosphorylation enhancing TRAIL-mediated apoptosis.
P2860
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P2860
p53-independent upregulation of KILLER/DR5 TRAIL receptor expression by glucocorticoids and interferon-gamma.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
name
p53-independent upregulation o ...... rticoids and interferon-gamma.
@ast
p53-independent upregulation o ...... rticoids and interferon-gamma.
@en
type
label
p53-independent upregulation o ...... rticoids and interferon-gamma.
@ast
p53-independent upregulation o ...... rticoids and interferon-gamma.
@en
prefLabel
p53-independent upregulation o ...... rticoids and interferon-gamma.
@ast
p53-independent upregulation o ...... rticoids and interferon-gamma.
@en
P356
P1476
p53-independent upregulation o ...... rticoids and interferon-gamma.
@en
P2093
P304
P356
10.1006/EXCR.2000.5073
P407
P577
2001-01-01T00:00:00Z