CCK independently activates intracellular trypsinogen and NF-kappaB in rat pancreatic acinar cells.
about
Dominant negative p38 mitogen-activated protein kinase expression inhibits NF-kappaB activation in AR42J cells.Transcriptional regulation of CXC-ELR chemokines KC and MIP-2 in mouse pancreatic acini.Src Dependent Pancreatic Acinar Injury Can Be Initiated Independent of an Increase in Cytosolic Calcium.Intracellular activation of trypsinogen in transgenic mice induces acute but not chronic pancreatitis.High-mobility group box 1 protein and its role in severe acute pancreatitis.Pancreas-specific RelA/p65 truncation increases susceptibility of acini to inflammation-associated cell death following cerulein pancreatitis.Mechanisms of acinar cell injury in acute pancreatitis.Innate Immune Molecule Surfactant Protein D Attenuates Sepsis-induced Acute Pancreatic Injury through Modulating Apoptosis and NF-κB-mediated Inflammation.The p21-activated kinase, PAK2, is important in the activation of numerous pancreatic acinar cell signaling cascades and in the onset of early pancreatitis eventsSrc kinases play a novel dual role in acute pancreatitis affecting severity but no role in stimulated enzyme secretionNelfinavir/ritonavir reduces acinar injury but not inflammation during mouse caerulein pancreatitisIntracellular trypsin induces pancreatic acinar cell death but not NF-kappaB activation.Cerulein-induced chronic pancreatitis does not require intra-acinar activation of trypsinogen in mice.New insights into the pathogenesis of pancreatitis.Molecular mechanisms of pancreatic injury.Pathogenic mechanisms of acute pancreatitis.The role of protein synthesis and digestive enzymes in acinar cell injury.Evidence for a role of mitogen-activated protein kinases in the treatment of experimental acute pancreatitis.Noggin attenuates cerulein-induced acute pancreatitis and impaired autophagy.Intra-acinar trypsinogen activation mediates early stages of pancreatic injury but not inflammation in mice with acute pancreatitis.Constitutive IKK2 activation in acinar cells is sufficient to induce pancreatitis in vivoRegulator of calcineurin 1 controls growth plasticity of adult pancreas.Activation of nuclear factor-κB in acinar cells increases the severity of pancreatitis in mice.Activation of nuclear factor kappaB in acinar cells does not provoke acute pancreatitisDigesting new information about the role of trypsin in pancreatitis.Resolvin D1 protects against inflammation in experimental acute pancreatitis and associated lung injury.
P2860
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P2860
CCK independently activates intracellular trypsinogen and NF-kappaB in rat pancreatic acinar cells.
description
2001 nî lūn-bûn
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2001 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի մարտին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
CCK independently activates in ...... n rat pancreatic acinar cells.
@ast
CCK independently activates in ...... n rat pancreatic acinar cells.
@en
type
label
CCK independently activates in ...... n rat pancreatic acinar cells.
@ast
CCK independently activates in ...... n rat pancreatic acinar cells.
@en
prefLabel
CCK independently activates in ...... n rat pancreatic acinar cells.
@ast
CCK independently activates in ...... n rat pancreatic acinar cells.
@en
P2093
P2860
P1476
CCK independently activates in ...... n rat pancreatic acinar cells.
@en
P2093
P2860
P304
P356
10.1152/AJPCELL.2001.280.3.C465
P577
2001-03-01T00:00:00Z