Neuronal death is an active, caspase-dependent process after moderate but not severe DNA damage.
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Irradiation Can Selectively Kill Tumor Cells while Preserving Erythrocyte Viability in a Co-Culture SystemRapid phosphorylation of histone H2A.X following ionotropic glutamate receptor activationThe repair function of the multifunctional DNA repair/redox protein APE1 is neuroprotective after ionizing radiation.Mechanisms of radiation toxicity in transformed and non-transformed cellsCross-talk between calpain and caspase proteolytic systems during neuronal apoptosis.Tetracycline derivatives and ceftriaxone, a cephalosporin antibiotic, protect neurons against apoptosis induced by ionizing radiation.
P2860
Neuronal death is an active, caspase-dependent process after moderate but not severe DNA damage.
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2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Neuronal death is an active, c ...... ate but not severe DNA damage.
@ast
Neuronal death is an active, c ...... ate but not severe DNA damage.
@en
type
label
Neuronal death is an active, c ...... ate but not severe DNA damage.
@ast
Neuronal death is an active, c ...... ate but not severe DNA damage.
@en
prefLabel
Neuronal death is an active, c ...... ate but not severe DNA damage.
@ast
Neuronal death is an active, c ...... ate but not severe DNA damage.
@en
P2860
P1476
Neuronal death is an active, c ...... ate but not severe DNA damage.
@en
P2093
P2860
P304
P356
10.1046/J.1471-4159.2001.00070.X
P407
P577
2001-01-01T00:00:00Z