Arrhythmic disorder mapped to chromosome 1q42-q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts.
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Calsequestrin 2 and arrhythmiasDysfunctional ryanodine receptors in the heart: new insights into complex cardiovascular diseasesCatecholaminergic polymorphic ventricular tachycardia: electrocardiographic characteristics and optimal therapeutic strategies to prevent sudden deathPatient Specific Induced Pluripotent Stem Cell-Derived Cardiomyocytes for Drug Development and Screening In Catecholaminergic Polymorphic Ventricular TachycardiaA missense mutation in a highly conserved region of CASQ2 is associated with autosomal recessive catecholamine-induced polymorphic ventricular tachycardia in Bedouin families from IsraelInvolvement of the cardiac ryanodine receptor/calcium release channel in catecholaminergic polymorphic ventricular tachycardiaCalsequestrin mutations and catecholaminergic polymorphic ventricular tachycardiaThe RYR2-encoded ryanodine receptor/calcium release channel in patients diagnosed previously with either catecholaminergic polymorphic ventricular tachycardia or genotype negative, exercise-induced long QT syndrome: a comprehensive open reading framInherited dysfunction of sarcoplasmic reticulum Ca2+ handling and arrhythmogenesisAntiarrhythmic Effects of Dantrolene in Patients with Catecholaminergic Polymorphic Ventricular Tachycardia and Replication of the Responses Using iPSC ModelsEpidemiology of ventricular tachyarrhythmia : Any changes in the past decades?Exercise-induced bidirectional ventricular tachycardia with alternating right and left bundle branch block-type patterns--a case report.Genes, exercise and sudden death: molecular basis of familial catecholaminergic polymorphic ventricular tachycardia.A missense mutation in CASQ2 is associated with autosomal recessive catecholamine-induced polymorphic ventricular tachycardia in Bedouin families from Israel.Identification of novel missense mutations of cardiac ryanodine receptor gene in exercise-induced sudden death at autopsy.Search for cardiac calcium cycling gene mutations in familial ventricular arrhythmias resembling catecholaminergic polymorphic ventricular tachycardia.Catecholaminergic polymorphic ventricular tachycardia: a current overview.Autosomal recessive catecholamine-induced polymorphic ventricular tachycardia.Recent developments in the management of patients at risk for sudden cardiac death.Therapeutic approach for patients with catecholaminergic polymorphic ventricular tachycardia: state of the art and future developments.The role of stress test for predicting genetic mutations and future cardiac events in asymptomatic relatives of catecholaminergic polymorphic ventricular tachycardia probands.Clinical characteristics and mutational analysis of the RyR2 gene in seven Czech families with catecholaminergic polymorphic ventricular tachycardia.A novel cardiac ryanodine receptor gene (RyR2) mutation in an athlete with aborted sudden cardiac death: a case of adult-onset catecholaminergic polymorphic ventricular tachycardia.Catecholaminergic polymorphic ventricular tachycardia in children: analysis of therapeutic strategies and outcomes from an international multicenter registry.Potential role of cardiac calsequestrin in the lethal arrhythmic effects of cocaineOptimizing catecholaminergic polymorphic ventricular tachycardia therapy in calsequestrin-mutant miceGenetics of sudden cardiac death.The ryanodine receptor in cardiac physiology and disease.Acute atrial arrhythmogenicity and altered Ca(2+) homeostasis in murine RyR2-P2328S hearts.Flecainide therapy reduces exercise-induced ventricular arrhythmias in patients with catecholaminergic polymorphic ventricular tachycardiaCellular mechanisms underlying the development of catecholaminergic ventricular tachycardia.Modulation of transmural repolarization.Calcium and arrhythmogenesis.Myocardial disease in failing hearts: defective excitation-contraction coupling.Array comparative genomic hybridization identifies a heterozygous deletion of exon 3 of the RYR2 geneCardiac and skeletal muscle disorders caused by mutations in the intracellular Ca2+ release channels.Ryanodine receptor-targeted anti-arrhythmic therapy.Cardiac repolarization. The long and short of it.Sudden cardiac death and inherited arrhythmia syndromes.Amplification of spatial dispersion of repolarization underlies sudden cardiac death associated with catecholaminergic polymorphic VT, long QT, short QT and Brugada syndromes.
P2860
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P2860
Arrhythmic disorder mapped to chromosome 1q42-q43 causes malignant polymorphic ventricular tachycardia in structurally normal hearts.
description
1999 nî lūn-bûn
@nan
1999 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Arrhythmic disorder mapped to ...... in structurally normal hearts.
@ast
Arrhythmic disorder mapped to ...... in structurally normal hearts.
@en
type
label
Arrhythmic disorder mapped to ...... in structurally normal hearts.
@ast
Arrhythmic disorder mapped to ...... in structurally normal hearts.
@en
prefLabel
Arrhythmic disorder mapped to ...... in structurally normal hearts.
@ast
Arrhythmic disorder mapped to ...... in structurally normal hearts.
@en
P2093
P1476
Arrhythmic disorder mapped to ...... in structurally normal hearts.
@en
P2093
Heikkilä P
Kainulainen K
Paavonen T
Toivonen L
Viitasalo M
P304
P356
10.1016/S0735-1097(99)00461-1
P407
P50
P577
1999-12-01T00:00:00Z