Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
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Generation of 5-(2'-deoxycytidyl)methyl radical and the formation of intrastrand cross-link lesions in oligodeoxyribonucleotidesMutagenic effects of abasic and oxidized abasic lesions in Saccharomyces cerevisiae.Mutational specificity of gamma-radiation-induced guanine-thymine and thymine-guanine intrastrand cross-links in mammalian cells and translesion synthesis past the guanine-thymine lesion by human DNA polymerase etaA powerful antiradiation compound revealed by a new high-throughput screening method.DNA damage and interstrand cross-link formation upon irradiation of aryl iodide C-nucleotide analoguesReactivity of Nucleic Acid Radicals.Direct strand scission from a nucleobase radical in RNAProduct and mechanistic analysis of the reactivity of a C6-pyrimidine radical in RNA.Oxygen independent DNA interstrand cross-link formation by a nucleotide radical.Double-strand breaks from a radical commonly produced by DNA-damaging agents.Use of fluorescence sensors to determine that 2-deoxyribonolactone is the major alkali-labile deoxyribose lesion produced in oxidatively damaged DNA.Direct strand scission in double stranded RNA via a C5-pyrimidine radical.Photochemical generation and reactivity of the major hydroxyl radical adduct of thymidineHuman DNA polymerase β, but not λ, can bypass a 2-deoxyribonolactone lesion together with proliferating cell nuclear antigen.Elucidating DNA damage and repair processes by independently generating reactive and metastable intermediates.Synthesis and analysis of oligonucleotides containing abasic site analoguesDNA tandem lesion repair by strand displacement synthesis and nucleotide excision repair.The mutagenicity of thymidine glycol in Escherichia coli is increased when it is part of a tandem lesionDNA double strand cleavage via interstrand hydrogen atom abstraction.Quantification of 8-oxodGuo lesions in double-stranded DNA using a photoelectrochemical DNA sensor.An overview of chemical processes that damage cellular DNA: spontaneous hydrolysis, alkylation, and reactions with radicals.5,6-Dihydropyrimidine peroxyl radical reactivity in DNA.Formation and repair of oxidatively generated damage in cellular DNA.DNA strand damage product analysis provides evidence that the tumor cell-specific cytotoxin tirapazamine produces hydroxyl radical and acts as a surrogate for O(2).Pyrimidine Nucleobase Radical Reactivity in DNA and RNA.DNA Damage Emanating From a Neutral Purine Radical Reveals the Sequence Dependent Convergence of the Direct and Indirect Effects of γ-Radiolysis.Hydrated Electron Transfer to Nucleobases in Aqueous Solutions Revealed by Ab Initio Molecular Dynamics Simulations.A new understanding towards the reactivity of DNA peroxy radicals.
P2860
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P2860
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
description
2003 nî lūn-bûn
@nan
2003 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
@ast
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
@en
type
label
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
@ast
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
@en
prefLabel
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
@ast
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
@en
P356
P1476
Tandem lesions are the major products resulting from a pyrimidine nucleobase radical.
@en
P2093
K Nolan Carter
Marc M Greenberg
P304
13376-13378
P356
10.1021/JA036629U
P407
P577
2003-11-01T00:00:00Z