Implication of the hypoxia response element of the Vegf promoter in mouse models of retinal and choroidal neovascularization, but not retinal vascular development.
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Placental growth factor contributes to micro-vascular abnormalization and blood-retinal barrier breakdown in diabetic retinopathyBlockade of VEGFR1 and 2 suppresses pathological angiogenesis and vascular leakage in the eyeAstrocyte-derived vascular endothelial growth factor stabilizes vessels in the developing retinal vasculatureNetrin-4 inhibits angiogenesis via binding to neogenin and recruitment of Unc5B.Genetic and pharmacological inhibition of JNK ameliorates hypoxia-induced retinopathy through interference with VEGF expression.Inhibitory efficacy of hypoxia-inducible factor 1alpha short hairpin RNA plasmid DNA-loaded poly (D, L-lactide-co-glycolide) nanoparticles on choroidal neovascularization in a laser-induced rat model.Genome-wide identification of hypoxia-inducible factor binding sites and target genes by a probabilistic model integrating transcription-profiling data and in silico binding site prediction.The Effect of Interleukin 38 on Angiogenesis in a Model of Oxygen-induced Retinopathy.Cedilanid inhibits retinal neovascularization in a mouse model of oxygen-induced retinopathy.Vascular endothelial growth factor-B gene transfer exacerbates retinal and choroidal neovascularization and vasopermeability without promoting inflammation.Digoxin inhibits retinal ischemia-induced HIF-1alpha expression and ocular neovascularization.Aflibercept for the treatment of age-related macular degeneration.Hypoxia specific SDF-1 expression by retinal pigment epithelium initiates bone marrow-derived cells to participate in Choroidal neovascularization in a laser-induced mouse model.Targeting VE-PTP activates TIE2 and stabilizes the ocular vasculaturePeroxisome Proliferator-Activated Receptor Expression in Murine Models and Humans with Age-related Macular Degeneration.TOR-centric view on insulin resistance and diabetic complications: perspective for endocrinologists and gerontologists.Alterations of choroidal blood flow regulation in young healthy subjects with complement factor H polymorphism.VEGF receptor blockade markedly reduces retinal microglia/macrophage infiltration into laser-induced CNV.TNFalpha is required for late BRB breakdown in diabetic retinopathy, and its inhibition prevents leukostasis and protects vessels and neurons from apoptosisAntagonism of PDGF-BB suppresses subretinal neovascularization and enhances the effects of blocking VEGF-AVascular cell-adhesion molecule-1 plays a central role in the proangiogenic effects of oxidative stress.Integrin-dependent and -independent functions of astrocytic fibronectin in retinal angiogenesis.Oxidative stress in myopia.Proteasome-dependent regulation of signal transduction in retinal pigment epithelial cells.Molecular pathogenesis of retinal and choroidal vascular diseases.Peroxisome proliferator-activated receptor and age-related macular degenerationTherapeutic effects of PPARα agonists on diabetic retinopathy in type 1 diabetes modelsPGC-1α regulates normal and pathological angiogenesis in the retinaPegaptanib in the treatment of wet, age-related macular degeneration.Role of the retinal vascular endothelial cell in ocular diseaseIntake of dietary salt and drinking water: Implications for the development of age-related macular degeneration.VEGF Trap-Eye for the treatment of neovascular age-related macular degeneration.Autophagy in Diabetic Retinopathy.Ocular neovascularization.Physical disruption of cell-cell contact induces VEGF expression in RPE cells.Manipulating Angiogenesis by Targeting Endothelial Metabolism: Hitting the Engine Rather than the Drivers-A New Perspective?The HIF-1 antagonist acriflavine: visualization in retina and suppression of ocular neovascularization.Central Role of Metabolism in Endothelial Cell Function and Vascular Disease.Potential role of the methylation of VEGF gene promoter in response to hypoxia in oxygen-induced retinopathy: beneficial effect of the absence of AQP4.Tyrosine kinase blocking collagen IV-derived peptide suppresses ocular neovascularization and vascular leakage.
P2860
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P2860
Implication of the hypoxia response element of the Vegf promoter in mouse models of retinal and choroidal neovascularization, but not retinal vascular development.
description
2006 nî lūn-bûn
@nan
2006 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մարտին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Implication of the hypoxia res ...... retinal vascular development.
@ast
Implication of the hypoxia res ...... retinal vascular development.
@en
type
label
Implication of the hypoxia res ...... retinal vascular development.
@ast
Implication of the hypoxia res ...... retinal vascular development.
@en
prefLabel
Implication of the hypoxia res ...... retinal vascular development.
@ast
Implication of the hypoxia res ...... retinal vascular development.
@en
P2093
P921
P356
P1476
Implication of the hypoxia res ...... retinal vascular development.
@en
P2093
Hansheng Liu
Hiroyuki Nambu
Jikui Shen
Peter A Campochiaro
Sadia Aslam
Stanley A Vinores
Wei-Hong Xiao
Yuji Oshima
P304
P356
10.1002/JCP.20525
P577
2006-03-01T00:00:00Z