Genetic and pharmacological inhibition of JNK ameliorates hypoxia-induced retinopathy through interference with VEGF expression.
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The potential utilizations of hydrogen as a promising therapeutic strategy against ocular diseasesNF-κB, JNK, and TLR Signaling Pathways in HepatocarcinogenesisComputer-aided quantification of retinal neovascularization.Perturbation of mouse retinal vascular morphogenesis by anthrax lethal toxin.Cucurbitacin-I (JSI-124) activates the JNK/c-Jun signaling pathway independent of apoptosis and cell cycle arrest in B leukemic cells.Retinopathy of prematurity promising newer modalities of treatment.JNK inhibition reduces apoptosis and neovascularization in a murine model of age-related macular degeneration.TNF-α decreases VEGF secretion in highly polarized RPE cells but increases it in non-polarized RPE cells related to crosstalk between JNK and NF-κB pathways.AMD-associated genes encoding stress-activated MAPK pathway constituents are identified by interval-based enrichment analysisJNK-1 deficiency limits macrophage-mediated antigen-induced arthritis.Activation of cytosolic phospholipase A2 downstream of the Src-phospholipase D1 (PLD1)-protein kinase C γ (PKCγ) signaling axis is required for hypoxia-induced pathological retinal angiogenesisConsequences and utility of the zinc-dependent metalloprotease activity of anthrax lethal toxin.Oxygen-dependent cleavage of the p75 neurotrophin receptor triggers stabilization of HIF-1α.Erythropoietin receptor antibody inhibits oxidative stress induced retinal neovascularization in mice.Endoplasmic reticulum stress and inflammation: mechanisms and implications in diabetic retinopathy.Prevention of VEGF-induced growth and tube formation in human retinal endothelial cells by aldose reductase inhibitionc-Jun N-Terminal Kinase in Inflammation and Rheumatic Diseases.SPAG9 controls the cell motility, invasion and angiogenesis of human osteosarcoma cells.Suppression of ischemia in arterial occlusive disease by JNK-promoted native collateral artery development.Sustained inhibition of neovascularization in vldlr-/- mice following intravitreal injection of cerium oxide nanoparticles and the role of the ASK1-P38/JNK-NF-κB pathway.RhoB regulates the function of macrophages in the hypoxia-induced inflammatory response.HIF-1α siRNA reduces retinal neovascularization in a mouse model of retinopathy of prematurity.Activation of endoplasmic reticulum stress by hyperglycemia is essential for Müller cell-derived inflammatory cytokine production in diabetes.ERK5 Contributes to VEGF Alteration in Diabetic Retinopathy.JNK2 up-regulates hypoxia-inducible factors and contributes to hypoxia-induced erythropoiesis and pulmonary hypertension.Hydrogen saline treatment attenuates hyperoxia-induced retinopathy by inhibition of oxidative stress and reduction of VEGF expression.Increased expression of c-Fos, c-Jun and c-Jun N-terminal kinase associated with neuronal cell death in retinas of diabetic patients.
P2860
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P2860
Genetic and pharmacological inhibition of JNK ameliorates hypoxia-induced retinopathy through interference with VEGF expression.
description
2009 nî lūn-bûn
@nan
2009 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Genetic and pharmacological in ...... rference with VEGF expression.
@ast
Genetic and pharmacological in ...... rference with VEGF expression.
@en
type
label
Genetic and pharmacological in ...... rference with VEGF expression.
@ast
Genetic and pharmacological in ...... rference with VEGF expression.
@en
prefLabel
Genetic and pharmacological in ...... rference with VEGF expression.
@ast
Genetic and pharmacological in ...... rference with VEGF expression.
@en
P2093
P2860
P356
P1476
Genetic and pharmacological in ...... rference with VEGF expression.
@en
P2093
Gabriel G Haddad
James D Lindsey
Jordi Rius
Karen X Duong-Polk
Monica Guma
P2860
P304
P356
10.1073/PNAS.0902659106
P407
P577
2009-05-11T00:00:00Z