Reduction in podocyte density as a pathologic feature in early diabetic nephropathy in rodents: prevention by lipoic acid treatment. - Wikidata - awgldk - TriplyDB

Reduction in podocyte density as a pathologic feature in early diabetic nephropathy in rodents: prevention by lipoic acid treatment.

Reduction in podocyte density as a pathologic feature in early diabetic nephropathy in rodents: prevention by lipoic acid treatment.

http://www.wikidata.org/entity/Q33236615

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High glucose induces podocyte injury via enhanced (pro)renin receptor-Wnt-β-catenin-snail signaling pathway.The expression and significance of neuronal iconic proteins in podocytes.Mouse models of diabetic nephropathy.Podocyte-specific overexpression of GLUT1 surprisingly reduces mesangial matrix expansion in diabetic nephropathy in mice High glucose induces podocyte apoptosis by stimulating TRPC6 via elevation of reactive oxygen species Astragaloside IV, a novel antioxidant, prevents glucose-induced podocyte apoptosis in vitro and in vivo.BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.Blocking ligand occupancy of the αVβ3 integrin inhibits the development of nephropathy in diabetic pigs Mycophenolate mofetil and valsartan inhibit podocyte apoptosis in streptozotocin-induced diabetic rats.Advances in the biology and genetics of the podocytopathies: implications for diagnosis and therapy Root of Polygonum cuspidatum extract reduces progression of diabetes-induced mesangial cell dysfunction via inhibition of platelet-derived growth factor-BB (PDGF-BB) and interaction with its receptor in streptozotocin-induced diabetic rats.Glomerular expression of kidney injury molecule-1 and podocytopenia in diabetic glomerulopathy.Reduced expression of lipoic acid synthase accelerates diabetic nephropathy Extract of Rhizoma Polygonum cuspidatum reduces early renal podocyte injury in streptozotocin‑induced diabetic rats and its active compound emodin inhibits methylglyoxal‑mediated glycation of proteins.Twenty years after ACEIs and ARBs: emerging treatment strategies for diabetic nephropathy.The protective effect of α-Lipoic acid on mitochondria in the kidney of diabetic rats.The Ethanol Extract of Zingiber zerumbet Attenuates Streptozotocin-Induced Diabetic Nephropathy in Rats The protective effects of α-lipoic acid on kidneys in type 2 diabetic Goto-Kakisaki rats via reducing oxidative stress.Rosiglitazone reduces renal and plasma markers of oxidative injury and reverses urinary metabolite abnormalities in the amelioration of diabetic nephropathy.Enhanced expression of Janus kinase-signal transducer and activator of transcription pathway members in human diabetic nephropathy.Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.Effect of insulin on ACE2 activity and kidney function in the non-obese diabetic mouse.Role of endoplasmic reticulum stress in apoptosis of differentiated mouse podocytes induced by high glucose.Oxidative stress and diabetic kidney disease.Nox4 and diabetic nephropathy: with a friend like this, who needs enemies?Molecular and cellular events mediating glomerular podocyte dysfunction and depletion in diabetes mellitus.Renin-angiotensin system within the diabetic podocyte.Advanced glycation end products induce the apoptosis of and inflammation in mouse podocytes through CXCL9-mediated JAK2/STAT3 pathway activation.Overexpression of miR-34c inhibits high glucose-induced apoptosis in podocytes by targeting Notch signaling pathways The effects of nutrition-induced abnormal food metabolism in the Southern Plains woodrat (Neotoma micropus): comparisons of variations of the Western diet.GS-E3D, a new pectin lyase-modified red ginseng extract, inhibited diabetes-related renal dysfunction in streptozotocin-induced diabetic rats.Podocyte COX-2 exacerbates diabetic nephropathy by increasing podocyte (pro)renin receptor expression Losartan reverses glomerular podocytes injury induced by AngII via stabilizing the expression of GLUT1.The effect of oral alpha-lipoic acid on oxidative stress in adolescents with type 1 diabetes mellitus.Glyoxalase-1 overexpression reduces endothelial dysfunction and attenuates early renal impairment in a rat model of diabetes.

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P2860

Reduction in podocyte density as a pathologic feature in early diabetic nephropathy in rodents: prevention by lipoic acid treatment.

http://www.wikidata.org/entity/Q33236615

description

2006 nî lūn-bûn

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2006 թուականի Մարտին հրատարակուած գիտական յօդուած

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2006年の論文

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Reduction in podocyte density ...... tion by lipoic acid treatment.

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Brian Siu

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Frank C Brosius

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Jharna Saha

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Kelli A Sullivan

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William E Smoyer

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P2860

Molecular cloning of cDNAs encoding human GLEPP1, a membrane protein tyrosine phosphatase: characterization of the GLEPP1 protein distribution in human kidney and assignment of the GLEPP1 gene to human chromosome 12p12-p13

Biochemistry and molecular cell biology of diabetic complications

The mechanism of alloxan and streptozotocin action in B cells of the rat pancreas

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Structural-functional relationships in diabetic nephropathy.

Overview: combating diabetic nephropathy.

From the periphery of the glomerular capillary wall toward the center of disease: podocyte injury comes of age in diabetic nephropathy.

Podocyte loss and progressive glomerular injury in type II diabetes.

Podocyte injury promotes progressive nephropathy in zucker diabetic fatty rats.

Podocyte number predicts long-term urinary albumin excretion in Pima Indians with Type II diabetes and microalbuminuria.

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