Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
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HIV gp120- and methamphetamine-mediated oxidative stress induces astrocyte apoptosis via cytochrome P450 2E1Drug Targets for Oxidative Podocyte Injury in Diabetic NephropathyThe balance of powers: Redox regulation of fibrogenic pathways in kidney injuryHypertension is a major contributor to 20-hydroxyeicosatetraenoic acid-mediated kidney injury in diabetic nephropathy.Pericytes, an overlooked player in vascular pathobiologyRole of NADPH Oxidase in Metabolic Disease-Related Renal Injury: An UpdateStore-operated calcium entry and diabetic complicationsCytochrome P450 eicosanoids in hypertension and renal diseaseTranscriptome-based analysis of kidney gene expression changes associated with diabetes in OVE26 mice, in the presence and absence of losartan treatment20-HETE induces hyperglycemia through the cAMP/PKA-PhK-GP pathway.RNA Viruses: ROS-Mediated Cell Death.Protection of podocytes from hyperhomocysteinemia-induced injury by deletion of the gp91phox gene.Redox signaling via lipid raft clustering in homocysteine-induced injury of podocytes.The Role of SIRT1 in Diabetic Kidney Disease.Astragaloside IV, a novel antioxidant, prevents glucose-induced podocyte apoptosis in vitro and in vivo.AMP-activated protein kinase (AMPK) negatively regulates Nox4-dependent activation of p53 and epithelial cell apoptosis in diabetes.Global renal gene expression profiling analysis in B2-kinin receptor null mice: impact of diabetesPoly(ADP-ribose) polymerase-1 (PARP-1) gene deficiency alleviates diabetic kidney disease.Poly(ADP-ribose) polymerase (PARP) inhibition counteracts multiple manifestations of kidney disease in long-term streptozotocin-diabetic rat modelIncreased SHP-1 protein expression by high glucose levels reduces nephrin phosphorylation in podocytes20-HETE and EETs in diabetic nephropathy: a novel mechanistic pathwayLipid raft redox signaling: molecular mechanisms in health and disease.Upstream regulators and downstream effectors of NADPH oxidases as novel therapeutic targets for diabetic kidney disease.EGF receptor deletion in podocytes attenuates diabetic nephropathy.p47(phox) contributes to albuminuria and kidney fibrosis in mice.Osmolarity and glucose differentially regulate aldose reductase activity in cultured mouse podocytes.Targeting NADPH oxidase with a novel dual Nox1/Nox4 inhibitor attenuates renal pathology in type 1 diabetes.Cytochrome P450 1B1 contributes to renal dysfunction and damage caused by angiotensin II in mice.Prediabetic nephropathy as an early consequence of the high-calorie/high-fat diet: relation to oxidative stress.IGF-I increases the expression of fibronectin by Nox4-dependent Akt phosphorylation in renal tubular epithelial cellsContribution of cytochrome P450 1B1 to hypertension and associated pathophysiology: a novel target for antihypertensive agents.The macrophage phagocytic receptor CD36 promotes fibrogenic pathways on removal of apoptotic cells during chronic kidney injury.Salidroside alleviates oxidative stress in the liver with non- alcoholic steatohepatitis in rats.NADPH oxidase-derived H(2)O(2) contributes to angiotensin II-induced aldosterone synthesis in human and rat adrenal cortical cellsProtective effects of leukemia inhibitory factor against oxidative stress during high glucose-induced apoptosis in podocytes.Effect of exercise on kidney function, oxidative stress, and inflammation in type 2 diabetic KK-A(y) miceAngiotensin II stimulates superoxide production in the thick ascending limb by activating NOX4.Thioredoxin-Interacting Protein Deficiency Protects against Diabetic Nephropathy.Mechanisms of bradykinin-induced expression of connective tissue growth factor and nephrin in podocytes.Palmitate induces ER calcium depletion and apoptosis in mouse podocytes subsequent to mitochondrial oxidative stress
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Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 10 February 2009
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vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
@en
Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
@nl
type
label
Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
@en
Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
@nl
prefLabel
Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
@en
Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
@nl
P2093
P2860
P356
P1433
P1476
Mechanisms of podocyte injury in diabetes: role of cytochrome P450 and NADPH oxidases.
@en
P2093
Assaad A Eid
Bridget M Fagg
Hanna E Abboud
Jeffrey L Barnes
Karen Block
Rita Maalouf
P2860
P304
P356
10.2337/DB08-1536
P407
P50
P577
2009-02-10T00:00:00Z