Deletion of IL-4Ralpha on CD4 T cells renders BALB/c mice resistant to Leishmania major infection.
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Lack of IL-4 receptor expression on T helper cells reduces T helper 2 cell polyfunctionality and confers resistance in allergic bronchopulmonary mycosisDendritic cell-mediated vaccination relies on interleukin-4 receptor signaling to avoid tissue damage after Leishmania major infection of BALB/c miceNeutrophil Extracellular Traps Reprogram IL-4/GM-CSF-Induced Monocyte Differentiation to Anti-inflammatory Macrophages.BALB/c mice deficient in CD4 T cell IL-4Rα expression control Leishmania mexicana Load although female but not male mice develop a healer phenotypeLung tumor growth is stimulated in IFN-gamma-/- mice and inhibited in IL-4Ralpha-/- mice.Inflammasomes in host response to protozoan parasites.Nippostrongylus-induced intestinal hypercontractility requires IL-4 receptor alpha-responsiveness by T cells in mice.Endogenous IL-13 plays a crucial role in liver granuloma maturation during Leishmania donovani infection, independent of IL-4Rα-responsive macrophages and neutrophils.Deletion of IL-4 receptor alpha on dendritic cells renders BALB/c mice hypersusceptible to Leishmania major infection.Murine model of chronic L. (Viannia) panamensis infection: role of IL-13 in disease.Does T Helper Differentiation Correlate with Resistance or Susceptibility to Infection with L. major? Some Insights From the Murine Model.T helper1/t helper2 cells and resistance/susceptibility to leishmania infection: is this paradigm still relevant?Prostanoid receptor 2 signaling protects T helper 2 cells from BALB/c mice against activation-induced cell deathUNC93B1 and nucleic acid-sensing Toll-like receptors mediate host resistance to infection with Leishmania majorIL-4Rα on CD4+ T cells plays a pathogenic role in respiratory syncytial virus reinfection in mice infected initially as neonatesInterleukin-4-promoted T helper 2 responses enhance Nippostrongylus brasiliensis-induced pulmonary pathology.IL-4Ralpha responsiveness of non-CD4 T cells contributes to resistance in schistosoma mansoni infection in pan-T cell-specific IL-4Ralpha-deficient mice.Role of IL-15 and IL-21 in viral immunity: applications for vaccines and therapies.Inhibition of caspase-8 activity promotes protective Th1- and Th2-mediated immunity to Leishmania major infection.CD28 and IL-4: two heavyweights controlling the balance between immunity and inflammation.Immune responses during cutaneous and visceral leishmaniasis.Pro- and anti-inflammatory cytokines in cutaneous leishmaniasis: a review.Interleukin-33 amplifies IgE synthesis and triggers mast cell degranulation via interleukin-4 in naïve mice.IL-4-producing B cells regulate T helper cell dichotomy in type 1- and type 2-controlled diseases.IL-4Rα Signaling in Keratinocytes and Early IL-4 Production Are Dispensable for Generating a Curative T Helper 1 Response in Leishmania major-Infected C57BL/6 Mice.IL-4Rα-responsive smooth muscle cells contribute to initiation of TH2 immunity and pulmonary pathology in Nippostrongylus brasiliensis infections.Influence of endogenous opioid systems on T lymphocytes as assessed by the knockout of mu, delta and kappa opioid receptors.PKCδ regulates IL-12p40/p70 production by macrophages and dendritic cells, driving a type 1 healer phenotype in cutaneous leishmaniasis.All-Trans Retinoic Acid Promotes an M1- to M2-Phenotype Shift and Inhibits Macrophage-Mediated Immunity to Leishmania major.Interleukin-4 Receptor Alpha: From Innate to Adaptive Immunity in Murine Models of Cutaneous Leishmaniasis.Protection from EAE by IL-4Ralpha(-/-) macrophages depends upon T regulatory cell involvement.Potential Risks Related to Modulating Interleukin-13 and Interleukin-4 Signalling: A Systematic Review.Dectin-1 Positive Dendritic Cells Expand after Infection with Leishmania major Parasites and Represent Promising Targets for Vaccine Development.IL-4R{alpha}-responsive smooth muscle cells increase intestinal hypercontractility and contribute to resistance during acute Schistosomiasis.
P2860
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P2860
Deletion of IL-4Ralpha on CD4 T cells renders BALB/c mice resistant to Leishmania major infection.
description
2007 nî lūn-bûn
@nan
2007 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Deletion of IL-4Ralpha on CD4 ...... to Leishmania major infection.
@ast
Deletion of IL-4Ralpha on CD4 ...... to Leishmania major infection.
@en
type
label
Deletion of IL-4Ralpha on CD4 ...... to Leishmania major infection.
@ast
Deletion of IL-4Ralpha on CD4 ...... to Leishmania major infection.
@en
prefLabel
Deletion of IL-4Ralpha on CD4 ...... to Leishmania major infection.
@ast
Deletion of IL-4Ralpha on CD4 ...... to Leishmania major infection.
@en
P2093
P2860
P50
P1433
P1476
Deletion of IL-4Ralpha on CD4 ...... to Leishmania major infection
@en
P2093
Andreas Bohms
Berenice Arendse
Christoph Holscher
Frank Brombacher
J Claire Hoving
James Alexander
Richard Kirsch
Thomas Hunig
P2860
P356
10.1371/JOURNAL.PPAT.0030068
P577
2007-05-01T00:00:00Z