The relationship between Abeta and memory in the Tg2576 mouse model of Alzheimer's disease. - Wikidata - awgldk - TriplyDB

The relationship between Abeta and memory in the Tg2576 mouse model of Alzheimer's disease.

The relationship between Abeta and memory in the Tg2576 mouse model of Alzheimer's disease.

http://www.wikidata.org/entity/Q33292445

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Systematic in vivo analysis of the intrinsic determinants of amyloid Beta pathogenicity Days to criterion as an indicator of toxicity associated with human Alzheimer amyloid-beta oligomers Role of ROS and RNS Sources in Physiological and Pathological Conditions Prions, prion-like prionoids, and neurodegenerative disorders Dissecting Alzheimer disease in Down syndrome using mouse models TDP-43: a new player on the AD field?Alzheimer's therapeutics: translation of preclinical science to clinical drug development Behavioral assays with mouse models of Alzheimer's disease: practical considerations and guidelines.Inconsistencies and controversies surrounding the amyloid hypothesis of Alzheimer's disease Entorhinal cortical defects in Tg2576 mice are present as early as 2-4 months of age.Structural Correlates of Antibodies Associated with Acute Reversal of Amyloid -related Behavioral Deficits in a Mouse Model of Alzheimer Disease Spines, plasticity, and cognition in Alzheimer's model mice Inhibiting α-synuclein oligomerization by stable cell-penetrating β-synuclein fragments recovers phenotype of Parkinson's disease model flies Ferulic acid is a nutraceutical β-secretase modulator that improves behavioral impairment and alzheimer-like pathology in transgenic mice p21-activated kinase-aberrant activation and translocation in Alzheimer disease pathogenesis Disassembly of shank and homer synaptic clusters is driven by soluble beta-amyloid(1-40) through divergent NMDAR-dependent signalling pathways Selective butyrylcholinesterase inhibition elevates brain acetylcholine, augments learning and lowers Alzheimer beta-amyloid peptide in rodent Loss of alpha7 nicotinic receptors enhances beta-amyloid oligomer accumulation, exacerbating early-stage cognitive decline and septohippocampal pathology in a mouse model of Alzheimer's disease.Effects of NK-4 in a transgenic mouse model of Alzheimer's disease Amyloid-β oligomer specificity mediated by the IgM isotype--implications for a specific protective mechanism exerted by endogenous auto-antibodies Tau suppression in a neurodegenerative mouse model improves memory function Paternal spatial training enhances offspring's cognitive performance and synaptic plasticity in wild-type but not improve memory deficit in Alzheimer's mice.Annular protofibrils are a structurally and functionally distinct type of amyloid oligomer.Alzheimer disease: a tale of two prions Microglia demonstrate age-dependent interaction with amyloid-β fibrils Experimental sleep deprivation as a tool to test memory deficits in rodents.APP transgenic mice for modelling behavioural and psychological symptoms of dementia (BPSD).Rosiglitazone reversal of Tg2576 cognitive deficits is independent of peripheral gluco-regulatory status.Overexpression of SOD-2 reduces hippocampal superoxide and prevents memory deficits in a mouse model of Alzheimer's disease Structural classification of toxic amyloid oligomers Amyloid-beta protofibril levels correlate with spatial learning in Arctic Alzheimer's disease transgenic mice.Reduced IGF-1 signaling delays age-associated proteotoxicity in mice Presenilin-2 mutation causes early amyloid accumulation and memory impairment in a transgenic mouse model of Alzheimer's disease Beta-secretase inhibitor GRL-8234 rescues age-related cognitive decline in APP transgenic mice.Alzheimer disease models and human neuropathology: similarities and differences Increased tau phosphorylation on mitogen-activated protein kinase consensus sites and cognitive decline in transgenic models for Alzheimer's disease and FTDP-17: evidence for distinct molecular processes underlying tau abnormalities Highly stabilized curcumin nanoparticles tested in an in vitro blood-brain barrier model and in Alzheimer's disease Tg2576 mice Mechanistic and environmental control of the prevalence and lifetime of amyloid oligomers FcγRIIb mediates amyloid-β neurotoxicity and memory impairment in Alzheimer's disease.Chronic administration of R-flurbiprofen attenuates learning impairments in transgenic amyloid precursor protein mice.

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The relationship between Abeta and memory in the Tg2576 mouse model of Alzheimer's disease.

http://www.wikidata.org/entity/Q33292445

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2002 nî lūn-bûn

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2002 թուականի Մարտին հրատարակուած գիտական յօդուած

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2002 թվականի մարտին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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JNEUROSCI.22-05-01858.2002

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Journal of Neuroscience

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The relationship between Abeta ...... e model of Alzheimer's disease

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Ami Mariash

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Deirdre Cooper-Blacketer

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George A Carlson

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Karen H Ashe

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Linda H Younkin

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Linda Kotilinek

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Marcus A Westerman

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Takeshi Kawarabayashi

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Alzheimer's disease

positive regulation of amyloid fibril formation

Amyloid beta precursor protein

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6758862

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