Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons. - Wikidata - awgldk - TriplyDB

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

http://www.wikidata.org/entity/Q33305201

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Pathological and protective roles of glia in chronic pain Mechanical allodynia Cellular and Molecular Mechanisms of Pain Mesoporous silica-supported lipid bilayers (protocells) for DNA cargo delivery to the spinal cord.The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition Aberrant sensory responses are dependent on lesion severity after spinal cord contusion injury in mice.Improvement of spinal non-viral IL-10 gene delivery by D-mannose as a transgene adjuvant to control chronic neuropathic pain.Long-term potentiation of glycinergic synapses triggered by interleukin 1β.NR2 subunits and NMDA receptors on lamina II inhibitory and excitatory interneurons of the mouse dorsal horn.Synaptic GluN2A and GluN2B containing NMDA receptors within the superficial dorsal horn activated following primary afferent stimulation.Mas-related G-protein-coupled receptors inhibit pathological pain in mice.Synaptic pathways and inhibitory gates in the spinal cord dorsal horn VGLUT2 expression in primary afferent neurons is essential for normal acute pain and injury-induced heat hypersensitivity.Pre- and postsynaptic inhibitory control in the spinal cord dorsal horn.Identification of spinal circuits transmitting and gating mechanical pain Three-dimensional organization of local excitatory and inhibitory inputs to neurons in laminae III-IV of the spinal dorsal horn.A feed-forward spinal cord glycinergic neural circuit gates mechanical allodynia Pain processing by spinal microcircuits: afferent combinatorics C-fiber activity-dependent maturation of glycinergic inhibition in the spinal dorsal horn of the postnatal rat.Transient, activity dependent inhibition of transmitter release from low threshold afferents mediated by GABAA receptors in spinal cord lamina III/IV.Synaptic ultrastructure changes in trigeminocervical complex posttrigeminal nerve injury.Fast synaptic inhibition in spinal sensory processing and pain control.Ontogeny of excitatory spinal neurons processing distinct somatic sensory modalities.Innocuous, not noxious, input activates PKCgamma interneurons of the spinal dorsal horn via myelinated afferent fibers.Nociceptive-induced myocardial remote conditioning is mediated by neuronal gamma protein kinase C.Neuropathic pain: a maladaptive response of the nervous system to damage.Glycinergic dysfunction in a subpopulation of dorsal horn interneurons in a rat model of neuropathic pain.Functional Synaptic Integration of Forebrain GABAergic Precursors into the Adult Spinal Cord.Population coding of somatic sensations.Normal and abnormal coding of somatosensory stimuli causing pain.Transmitting pain and itch messages: a contemporary view of the spinal cord circuits that generate gate control.Neural circuits for pain: Recent advances and current views.Antinociceptive effect of hydantoin 3-phenyl-5-(4-ethylphenyl)-imidazolidine-2,4-dione in mice.Identifying functional populations among the interneurons in laminae I-III of the spinal dorsal horn.Dorsal Horn Circuits for Persistent Mechanical Pain.Spinal sensory projection neuron responses to spinal cord stimulation are mediated by circuits beyond gate control.Inhibition Mediated by Glycinergic and GABAergic Receptors on Excitatory Neurons in Mouse Superficial Dorsal Horn Is Location-Specific but Modified by Inflammation.Light touch induces ERK activation in superficial dorsal horn neurons after inflammation: involvement of spinal astrocytes and JNK signaling in touch-evoked central sensitization and mechanical allodynia.Neuronal hyperexcitability in the ventral posterior thalamus of neuropathic rats: modality selective effects of pregabalin.Low-threshold primary afferent drive onto GABAergic interneurons in the superficial dorsal horn of the mouse.

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P2860

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

http://www.wikidata.org/entity/Q33305201

description

2007 nî lūn-bûn

@nan

2007 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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type

label

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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prefLabel

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

@ast

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

@en

Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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Glycine inhibitory dysfunction turns touch into pain through PKCgamma interneurons.

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Daniel L Voisin

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Loïs S Miraucourt

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P2860

Can we conquer pain?

GABA decreases in the spinal cord dorsal horn after peripheral neurectomy

Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma

A conditional deletion of the NR1 subunit of the NMDA receptor in adult spinal cord dorsal horn reduces NMDA currents and injury-induced pain

Mechanisms of Disease: neuropathic pain—a clinical perspective

BDNF from microglia causes the shift in neuronal anion gradient underlying neuropathic pain

Nociceptive stimulation activates locus coeruleus neurones projecting to the somatosensory thalamus in the rat.

Neuropathic pain: aetiology, symptoms, mechanisms, and management.

Acute and chronic craniofacial pain: brainstem mechanisms of nociceptive transmission and neuroplasticity, and their clinical correlates.

Pain pathways and parabrachial circuits in the rat.

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Radhouane Dallel

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