Aberrant sensory responses are dependent on lesion severity after spinal cord contusion injury in mice.

Aberrant sensory responses are dependent on lesion severity after spinal cord contusion injury in mice.

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http://www.wikidata.org/entity/Q33625945
Spinal cord transection-induced allodynia in rats--behavioral, physiopathological and pharmacological characterizationChronic at-level thermal hyperalgesia following rat cervical contusion spinal cord injury is accompanied by neuronal and astrocyte activation and loss of the astrocyte glutamate transporter, GLT1, in superficial dorsal horn.Sensory stimulation prior to spinal cord injury induces post-injury dysesthesia in mice.Persistent at-level thermal hyperalgesia and tactile allodynia accompany chronic neuronal and astrocyte activation in superficial dorsal horn following mouse cervical contusion spinal cord injuryInjured mice at the gym: review, results and considerations for combining chondroitinase and locomotor exercise to enhance recovery after spinal cord injuryThe animal model of spinal cord injury as an experimental pain model.Macrophage migration inhibitory factor (MIF) is essential for inflammatory and neuropathic pain and enhances pain in response to stressDecreased spinothalamic and dorsal column medial lemniscus-mediated function is associated with neuropathic pain after spinal cord injury.von Frey anesthesiometry to assess sensory impairment after acute spinal cord injury caused by thoracolumbar intervertebral disc extrusion in dogs.Oral administration of a small molecule targeted to block proNGF binding to p75 promotes myelin sparing and functional recovery after spinal cord injury.Elevated MMP-9 in the lumbar cord early after thoracic spinal cord injury impedes motor relearning in mice.Botulinum toxin B in the sensory afferent: transmitter release, spinal activation, and pain behaviorLumbar Myeloid Cell Trafficking into Locomotor Networks after Thoracic Spinal Cord Injury.Unique sensory and motor behavior in Thy1-GFP-M mice before and after spinal cord injury.Early transplantation of mesenchymal stem cells after spinal cord injury relieves pain hypersensitivity through suppression of pain-related signaling cascades and reduced inflammatory cell recruitment.D-β-hydroxybutyrate promotes functional recovery and relieves pain hypersensitivity in mice with spinal cord injury.Impaired CD8 T cell antiviral immunity following acute spinal cord injury.
P2860

Aberrant sensory responses are dependent on lesion severity after spinal cord contusion injury in mice.

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http://www.wikidata.org/entity/Q33625945
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Aberrant sensory responses are ...... cord contusion injury in mice.
@ast
Aberrant sensory responses are ...... cord contusion injury in mice.
@en
type
label
Aberrant sensory responses are ...... cord contusion injury in mice.
@ast
Aberrant sensory responses are ...... cord contusion injury in mice.
@en
prefLabel
Aberrant sensory responses are ...... cord contusion injury in mice.
@ast
Aberrant sensory responses are ...... cord contusion injury in mice.
@en
P1433
P1476
P2093
P2860
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P31
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Aberrant sensory responses are ...... cord contusion injury in mice
@en
P2093
P2860
P304
P31
P356
10.1016/J.PAIN.2009.11.023
P407
P433
P478
P50
P577
P5875
P698
P932