A quantitative method to assess extrasynaptic NMDA receptor function in the protective effect of synaptic activity against neurotoxicity

A quantitative method to assess extrasynaptic NMDA receptor function in the protective effect of synaptic activity against neurotoxicity

Item
http://www.wikidata.org/entity/Q33316376
Development of NMDAR antagonists with reduced neurotoxic side effects: a study on GK11Fingolimod Limits Acute Aβ Neurotoxicity and Promotes Synaptic Versus Extrasynaptic NMDA Receptor Functionality in Hippocampal NeuronsSynaptic versus extrasynaptic NMDA receptor signalling: implications for neurodegenerative disorders.Increasing levels of wild-type CREB up-regulates several activity-regulated inhibitor of death (AID) genes and promotes neuronal survival.Synaptic and extrasynaptic NMDA receptors differentially modulate neuronal cyclooxygenase-2 function, lipid peroxidation, and neuroprotection.Elevated synaptic activity preconditions neurons against an in vitro model of ischemiaStretch injury selectively enhances extrasynaptic, GluN2B-containing NMDA receptor function in cortical neuronsRecovery of NMDA receptor currents from MK-801 blockade is accelerated by Mg2+ and memantine under conditions of agonist exposureMagnesium for newborns with hypoxic-ischemic encephalopathy: a systematic review and meta-analysis.The nuclear calcium signaling target, activating transcription factor 3 (ATF3), protects against dendrotoxicity and facilitates the recovery of synaptic transmission after an excitotoxic insult.NMDA receptors: linking physiological output to biophysical operation.NMDA receptor-mediated excitotoxicity depends on the coactivation of synaptic and extrasynaptic receptors.Synaptic activity and nuclear calcium signaling protect hippocampal neurons from death signal-associated nuclear translocation of FoxO3a induced by extrasynaptic N-methyl-D-aspartate receptorsNMDA receptor mediated phosphorylation of GluR1 subunits contributes to the appearance of calcium-permeable AMPA receptors after mechanical stretch injury.GABAA receptor signaling induces osmotic swelling and cell cycle activation of neonatal prominin+ precursors.Synthesis of C5-tetrazole derivatives of 2-amino-adipic acid displaying NMDA glutamate receptor antagonismDysregulated Neurotransmission induces Trans-synaptic degeneration in reconstructed Neuronal Networks
P2860

A quantitative method to assess extrasynaptic NMDA receptor function in the protective effect of synaptic activity against neurotoxicity

Item
http://www.wikidata.org/entity/Q33316376
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
A quantitative method to asses ...... activity against neurotoxicity
@ast
A quantitative method to asses ...... activity against neurotoxicity
@en
A quantitative method to asses ...... ctivity against neurotoxicity.
@nl
type
label
A quantitative method to asses ...... activity against neurotoxicity
@ast
A quantitative method to asses ...... activity against neurotoxicity
@en
A quantitative method to asses ...... ctivity against neurotoxicity.
@nl
prefLabel
A quantitative method to asses ...... activity against neurotoxicity
@ast
A quantitative method to asses ...... activity against neurotoxicity
@en
A quantitative method to asses ...... ctivity against neurotoxicity.
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P478
P50
P577
P5875
P6179
P698
P921
P932
P356
P1433
P1476
A quantitative method to asses ...... activity against neurotoxicity
@en
P2093
P2860
P2888
P304
P31
P356
P478
P50
P577
P5875
P6179
P698
P921
P932