Excitatory amino acids as a final common pathway for neurologic disorders.
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MicroRNA-21 dysregulates the expression of MEF2C in neurons in monkey and human SIV/HIV neurological diseaseHippocampal AMPA receptor gating controlled by both TARP and cornichon proteinsMechanism of memantine block of NMDA-activated channels in rat retinal ganglion cells: uncompetitive antagonismDominant-interfering forms of MEF2 generated by caspase cleavage contribute to NMDA-induced neuronal apoptosisFailures and successes of NMDA receptor antagonists: molecular basis for the use of open-channel blockers like memantine in the treatment of acute and chronic neurologic insultsGeneral anaesthetic actions on ligand-gated ion channelsInhibition of calpain prevents NMDA-induced cell death and beta-amyloid-induced synaptic dysfunction in hippocampal slice culturesAn alpha-mercaptoacrylic acid derivative is a selective nonpeptide cell-permeable calpain inhibitor and is neuroprotective.Memantine preferentially blocks extrasynaptic over synaptic NMDA receptor currents in hippocampal autapsesMechanism of ceftriaxone induction of excitatory amino acid transporter-2 expression and glutamate uptake in primary human astrocytesAge-dependent alterations of corticostriatal activity in the YAC128 mouse model of Huntington diseaseBeneficial effects of n-acetylcysteine on ischaemic brain injuryChorea-acanthocytosis: genetic linkage to chromosome 9q21Equine estrogens differentially inhibit DNA fragmentation induced by glutamate in neuronal cells by modulation of regulatory proteins involved in programmed cell deathHuman neuroepithelial cells express NMDA receptorsGlutamate-induced apoptosis in primary cortical neurons is inhibited by equine estrogens via down-regulation of caspase-3 and prevention of mitochondrial cytochrome c releaseProtection of cortical cells by equine estrogens against glutamate-induced excitotoxicity is mediated through a calcium independent mechanismBehavioral Abnormality Induced by Enhanced Hypothalamo-Pituitary-Adrenocortical Axis Activity under Dietary Zinc Deficiency and Its Usefulness as a ModelNeuroprotection in GlaucomaCentral Role of Glutamate Metabolism in the Maintenance of Nitrogen Homeostasis in Normal and Hyperammonemic BrainSystematic Review of Pharmacological Properties of the Oligodendrocyte LineageCytidine 5'-Diphosphocholine (Citicoline) in Glaucoma: Rationale of Its Use, Current Evidence and Future PerspectivesOld Things New View: Ascorbic Acid Protects the Brain in Neurodegenerative DisordersOxidative stress associated with neuronal apoptosis in experimental models of epilepsyStress-induced deficits in cognition and emotionality: a role of glutamateThe injury resistant ability of melanopsin-expressing intrinsically photosensitive retinal ganglion cellsIs chronic hyponatremia a novel risk factor for hip fracture in the elderly?Neuropathogenesis of HIV-1-associated neurocognitive disorders: a possible involvement of D-serineRole of neurotrophic factor alterations in the neurodegenerative process in HIV associated neurocognitive disordersNeuroprotection by argon ventilation after perinatal asphyxia: a safety study in newborn pigletsUnique aspects of transcriptional regulation in neurons - nuances in NFkappaB and Sp1-related factorsCorrelating AMPA Receptor Activation and Cleft Closure across Subunits: Crystal Structures of the GluR4 Ligand-Binding Domain in Complex with Full and Partial Agonists †S-nitrosylation of Drp1 links excessive mitochondrial fission to neuronal injury in neurodegenerationThe fate of medications evaluated for ischemic stroke pharmacotherapy over the period 1995-2015Role of NMDA Receptor-Mediated Glutamatergic Signaling in Chronic and Acute NeuropathologiesMolecular analysis of RIM1 in autosomal recessive Retinitis pigmentosaThe chemical biology of clinically tolerated NMDA receptor antagonistsMaternal hypoxia during pregnancy delays the development of motor reflexes in newborn miceEndogenous sulphur-containing amino acids: potent agonists at presynaptic metabotropic glutamate autoreceptors in the rat central nervous systemCharacterization of ionotropic glutamate receptors in human lymphocytes
P2860
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P2860
Excitatory amino acids as a final common pathway for neurologic disorders.
description
1994 nî lūn-bûn
@nan
1994 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի մարտին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
name
Excitatory amino acids as a final common pathway for neurologic disorders.
@ast
Excitatory amino acids as a final common pathway for neurologic disorders.
@en
Excitatory amino acids as a final common pathway for neurologic disorders.
@nl
type
label
Excitatory amino acids as a final common pathway for neurologic disorders.
@ast
Excitatory amino acids as a final common pathway for neurologic disorders.
@en
Excitatory amino acids as a final common pathway for neurologic disorders.
@nl
prefLabel
Excitatory amino acids as a final common pathway for neurologic disorders.
@ast
Excitatory amino acids as a final common pathway for neurologic disorders.
@en
Excitatory amino acids as a final common pathway for neurologic disorders.
@nl
P1476
Excitatory amino acids as a final common pathway for neurologic disorders
@en
P2093
P304
P356
10.1056/NEJM199403033300907
P407
P577
1994-03-01T00:00:00Z