Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
about
A novel splicing variant form suppresses the activity of full-length signal transducer and activator of transcription 5A.HMGN2 inducibly binds a novel transactivation domain in nuclear PRLr to coordinate Stat5a-mediated transcription.Adiponectin is an endogenous anti-fibrotic mediator and therapeutic target.Cbl-mediated K63-linked ubiquitination of JAK2 enhances JAK2 phosphorylation and signal transduction.Cyclophilin B as a co-regulator of prolactin-induced gene expression and function in breast cancer cells.Unexploited therapies in breast and prostate cancer: blockade of the prolactin receptor.Mutant prolactin receptor and familial hyperprolactinemia.The early growth response gene Egr2 (Alias Krox20) is a novel transcriptional target of transforming growth factor-β that is up-regulated in systemic sclerosis and mediates profibrotic responses.Engineering synthetic antibody binders for allosteric inhibition of prolactin receptor signaling.Negative cross talk between NFAT1 and Stat5 signaling in breast cancer.Robust identification of transcriptional regulatory networks using a Gibbs sampler on outlier sum statisticMutation of thyroid hormone receptor-β in mice predisposes to the development of mammary tumorsThe adipokine adiponectin has potent anti-fibrotic effects mediated via adenosine monophosphate-activated protein kinase: novel target for fibrosis therapy.The role of prolactin receptor in GH signaling in breast cancer cellsExpression of cyclophilin B is associated with malignant progression and regulation of genes implicated in the pathogenesis of breast cancer.PRLR regulates hepatic insulin sensitivity in mice via STAT5.Role of c-Myb during prolactin-induced signal transducer and activator of transcription 5a signaling in breast cancer cellsImpaired turnover of prolactin receptor contributes to transformation of human breast cells.Early growth response 3 (Egr-3) is induced by transforming growth factor-β and regulates fibrogenic responses.Flaking process increases the NF-κB inhibition activity and melanoidin extractability of coffee.Histone H1 and Chromosomal Protein HMGN2 Regulate Prolactin-induced STAT5 Transcription Factor Recruitment and Function in Breast Cancer Cells.A synthetic TLR3 ligand mitigates profibrotic fibroblast responses by inducing autocrine IFN signaling.Toll-like Receptor 9 Signaling Is Augmented in Systemic Sclerosis and Elicits Transforming Growth Factor β-Dependent Fibroblast Activation.The prolactin receptor transactivation domain is associated with steroid hormone receptor expression and malignant progression of breast cancer.
P2860
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P2860
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
description
2008 nî lūn-bûn
@nan
2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
@ast
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
@en
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter.
@nl
type
label
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
@ast
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
@en
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter.
@nl
prefLabel
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
@ast
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
@en
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter.
@nl
P2093
P2860
P356
P1433
P1476
Quantification of PRL/Stat5 signaling with a novel pGL4-CISH reporter
@en
P2093
Charles V Clevenger
Giovanni Antico
Jiamao Zheng
P2860
P2888
P356
10.1186/1472-6750-8-11
P577
2008-02-06T00:00:00Z
P5875
P6179
1017011527