Increased choroidal neovascularization following laser induction in mice lacking lysyl oxidase-like 1. - Wikidata - awgldk - TriplyDB

Increased choroidal neovascularization following laser induction in mice lacking lysyl oxidase-like 1.

Increased choroidal neovascularization following laser induction in mice lacking lysyl oxidase-like 1.

http://www.wikidata.org/entity/Q33320965

about

Pseudoexfoliation syndrome, a systemic disorder with ocular manifestations Transscleral sustained vasohibin-1 delivery by a novel device suppressed experimentally-induced choroidal neovascularization ADAM8 is selectively up-regulated in endothelial cells and is associated with angiogenesis after spinal cord injury in adult mice Overexpression of HTRA1 leads to ultrastructural changes in the elastic layer of Bruch's membrane via cleavage of extracellular matrix components Reduced choroidal neovascular membrane formation in cyclooxygenase-2 null mice Heat treatment of retinal pigment epithelium induces production of elastic lamina components and antiangiogenic activity.Transcriptome analysis and molecular signature of human retinal pigment epithelium.In-vivo and ex-vivo characterization of laser-induced choroidal neovascularization variability in mice.In-vivo investigation of laser-induced choroidal neovascularization in rat using spectral-domain optical coherence tomography (SD-OCT).Endothelial TWIST1 promotes pathological ocular angiogenesis.Elastin-mediated choroidal endothelial cell migration: possible role in age-related macular degeneration.Genetics of pseudoexfoliation syndrome.Disruption of the blood-aqueous barrier and lens abnormalities in mice lacking lysyl oxidase-like 1 (LOXL1).Treatment of age-related macular degeneration: beyond VEGF.Next-generation therapeutic solutions for age-related macular degeneration.Molecular responses of choroidal endothelial cells to elastin derived peptides through the elastin-binding protein (GLB1).Ketorolac inhibits choroidal neovascularization by suppression of retinal VEGF.High glucose induced differential expression of lysyl oxidase and its isoform in ARPE-19 cells.Multimodal imaging in a patient with traumatic choroidal ruptures.

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P2860

Increased choroidal neovascularization following laser induction in mice lacking lysyl oxidase-like 1.

http://www.wikidata.org/entity/Q33320965

description

2008 nî lūn-bûn

@nan

2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի փետրվարին հրատարակված գիտական հոդված

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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name

Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

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Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

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Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

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type

label

Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

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Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

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Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

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prefLabel

Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

@ast

Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

@en

Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

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Investigative Ophthalmology & Visual Science

P1476

Increased choroidal neovascula ...... lacking lysyl oxidase-like 1.

@en

P2093

Edward Connolly

http://www.w3.org/2001/XMLSchema#string

Evangelos S Gragoudas

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Ivana K Kim

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Margaret M DeAngelis

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Michael Adamian

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Norman A Michaud

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Oleg V Bulgakov

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Szilard Kiss

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Tiansen Li

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Xiaoqing Liu

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P2860

Lysyl oxidase: properties, specificity, and biological roles inside and outside of the cell

Elastic fiber homeostasis requires lysyl oxidase-like 1 protein

Missense variations in the fibulin 5 gene and age-related macular degeneration

Matrix metalloproteinase (MMP) expression in experimental choroidal neovascularization.

MMP-2 and MMP-9 synergize in promoting choroidal neovascularization.

Serum elastin-derived peptides in age-related macular degeneration.

Endogenous endostatin inhibits choroidal neovascularization.

Decreased thickness and integrity of the macular elastic layer of Bruch's membrane correspond to the distribution of lesions associated with age-related macular degeneration.

Failure of elastic fiber homeostasis leads to pelvic floor disorders.

Current concepts in the pathogenesis of age-related macular degeneration.

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2599-2605

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scholarly article

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10.1167/IOVS.07-1508

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6

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49

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5554924

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18296663

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neovascularization

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2770799

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