Mitochondrial membrane potential in human neutrophils is maintained by complex III activity in the absence of supercomplex organisation - Wikidata - awgldk - TriplyDB

Mitochondrial membrane potential in human neutrophils is maintained by complex III activity in the absence of supercomplex organisation

Mitochondrial membrane potential in human neutrophils is maintained by complex III activity in the absence of supercomplex organisation

http://www.wikidata.org/entity/Q33330306

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Genetic variability of respiratory complex abundance, organization and activity in mouse brain Reactive oxygen species in unstimulated hemocytes of the pacific oyster Crassostrea gigas: a mitochondrial involvement Metabolic shifts in immunity and inflammation.Age-related decline in mitochondrial bioenergetics: does supercomplex destabilization determine lower oxidative capacity and higher superoxide production?Inflammation and immunity in the pathogenesis of pulmonary arterial hypertension Coenzyme q and the respiratory chain: coenzyme q pool and mitochondrial supercomplexes Severe exercise and exercise training exert opposite effects on human neutrophil apoptosis via altering the redox status Defective complex I assembly due to C20orf7 mutations as a new cause of Leigh syndrome.Dying for a cause: NETosis, mechanisms behind an antimicrobial cell death modality Reactive oxygen species originating from mitochondria regulate the cardiac sodium channel.OXPHOS supercomplexes as a hallmark of the mitochondrial phenotype of adipogenic differentiated human MSCs Patients with chronic fatigue syndrome performed worse than controls in a controlled repeated exercise study despite a normal oxidative phosphorylation capacity.Hypoxia and metabolic factors that influence inflammatory bowel disease pathogenesis.Pioglitazone restores phagocyte mitochondrial oxidants and bactericidal capacity in chronic granulomatous disease.Megakaryocytic differentiation of K562 cells induced by PMA reduced the activity of respiratory chain complex IV A novel model for studies of blood-mediated long-term responses to cellular transplants Phospholipase D2 (PLD2) shortens the time required for myeloid leukemic cell differentiation: mechanism of action Relevance of mitochondrial genetics and metabolism in cancer development.New tricks from an old dog: mitochondrial redox signaling in cellular inflammation.A review of the mitochondrial and glycolytic metabolism in human platelets and leukocytes: implications for their use as bioenergetic biomarkers Hypoxia and Mucosal Inflammation.Metabolic Factors that Contribute to Lupus Pathogenesis Mitochondrial Dysfunction and Immune Cell Metabolism in Sepsis.New developments on the functions of coenzyme Q in mitochondria.Advances in the understanding of Barth syndrome.Metabolic pathways in immune cell activation and quiescence.Aluminium phosphide-induced leukopenia.Assessment of gold nanoparticles on human peripheral blood cells by metabolic profiling with 1H-NMR spectroscopy, a novel translational approach on a patient-specific basis.Neutrophil extracellular trap cell death requires both autophagy and superoxide generation.Complement C5a Functions as a Master Switch for the pH Balance in Neutrophils Exerting Fundamental Immunometabolic Effects.Gene networks in skeletal muscle following endurance exercise are coexpressed in blood neutrophils and linked with blood inflammation markers.Myeloid nuclear differentiation antigen, neutrophil apoptosis and sepsis Highly coordinated proteome dynamics during reprogramming of somatic cells to pluripotency.Energy metabolic pathways control the fate and function of myeloid immune cells.The cellular and molecular mechanisms for neutropenia in Barth syndrome.To Eat and to Be Eaten: Mutual Metabolic Adaptations of Immune Cells and Intracellular Bacterial Pathogens upon Infection.A Metabolic Shift toward Pentose Phosphate Pathway Is Necessary for Amyloid Fibril- and Phorbol 12-Myristate 13-Acetate-induced Neutrophil Extracellular Trap (NET) Formation Metabolic requirements for neutrophil extracellular traps formation.Subproteome analysis of the neutrophil cytoskeleton Mitochondrial respiratory supercomplex association limits production of reactive oxygen species from complex I.

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P2860

Mitochondrial membrane potential in human neutrophils is maintained by complex III activity in the absence of supercomplex organisation

http://www.wikidata.org/entity/Q33330306

description

2008 nî lūn-bûn

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2008年の論文

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Mitochondrial membrane potenti ...... e of supercomplex organisation

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Mitochondrial membrane potenti ...... e of supercomplex organisation

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Mitochondrial membrane potenti ...... e of supercomplex organisation

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Mitochondrial membrane potenti ...... e of supercomplex organisation

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Arthur J Verhoeven

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Dirk Roos

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Elly de Wit

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Wim Sluiter

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P2860

Respiratory complex III is required to maintain complex I in mammalian mitochondria

Supercomplexes in the respiratory chains of yeast and mammalian mitochondria

Molecular architecture of the rotary motor in ATP synthase

Neutrophil serine proteases: specific regulators of inflammation

The human EGF-TM7 receptor EMR3 is a marker for mature granulocytes

A structure-based model for the synthesis and hydrolysis of ATP by F1-ATPase

Rates of various reactions catalyzed by ATP synthase as related to the mechanism of ATP synthesis.

Structure of a mitochondrial supercomplex formed by respiratory-chain complexes I and III

The ratio of oxidative phosphorylation complexes I-V in bovine heart mitochondria and the composition of respiratory chain supercomplexes.

Architecture of active mammalian respiratory chain supercomplexes.

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Bram J van Raam

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