Subtilase cytotoxin activates PERK, IRE1 and ATF6 endoplasmic reticulum stress-signalling pathways.
about
Escherichia coli Subtilase CytotoxinThe B subunit of an AB5 toxin produced by Salmonella enterica serovar Typhi up-regulates chemokines, cytokines, and adhesion molecules in human macrophage, colonic epithelial, and brain microvascular endothelial cell linesChaperone-targeting cytotoxin and endoplasmic reticulum stress-inducing drug synergize to kill cancer cellsInnate immunity at mucosal surfaces: the IRE1-RIDD-RIG-I pathwayActivation of cell stress response pathways by Shiga toxinsDengue Virus Infection Induces Upregulation of GRP78, Which Acts To Chaperone Viral Antigen ProductionPneumococcal hydrogen peroxide-induced stress signaling regulates inflammatory genesSignaling from the secretory granule to the nucleus: Uhmk1 and PAMEndoplasmic Reticulum Stress and Unfolded Protein Response in Cartilage Pathophysiology; Contributing Factors to Apoptosis and Osteoarthritis.A dietary non-human sialic acid may facilitate hemolytic-uremic syndromeFatal hemorrhage induced by subtilase cytotoxin from Shiga-toxigenic Escherichia coli.Shiga toxins and the pathophysiology of hemolytic uremic syndrome in humans and animals.Escherichia coli subtilase cytotoxin induces apoptosis regulated by host Bcl-2 family proteins Bax/Bak.Induction of apoptosis by Shiga toxins.In Vivo leukocyte changes induced by Escherichia coli subtilase cytotoxin.An intronic variant in the GRP78, a stress-associated gene, improves prediction for liver cirrhosis in persistent HBV carriers.Tissue factor–dependent procoagulant activity of subtilase cytotoxin, a potent AB5 toxin produced by shiga toxigenic Escherichia coli.Differential effects of Escherichia coli subtilase cytotoxin and Shiga toxin 2 on chemokine and proinflammatory cytokine expression in human macrophage, colonic epithelial, and brain microvascular endothelial cell lines.Bioengineered bugs expressing oligosaccharide receptor mimics: toxin-binding probiotics for treatment and prevention of enteric infectionsIdentification of subtilase cytotoxin (SubAB) receptors whose signaling, in association with SubAB-induced BiP cleavage, is responsible for apoptosis in HeLa cells.Action of shiga toxin type-2 and subtilase cytotoxin on human microvascular endothelial cellsShiga toxins: intracellular trafficking to the ER leading to activation of host cell stress responses.Preconditioning with endoplasmic reticulum stress ameliorates endothelial cell inflammation.Regulation of subtilase cytotoxin-induced cell death by an RNA-dependent protein kinase-like endoplasmic reticulum kinase-dependent proteasome pathway in HeLa cellsParasite-induced ER stress response in hepatocytes facilitates Plasmodium liver stage infection.Comparative Characterization of Shiga Toxin Type 2 and Subtilase Cytotoxin Effects on Human Renal Epithelial and Endothelial Cells Grown in Monolayer and Bilayer Conditions.Subtilase cytotoxin induces apoptosis in HeLa cells by mitochondrial permeabilization via activation of Bax/Bak, independent of C/EBF-homologue protein (CHOP), Ire1alpha or JNK signaling.Subtilase cytotoxin enhances Escherichia coli survival in macrophages by suppression of nitric oxide production through the inhibition of NF-κB activation.Limitation of individual folding resources in the ER leads to outcomes distinct from the unfolded protein response.Hyperhomocysteinemia promotes insulin resistance by inducing endoplasmic reticulum stress in adipose tissueDecreased ER-associated degradation of alpha-TCR induced by Grp78 depletion with the SubAB cytotoxin.Activation of the Akt-NF-kappaB pathway by subtilase cytotoxin through the ATF6 branch of the unfolded protein response.Endoplasmic reticulum stress in the intestinal epithelium and inflammatory bowel disease.Subtilase cytotoxin cleaves newly synthesized BiP and blocks antibody secretion in B lymphocytes.GRP94 in ER quality control and stress responsesControl of NF-κB and inflammation by the unfolded protein response.The chlamydial organism Simkania negevensis forms ER vacuole contact sites and inhibits ER-stress.The unfolded protein response triggered by environmental factors.A molecular web: endoplasmic reticulum stress, inflammation, and oxidative stress.Bacteria, the endoplasmic reticulum and the unfolded protein response: friends or foes?
P2860
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P2860
Subtilase cytotoxin activates PERK, IRE1 and ATF6 endoplasmic reticulum stress-signalling pathways.
description
2008 nî lūn-bûn
@nan
2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@ast
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@en
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@nl
type
label
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@ast
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@en
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@nl
prefLabel
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@ast
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@en
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@nl
P2093
P2860
P1476
Subtilase cytotoxin activates ...... um stress-signalling pathways.
@en
P2093
Cheleste M Thorpe
Dakshina M Jandhyala
James C Paton
Jennifer J Wolfson
Kerrie L May
P2860
P304
P356
10.1111/J.1462-5822.2008.01164.X
P577
2008-04-21T00:00:00Z