Parasite burden and CD36-mediated sequestration are determinants of acute lung injury in an experimental malaria model.
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Innate receptors and cellular defense against pulmonary infectionsCD36 and Fyn kinase mediate malaria-induced lung endothelial barrier dysfunction in mice infected with Plasmodium bergheiThe role of animal models for research on severe malariaSequestration and Tissue Accumulation of Human Malaria Parasites: Can We Learn Anything from Rodent Models of Malaria?Imaging Plasmodium immunobiology in the liver, brain, and lung.Plasmodium chabaudi limits early Nippostrongylus brasiliensis-induced pulmonary immune activation and Th2 polarization in co-infected mice.VEGF promotes malaria-associated acute lung injury in miceThe murine cerebral malaria phenomenonICAM-1 is a key receptor mediating cytoadherence and pathology in the Plasmodium chabaudi malaria modelNatural transmission of Plasmodium berghei exacerbates chronic tuberculosis in an experimental co-infection modelSequestration and histopathology in Plasmodium chabaudi malaria are influenced by the immune response in an organ-specific mannerHigh parasite burdens cause liver damage in mice following Plasmodium berghei ANKA infection independently of CD8(+) T cell-mediated immune pathology.Multivariate modelling with (1)H NMR of pleural effusion in murine cerebral malariaExperimental Models of Microvascular Immunopathology: The Example of Cerebral Malaria.A potential role for interleukin-33 and γ-epithelium sodium channel in the pathogenesis of human malaria associated lung injury.Enhanced expression of Fas and FasL modulates apoptosis in the lungs of severe P. falciparum malaria patients with pulmonary edema.Where have all the parasites gone? Modelling early malaria parasite sequestration dynamicsIntegrin αDβ2 (CD11d/CD18) mediates experimental malaria-associated acute respiratory distress syndrome (MA-ARDS).Blockage of Galectin-receptor Interactions by α-lactose Exacerbates Plasmodium berghei-induced Pulmonary Immunopathology.Development of severe pathology in immunized pregnant mice challenged with lethal malaria parasitesCytoadherence of Plasmodium berghei-infected red blood cells to murine brain and lung microvascular endothelial cells in vitro.Association of Heme Oxygenase 1 with Lung Protection in Malaria-Associated ALI/ARDS.Murine infection models for vaccine development: the malaria example.The antimicrobial molecule trappin-2/elafin has anti-parasitic properties and is protective in vivo in a murine model of cerebral malaria.Gancidin W, a potential low-toxicity antimalarial agent isolated from an endophytic Streptomyces SUK10Nitric oxide for the adjunctive treatment of severe malaria: hypothesis and rationale.Biomarkers of hypoxia, endothelial and circulatory dysfunction among climbers in Nepal with AMS and HAPE: a prospective case-control study.Predictive criteria to study the pathogenesis of malaria-associated ALI/ARDS in mice.Targeting Neutrophils to Prevent Malaria-Associated Acute Lung Injury/Acute Respiratory Distress Syndrome in Mice.Antecedent Nippostrongylus infection alters the lung immune response to Plasmodium berghei.CD36 receptor regulates malaria-induced immune responses primarily at early blood stage infection contributing to parasitemia control and resistance to mortality.Ultrastructure of the lung in a murine model of malaria-associated acute lung injury/acute respiratory distress syndrome.Interleukin-18 Antagonism Improved Histopathological Conditions of Malaria Infection in MiceInsights into the Cytoadherence Phenomenon of Plasmodium vivax: The Putative Role of Phosphatidylserine.Editorial: CD36: Russian roulette of host and parasites during malaria infection.A single rapamycin dose protects against late-stage experimental cerebral malaria via modulation of host immunity, endothelial activation and parasite sequestration.Flt3 ligand treatment modulates parasitemia during infection with rodent malaria parasites via MyD88- and IFN-γ-dependent mechanisms.Experimental malaria-associated acute respiratory distress syndrome is dependent on the parasite-host combination and coincides with normocyte invasion.Could Heme Oxygenase-1 Be a New Target for Therapeutic Intervention in Malaria-Associated Acute Lung Injury/Acute Respiratory Distress Syndrome?Immunopathology and Dexamethasone Therapy in a New Model for Malaria-associated Acute Respiratory Distress Syndrome
P2860
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P2860
Parasite burden and CD36-mediated sequestration are determinants of acute lung injury in an experimental malaria model.
description
2008 nî lūn-bûn
@nan
2008 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
name
Parasite burden and CD36-media ...... an experimental malaria model.
@ast
Parasite burden and CD36-media ...... an experimental malaria model.
@en
Parasite burden and CD36-media ...... an experimental malaria model.
@nl
type
label
Parasite burden and CD36-media ...... an experimental malaria model.
@ast
Parasite burden and CD36-media ...... an experimental malaria model.
@en
Parasite burden and CD36-media ...... an experimental malaria model.
@nl
prefLabel
Parasite burden and CD36-media ...... an experimental malaria model.
@ast
Parasite burden and CD36-media ...... an experimental malaria model.
@en
Parasite burden and CD36-media ...... an experimental malaria model.
@nl
P2093
P2860
P50
P1433
P1476
Parasite burden and CD36-media ...... an experimental malaria model
@en
P2093
Fiona E Lovegrove
John T Ruzinski
Samir N Patel
Timothy R Hughes
W Conrad Liles
P2860
P304
P356
10.1371/JOURNAL.PPAT.1000068
P577
2008-05-16T00:00:00Z