Different atrophy-hypertrophy transcription pathways in muscles affected by severe and mild spinal muscular atrophy.
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Systems analysis of transcriptional data provides insights into muscle's biological response to botulinum toxin.Inflammation in ALS and SMA: sorting out the good from the evil.Proteomic assessment of a cell model of spinal muscular atrophyDrug discovery and development for spinal muscular atrophy: lessons from screening approaches and future challenges for clinical development.SMA-MAP: a plasma protein panel for spinal muscular atrophyp38 Mitogen-activated protein kinase stabilizes SMN mRNA through RNA binding protein HuR.Mechanism of neuromuscular dysfunction in Krabbe disease.eIF4EBP3L acts as a gatekeeper of TORC1 in activity-dependent muscle growth by specifically regulating Mef2ca translational initiation.Role of TGF-β signaling in inherited and acquired myopathies.Peripheral SMN restoration is essential for long-term rescue of a severe spinal muscular atrophy mouse model.Identification of a Maleimide-Based Glycogen Synthase Kinase-3 (GSK-3) Inhibitor, BIP-135, that Prolongs the Median Survival Time of Δ7 SMA KO Mouse Model of Spinal Muscular Atrophy.Impaired expression of insulin-like growth factor-1 system in skeletal muscle of amyotrophic lateral sclerosis patients.An Integrative Transcriptomic Analysis for Identifying Novel Target Genes Corresponding to Severity Spectrum in Spinal Muscular AtrophyHistone deacetylase inhibition suppresses myogenin-dependent atrogene activation in spinal muscular atrophy miceConditional expression of TGF-β1 in skeletal muscles causes endomysial fibrosis and myofibers atrophyDevelopments in the discovery of drugs for spinal muscular atrophy: successful beginnings and future prospects.Perspectives in Pediatric Pathology, Chapter 17. Other Hypergonadotropic Hypogonadisms.Supraphysiological expression of survival motor neuron protein from an adenovirus vector does not adversely affect cell function.The Rho Termination Factor of Clostridium botulinum Contains a Prion-Like Domain with a Highly Amyloidogenic Core.Differential induction of muscle atrophy pathways in two mouse models of spinal muscular atrophy.VPAC2 receptor agonist BAY 55-9837 increases SMN protein levels and moderates disease phenotype in severe spinal muscular atrophy mouse models.Effects of Arm Cycling Exercise in Spinal Muscular Atrophy Type II Patients: A Pilot Study.Interventions Targeting Glucocorticoid-Krüppel-like Factor 15-Branched-Chain Amino Acid Signaling Improve Disease Phenotypes in Spinal Muscular Atrophy Mice.Blocking p62-dependent SMN degradation ameliorates spinal muscular atrophy disease phenotypes
P2860
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P2860
Different atrophy-hypertrophy transcription pathways in muscles affected by severe and mild spinal muscular atrophy.
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Different atrophy-hypertrophy ...... mild spinal muscular atrophy.
@ast
Different atrophy-hypertrophy ...... mild spinal muscular atrophy.
@en
type
label
Different atrophy-hypertrophy ...... mild spinal muscular atrophy.
@ast
Different atrophy-hypertrophy ...... mild spinal muscular atrophy.
@en
prefLabel
Different atrophy-hypertrophy ...... mild spinal muscular atrophy.
@ast
Different atrophy-hypertrophy ...... mild spinal muscular atrophy.
@en
P2093
P2860
P356
P1433
P1476
Different atrophy-hypertrophy ...... mild spinal muscular atrophy.
@en
P2093
Caterina Millino
Gerolamo Lanfranchi
Maria Luisa Mostacciuolo
Marina Fanin
Paolo Laveder
P2860
P2888
P356
10.1186/1741-7015-7-14
P407
P577
2009-04-07T00:00:00Z
P5875
P6179
1038370695